{"meta":{"page":1,"per_page":50,"max_per_page":100,"total":3015,"total_is_capped":false,"direct_labels_cover":8,"predictions_cover":3015,"direct_label_status":"direct model label, unvalidated","prediction_status":"machine_predicted_unvalidated (Codex and Gemma teacher distillation)","score_status":"score_only:v0-immature-baseline (scores rank; they never assert a category)","snapshot":{"source":"OpenAlex, pinned release, all 482 partitions","release":"2026-06-24","frame_built":"2026-07-12"},"query_hash":"9a2251501d18","filters":{"topic":"Genetic Neurodegenerative Diseases"}},"results":[{"id":"W4211110682","doi":"10.1038/nrdp.2015.5","title":"Huntington disease","year":2015,"lang":"en","type":"review","venue":"Nature Reviews Disease Primers","topic":"Genetic Neurodegenerative Diseases","field":"Neuroscience","cited_by":1577,"is_retracted":false,"has_abstract":false,"routes":{"ca_aff":true,"ca_fund":false,"ca_venue":false,"about_ca":false},"ca_institutions":"Child and Family Research Institute; University of British Columbia","funders":"Academy of Medical Sciences; Medical Research Council; National Institute for Health and Care Research","keywords":"Huntingtin; Huntington's disease; Huntingtin Protein; Disease; Trinucleotide repeat expansion; Genetics; Biology; Bioinformatics; Medicine; Neuroscience; Gene; Internal medicine; Allele","retraction":null,"screen_n_in":null,"score":{"opus":0.09360441459597763,"gpt":0.3921763582117396,"spread":0.2985719436157619,"validation_status":"score_only:v0-immature-baseline"},"prediction":{"model_version":"codex-gemma-dda1882f352a","candidate_categories":["metaresearch","metaepi_narrow","insufficient_payload"],"consensus_categories":["metaepi_narrow"],"category_scores_codex":[0.000636417,0.001662238,0.003482331,0.0004459031,0.0002609871,0.0002350186,0.001913446,0.0005042287,0.0003360239],"category_scores_gemma":[0.009150434,0.001262208,0.002606701,0.001463968,0.0003156282,0.0002459198,0.0005627969,0.001923933,0.002947278],"about_ca_system_candidate":false,"about_ca_system_consensus":false,"about_ca_system_score_codex":0.0003250877,"about_ca_system_score_gemma":0.002796928,"about_ca_topic_candidate":false,"about_ca_topic_consensus":false,"about_ca_topic_score_codex":9.078864e-7,"about_ca_topic_score_gemma":3.148372e-7,"domain_scores_codex":[0.9905703,0.002880139,0.001487137,0.002654542,0.00139889,0.001008975],"domain_scores_gemma":[0.9924546,0.0003656731,0.001254341,0.002486007,0.0001708563,0.003268453],"domain_codex":null,"domain_gemma":null,"domain_candidate":null,"domain_consensus":null,"study_design_codex":"design_other","study_design_gemma":"not_applicable","study_design_scores_codex":[0.00003465255,0.0002841279,0.000006203618,0.02970293,0.00003192571,0.00009913297,0.000006726633,0.000001480608,0.00001117275,0.0005299086,0.03137249,0.9379193],"study_design_scores_gemma":[0.0001924222,0.00001922847,0.000009514317,0.01047153,0.003561755,0.000003267372,8.958252e-7,0.000002833704,0.00006288703,0.0001532191,0.9843275,0.001194927],"study_design_candidate":"not_applicable","study_design_consensus":null,"genre_codex":"review","genre_gemma":"review","genre_scores_codex":[0.00001003003,0.9905473,0.000006701327,0.0001030135,0.00152882,0.005335783,0.0008887686,0.0002598839,0.001319689],"genre_scores_gemma":[0.0000167776,0.9939851,0.0001356199,0.001998981,0.0007567445,0.001099745,0.0003897429,0.0002880414,0.001329242],"genre_candidate":"review","genre_consensus":"review","teacher_disagreement_score":0.952955,"threshold_uncertainty_score":0.9996125,"prediction_status":"machine_predicted_unvalidated"},"labels":[],"label_agreement":null},{"id":"W2146267270","doi":"10.1126/science.1059581","title":"Loss of Huntingtin-Mediated BDNF Gene Transcription in Huntington's Disease","year":2001,"lang":"en","type":"article","venue":"Science","topic":"Genetic Neurodegenerative Diseases","field":"Neuroscience","cited_by":1324,"is_retracted":false,"has_abstract":true,"routes":{"ca_aff":true,"ca_fund":false,"ca_venue":false,"about_ca":false},"ca_institutions":"University of British Columbia","funders":"","keywords":"Huntingtin; Huntington's disease; Neurotrophic factors; Huntingtin Protein; Brain-derived neurotrophic factor; Biology; Transcription factor; Neuroscience; Mutant; Cell biology; Gene; Internal medicine; Genetics; Disease; Medicine; Receptor","retraction":null,"screen_n_in":null,"score":{"opus":0.02601728161087213,"gpt":0.2697154884627513,"spread":0.2436982068518792,"validation_status":"score_only:v0-immature-baseline"},"prediction":{"model_version":"codex-gemma-dda1882f352a","candidate_categories":[],"consensus_categories":[],"category_scores_codex":[0.0003817507,0.0001377655,0.0001482922,0.0003212609,0.0001574015,0.00005654341,0.0006421962,0.00002117499,0.00006612788],"category_scores_gemma":[0.002330726,0.0001403749,0.00004944416,0.002417439,0.001028195,0.0003792543,0.00008179739,0.00009531502,0.00004500256],"about_ca_system_candidate":false,"about_ca_system_consensus":false,"about_ca_system_score_codex":0.00003564699,"about_ca_system_score_gemma":0.000289807,"about_ca_topic_candidate":false,"about_ca_topic_consensus":false,"about_ca_topic_score_codex":0.00001734447,"about_ca_topic_score_gemma":0.00001303505,"domain_scores_codex":[0.9976723,0.0001063966,0.0003084396,0.0006993665,0.0007293361,0.0004841197],"domain_scores_gemma":[0.9990596,0.0001043161,0.0001197109,0.0003754315,0.0001048654,0.0002360814],"domain_codex":null,"domain_gemma":null,"domain_candidate":null,"domain_consensus":null,"study_design_codex":"bench_or_experimental","study_design_gemma":"bench_or_experimental","study_design_scores_codex":[0.00003052179,0.0001319618,0.07635555,0.000009263023,2.025652e-7,0.00001747584,0.0001129154,0.0004278617,0.9221476,0.0001997613,0.000004216062,0.000562616],"study_design_scores_gemma":[0.0002447555,0.00002611176,0.3094607,0.00001496265,0.000003345974,0.000001850041,0.00001926061,0.0008681458,0.6889223,0.0001858584,0.0001465624,0.0001061181],"study_design_candidate":"bench_or_experimental","study_design_consensus":"bench_or_experimental","genre_codex":"empirical","genre_gemma":"empirical","genre_scores_codex":[0.9983799,0.00004061151,0.0001498551,0.0002761414,0.000269381,0.0002317411,0.0000262458,0.00004638407,0.0005796705],"genre_scores_gemma":[0.9991828,0.00006220665,0.0001546574,0.000423464,0.00003238301,0.00001580909,0.000001918198,0.0000120287,0.0001147004],"genre_candidate":"empirical","genre_consensus":"empirical","teacher_disagreement_score":0.2332253,"threshold_uncertainty_score":0.5724323,"prediction_status":"machine_predicted_unvalidated"},"labels":[],"label_agreement":null},{"id":"W4376133202","doi":"10.1038/s41586-023-05896-x","title":"A draft human pangenome reference","year":2023,"lang":"en","type":"article","venue":"Nature","topic":"Genetic Neurodegenerative Diseases","field":"Neuroscience","cited_by":1148,"is_retracted":false,"has_abstract":true,"routes":{"ca_aff":true,"ca_fund":false,"ca_venue":false,"about_ca":false},"ca_institutions":"McGill University","funders":"National Institute of General Medical Sciences; National Institutes of Health; National Human Genome Research Institute; Novo Nordisk Fonden; Nederlandse Organisatie voor Wetenschappelijk Onderzoek; Wellcome Trust","keywords":"Computational biology; Biology","retraction":null,"screen_n_in":null,"score":{"opus":0.05439339111178432,"gpt":0.331449616159246,"spread":0.2770562250474616,"validation_status":"score_only:v0-immature-baseline"},"prediction":{"model_version":"codex-gemma-dda1882f352a","candidate_categories":["insufficient_payload"],"consensus_categories":[],"category_scores_codex":[0.00004821552,0.0001134793,0.00008837646,0.00009854423,0.0001747996,0.00004445258,0.000325476,0.0002710348,0.0001917204],"category_scores_gemma":[0.0003631811,0.0001003602,0.00004024256,0.0005820002,0.00005835015,0.00005067782,0.0001102516,0.0007842524,0.0009874004],"about_ca_system_candidate":false,"about_ca_system_consensus":false,"about_ca_system_score_codex":0.0000125805,"about_ca_system_score_gemma":0.00002773347,"about_ca_topic_candidate":false,"about_ca_topic_consensus":false,"about_ca_topic_score_codex":0.000001915158,"about_ca_topic_score_gemma":0.000005811457,"domain_scores_codex":[0.998852,0.00008547041,0.00009033274,0.0004187319,0.0002967416,0.0002567641],"domain_scores_gemma":[0.9994597,0.0000734554,0.00003181355,0.000314249,0.00003342644,0.00008734434],"domain_codex":null,"domain_gemma":null,"domain_candidate":null,"domain_consensus":null,"study_design_codex":"bench_or_experimental","study_design_gemma":"bench_or_experimental","study_design_scores_codex":[0.000003268011,0.00001800281,0.0003754073,0.000007395102,9.334381e-7,0.0000417244,0.00006935772,0.00001578008,0.9837206,0.004669191,0.01083725,0.0002411128],"study_design_scores_gemma":[0.0001610915,0.00004381768,0.04593248,0.000006882704,0.000005536374,0.00000380635,0.00002130322,0.00002305021,0.9172335,0.00148543,0.03492841,0.0001547373],"study_design_candidate":"bench_or_experimental","study_design_consensus":"bench_or_experimental","genre_codex":"empirical","genre_gemma":"empirical","genre_scores_codex":[0.9900085,0.00009217142,0.000001625336,0.0007489367,0.0002348177,0.0001332823,0.00006579832,0.0002914997,0.008423379],"genre_scores_gemma":[0.9920434,0.00002560965,0.00001933588,0.002884441,0.0001027396,0.00002296589,0.00001478895,0.00001955793,0.004867154],"genre_candidate":"empirical","genre_consensus":"empirical","teacher_disagreement_score":0.06648712,"threshold_uncertainty_score":0.9997904,"prediction_status":"machine_predicted_unvalidated"},"labels":[],"label_agreement":null},{"id":"W1972771185","doi":"10.1038/nn884","title":"Early mitochondrial calcium defects in Huntington's disease are a direct effect of polyglutamines","year":2002,"lang":"en","type":"article","venue":"Nature Neuroscience","topic":"Genetic Neurodegenerative Diseases","field":"Neuroscience","cited_by":1024,"is_retracted":false,"has_abstract":false,"routes":{"ca_aff":true,"ca_fund":false,"ca_venue":false,"about_ca":false},"ca_institutions":"University of British Columbia","funders":"National Institute on Aging","keywords":"Huntingtin; Huntingtin Protein; Neurodegeneration; Mitochondrion; Biology; Huntington's disease; Calcium; Cell biology; Mutant; Genetically modified mouse; Trinucleotide repeat expansion; Transgene; Molecular biology; Genetics; Gene; Pathology; Internal medicine; Medicine; Disease; Allele","retraction":null,"screen_n_in":null,"score":{"opus":0.01598198027826318,"gpt":0.2652335088383072,"spread":0.249251528560044,"validation_status":"score_only:v0-immature-baseline"},"prediction":{"model_version":"codex-gemma-dda1882f352a","candidate_categories":["metaresearch","metaepi_narrow"],"consensus_categories":[],"category_scores_codex":[0.0001249447,0.0003840863,0.0004197432,0.0003713169,0.0001753196,0.000105455,0.0008820556,0.0001387995,0.00002646723],"category_scores_gemma":[0.01154528,0.000325558,0.0001924251,0.001780107,0.0005000948,0.0003116759,0.0002087517,0.0005844473,0.00002309176],"about_ca_system_candidate":false,"about_ca_system_consensus":false,"about_ca_system_score_codex":0.00003001492,"about_ca_system_score_gemma":0.00003983985,"about_ca_topic_candidate":false,"about_ca_topic_consensus":false,"about_ca_topic_score_codex":0.00001087133,"about_ca_topic_score_gemma":0.000006987916,"domain_scores_codex":[0.996038,0.0006691857,0.0003478363,0.001303734,0.0009774829,0.0006637583],"domain_scores_gemma":[0.9979153,0.0007820654,0.0002512401,0.0006545319,0.00005693103,0.0003399095],"domain_codex":null,"domain_gemma":null,"domain_candidate":null,"domain_consensus":null,"study_design_codex":"bench_or_experimental","study_design_gemma":"bench_or_experimental","study_design_scores_codex":[0.000113391,0.0001804528,0.06594844,0.0000533868,3.749688e-7,0.00010184,0.0000468716,0.0002339237,0.9327668,0.00004698968,0.0002678029,0.0002397391],"study_design_scores_gemma":[0.0006352354,0.0002674014,0.2393908,0.00005094127,0.00001510239,0.000003354606,0.000001505573,0.0007681352,0.7583879,0.00001771002,0.0002352791,0.0002266731],"study_design_candidate":"bench_or_experimental","study_design_consensus":"bench_or_experimental","genre_codex":"empirical","genre_gemma":"empirical","genre_scores_codex":[0.9959713,0.0002911726,0.00001014369,0.0002638115,0.001965493,0.0006591713,0.00008153809,0.00009802977,0.0006593783],"genre_scores_gemma":[0.998138,0.00003312026,0.00001752009,0.001180095,0.0001487588,0.00005794198,5.498898e-7,0.00003780741,0.0003861808],"genre_candidate":"empirical","genre_consensus":"empirical","teacher_disagreement_score":0.1743789,"threshold_uncertainty_score":0.9999197,"prediction_status":"machine_predicted_unvalidated"},"labels":[],"label_agreement":null},{"id":"W2020117714","doi":"10.1038/nrneurol.2014.24","title":"Huntington disease: natural history, biomarkers and prospects for therapeutics","year":2014,"lang":"en","type":"review","venue":"Nature Reviews Neurology","topic":"Genetic Neurodegenerative Diseases","field":"Neuroscience","cited_by":1009,"is_retracted":false,"has_abstract":false,"routes":{"ca_aff":true,"ca_fund":false,"ca_venue":false,"about_ca":false},"ca_institutions":"University of British Columbia","funders":"National Institute of Neurological Disorders and Stroke; Academy of Medical Sciences; National Institute for Health and Care Research","keywords":"Medicine; Natural history; Disease; Natural (archaeology); Internal medicine","retraction":null,"screen_n_in":null,"score":{"opus":0.0562553680185706,"gpt":0.3453506184619287,"spread":0.2890952504433581,"validation_status":"score_only:v0-immature-baseline"},"prediction":{"model_version":"codex-gemma-dda1882f352a","candidate_categories":["metaepi_narrow"],"consensus_categories":[],"category_scores_codex":[0.0004174814,0.001038296,0.002807479,0.0003011604,0.0001746657,0.00005830367,0.0008684549,0.0007247641,0.00002605632],"category_scores_gemma":[0.003089329,0.0007706168,0.0009949979,0.0003151061,0.0004271091,0.00007005738,0.0002320296,0.001625877,0.00004855454],"about_ca_system_candidate":false,"about_ca_system_consensus":false,"about_ca_system_score_codex":0.00008574482,"about_ca_system_score_gemma":0.0003090269,"about_ca_topic_candidate":false,"about_ca_topic_consensus":false,"about_ca_topic_score_codex":4.00605e-7,"about_ca_topic_score_gemma":0.000002171502,"domain_scores_codex":[0.9938927,0.002024373,0.0009802178,0.002068435,0.000309738,0.0007245513],"domain_scores_gemma":[0.9960704,0.001444849,0.001061697,0.00104684,0.00007891766,0.000297332],"domain_codex":null,"domain_gemma":null,"domain_candidate":null,"domain_consensus":null,"study_design_codex":"design_other","study_design_gemma":"not_applicable","study_design_scores_codex":[0.00008230892,0.00009110809,0.000010031,0.03242683,0.00004906482,0.0000183353,0.000009281659,1.133532e-7,0.0003535353,0.002041336,0.0132012,0.9517168],"study_design_scores_gemma":[0.0002526296,0.0001902798,0.00001758852,0.001106015,0.001193749,0.00004543479,7.392745e-8,0.00002197387,0.00009146167,0.0001101837,0.9963723,0.0005983502],"study_design_candidate":"not_applicable","study_design_consensus":null,"genre_codex":"review","genre_gemma":"review","genre_scores_codex":[0.00004486745,0.9903708,0.00001654079,0.0004040627,0.003347746,0.005476092,0.0000934815,0.0001103553,0.0001361234],"genre_scores_gemma":[0.0001044991,0.9862199,0.0001227482,0.01133042,0.0005671181,0.001021296,0.0000649612,0.0001842737,0.0003847141],"genre_candidate":"review","genre_consensus":"review","teacher_disagreement_score":0.983171,"threshold_uncertainty_score":0.9994745,"prediction_status":"machine_predicted_unvalidated"},"labels":[],"label_agreement":null},{"id":"W2124660038","doi":"10.1016/s1474-4422(09)70170-x","title":"Biological and clinical manifestations of Huntington's disease in the longitudinal TRACK-HD study: cross-sectional analysis of baseline data","year":2009,"lang":"en","type":"article","venue":"The Lancet Neurology","topic":"Genetic Neurodegenerative Diseases","field":"Neuroscience","cited_by":973,"is_retracted":false,"has_abstract":false,"routes":{"ca_aff":true,"ca_fund":false,"ca_venue":false,"about_ca":false},"ca_institutions":"University of British Columbia","funders":"National Center for Research Resources; National Institute of Neurological Disorders and Stroke; National Institute for Health and Care Research","keywords":"Huntington's disease; Medicine; Disease; Cohort; Physical medicine and rehabilitation; Cognition; Internal medicine; Psychology; Psychiatry","retraction":null,"screen_n_in":null,"score":{"opus":0.2365662374586302,"gpt":0.4351373238014206,"spread":0.1985710863427904,"validation_status":"score_only:v0-immature-baseline"},"prediction":{"model_version":"codex-gemma-dda1882f352a","candidate_categories":[],"consensus_categories":[],"category_scores_codex":[0.001346832,0.0001155658,0.0003693182,0.0001126169,0.0001000004,0.00002316737,0.0009132683,0.00003609006,0.00003912627],"category_scores_gemma":[0.002850041,0.00006751798,0.00008222769,0.0006066374,0.0006023898,0.00005355995,0.0001776165,0.0002461644,0.000001760455],"about_ca_system_candidate":false,"about_ca_system_consensus":false,"about_ca_system_score_codex":0.000001528809,"about_ca_system_score_gemma":0.00003338434,"about_ca_topic_candidate":false,"about_ca_topic_consensus":false,"about_ca_topic_score_codex":0.00001606846,"about_ca_topic_score_gemma":0.00009177005,"domain_scores_codex":[0.9963566,0.002135085,0.0004850421,0.0005851485,0.0002348309,0.0002032839],"domain_scores_gemma":[0.9963172,0.00245012,0.000219199,0.0009187382,0.00004710255,0.00004764802],"domain_codex":null,"domain_gemma":null,"domain_candidate":null,"domain_consensus":null,"study_design_codex":"observational","study_design_gemma":"observational","study_design_scores_codex":[0.0005311375,0.0008059426,0.9940367,0.000002203645,0.00002301507,0.00003504016,0.00004835442,0.001385377,0.002632153,0.0002273578,0.00003048747,0.0002422821],"study_design_scores_gemma":[0.0004960385,0.0005504765,0.9928454,6.068512e-7,0.000186458,0.000005469747,0.000008422338,0.005279518,0.0002516673,0.0003050208,0.00001767028,0.00005330072],"study_design_candidate":"observational","study_design_consensus":"observational","genre_codex":"empirical","genre_gemma":"empirical","genre_scores_codex":[0.9966213,0.00002505847,0.00005852075,0.002547659,0.00007172511,0.000286388,0.000352019,0.000009587261,0.00002777378],"genre_scores_gemma":[0.9978378,0.000088625,0.00001835263,0.001908983,0.0001081185,0.000007010128,0.00002318643,0.000003961984,0.000003941546],"genre_candidate":"empirical","genre_consensus":"empirical","teacher_disagreement_score":0.003894141,"threshold_uncertainty_score":0.3411972,"prediction_status":"machine_predicted_unvalidated"},"labels":[],"label_agreement":null},{"id":"W2040671431","doi":"10.1038/ng1219","title":"Huntingtin interacts with REST/NRSF to modulate the transcription of NRSE-controlled neuronal genes","year":2003,"lang":"en","type":"article","venue":"Nature Genetics","topic":"Genetic Neurodegenerative Diseases","field":"Neuroscience","cited_by":934,"is_retracted":false,"has_abstract":false,"routes":{"ca_aff":true,"ca_fund":false,"ca_venue":false,"about_ca":false},"ca_institutions":"University of British Columbia","funders":"","keywords":"Huntingtin; Biology; Transcription factor; Huntington's disease; Huntingtin Protein; Repressor; Genetics; Gene silencing; Transcription (linguistics); Cell biology; Gene; Mutant; Internal medicine; Medicine","retraction":null,"screen_n_in":null,"score":{"opus":0.01598519823449769,"gpt":0.259113801613656,"spread":0.2431286033791583,"validation_status":"score_only:v0-immature-baseline"},"prediction":{"model_version":"codex-gemma-dda1882f352a","candidate_categories":[],"consensus_categories":[],"category_scores_codex":[0.0001800205,0.0002683066,0.0002902579,0.0001154604,0.000150593,0.00006601747,0.0004330568,0.0001128873,0.00004657284],"category_scores_gemma":[0.000868444,0.0001760277,0.0001186671,0.0004675696,0.0001267469,0.00004801913,0.00003842709,0.0003872136,0.00001860313],"about_ca_system_candidate":false,"about_ca_system_consensus":false,"about_ca_system_score_codex":0.0000151513,"about_ca_system_score_gemma":0.0001113074,"about_ca_topic_candidate":false,"about_ca_topic_consensus":false,"about_ca_topic_score_codex":0.000001947812,"about_ca_topic_score_gemma":0.00002078129,"domain_scores_codex":[0.997686,0.0004542844,0.000349591,0.0005676351,0.0005884976,0.0003540332],"domain_scores_gemma":[0.9987368,0.000224944,0.0001646417,0.0005447157,0.0001913343,0.0001375893],"domain_codex":null,"domain_gemma":null,"domain_candidate":null,"domain_consensus":null,"study_design_codex":"bench_or_experimental","study_design_gemma":"bench_or_experimental","study_design_scores_codex":[0.0003648244,0.0001054486,0.0007653084,0.00001293042,0.00001132431,0.000006451954,0.0002123553,0.01141591,0.985254,0.0004878631,0.0001668046,0.001196712],"study_design_scores_gemma":[0.001550194,0.0003552394,0.01496342,0.00001807602,0.00006323762,0.00001548718,0.00003613509,0.0005130838,0.9792123,0.0001652155,0.002918899,0.0001886974],"study_design_candidate":"bench_or_experimental","study_design_consensus":"bench_or_experimental","genre_codex":"empirical","genre_gemma":"empirical","genre_scores_codex":[0.9960731,0.000549808,0.0003295426,0.0005954343,0.0005640082,0.0008402588,0.00002693028,0.00003308458,0.0009878495],"genre_scores_gemma":[0.9952177,0.00007352324,0.001400078,0.002781599,0.00009226477,0.00004518936,0.000001822546,0.00004535608,0.0003424932],"genre_candidate":"empirical","genre_consensus":"empirical","teacher_disagreement_score":0.01419811,"threshold_uncertainty_score":0.7178201,"prediction_status":"machine_predicted_unvalidated"},"labels":[],"label_agreement":null},{"id":"W2087505473","doi":"10.1038/nrg1689","title":"Repeat instability: mechanisms of dynamic mutations","year":2005,"lang":"en","type":"article","venue":"Nature Reviews Genetics","topic":"Genetic Neurodegenerative Diseases","field":"Neuroscience","cited_by":914,"is_retracted":false,"has_abstract":false,"routes":{"ca_aff":true,"ca_fund":false,"ca_venue":false,"about_ca":false},"ca_institutions":"SickKids Foundation; Hospital for Sick Children; University of Toronto","funders":"","keywords":"Genome instability; Biology; Genetics; Instability; DNA replication; Mutation; DNA; Trinucleotide repeat expansion; Gene; DNA repair; Chromosome instability; DNA mismatch repair; DNA damage; Allele; Chromosome","retraction":null,"screen_n_in":null,"score":{"opus":0.02740212823109252,"gpt":0.3223885165135724,"spread":0.2949863882824799,"validation_status":"score_only:v0-immature-baseline"},"prediction":{"model_version":"codex-gemma-dda1882f352a","candidate_categories":[],"consensus_categories":[],"category_scores_codex":[0.0002116678,0.0002186521,0.0003547812,0.00009373801,0.00008525544,0.00002000743,0.0003842906,0.0001465455,0.0001286545],"category_scores_gemma":[0.001320233,0.0001881365,0.0001854696,0.0004619857,0.0001239768,0.00005515517,0.00009363932,0.0003167421,0.00009160865],"about_ca_system_candidate":false,"about_ca_system_consensus":false,"about_ca_system_score_codex":0.00004599675,"about_ca_system_score_gemma":0.00007821411,"about_ca_topic_candidate":false,"about_ca_topic_consensus":false,"about_ca_topic_score_codex":3.644192e-7,"about_ca_topic_score_gemma":0.00001486744,"domain_scores_codex":[0.9979199,0.0003768963,0.0005611252,0.0005414293,0.0003601575,0.0002405337],"domain_scores_gemma":[0.9986481,0.0001326901,0.0002429346,0.0007537125,0.0001069597,0.0001156024],"domain_codex":null,"domain_gemma":null,"domain_candidate":null,"domain_consensus":null,"study_design_codex":"bench_or_experimental","study_design_gemma":"bench_or_experimental","study_design_scores_codex":[0.00000523481,0.0001470923,0.00004613011,0.0001023164,0.000002990015,0.000001843311,0.00006327643,0.0002581392,0.9049878,0.00269487,0.0002288186,0.09146152],"study_design_scores_gemma":[0.0001824788,0.00007438403,0.0007878717,0.00004012421,0.00005349958,0.00001091735,0.00000916958,0.0009490738,0.935244,0.002626714,0.05983102,0.0001907035],"study_design_candidate":"bench_or_experimental","study_design_consensus":"bench_or_experimental","genre_codex":"empirical","genre_gemma":"empirical","genre_scores_codex":[0.9222873,0.06323714,0.007676365,0.001215269,0.0006475304,0.002154505,0.0002114154,0.0001072585,0.002463241],"genre_scores_gemma":[0.9702919,0.009866734,0.01785474,0.001635247,0.0000504629,0.00004782351,0.0000102091,0.00002901377,0.0002139355],"genre_candidate":"empirical","genre_consensus":"empirical","teacher_disagreement_score":0.09127082,"threshold_uncertainty_score":0.7671986,"prediction_status":"machine_predicted_unvalidated"},"labels":[],"label_agreement":null},{"id":"W2113661629","doi":"10.1016/s1474-4422(13)70088-7","title":"Predictors of phenotypic progression and disease onset in premanifest and early-stage Huntington's disease in the TRACK-HD study: analysis of 36-month observational data","year":2013,"lang":"en","type":"article","venue":"The Lancet Neurology","topic":"Genetic Neurodegenerative Diseases","field":"Neuroscience","cited_by":839,"is_retracted":false,"has_abstract":false,"routes":{"ca_aff":true,"ca_fund":false,"ca_venue":false,"about_ca":false},"ca_institutions":"University of British Columbia","funders":"Academy of Medical Sciences; National Institute for Health and Care Research; CHDI Foundation","keywords":"Huntington's disease; Observational study; Medicine; Internal medicine; Disease; Longitudinal study; Pathology","retraction":null,"screen_n_in":null,"score":{"opus":0.07709197160351793,"gpt":0.3195445673346506,"spread":0.2424525957311327,"validation_status":"score_only:v0-immature-baseline"},"prediction":{"model_version":"codex-gemma-dda1882f352a","candidate_categories":[],"consensus_categories":[],"category_scores_codex":[0.0004253142,0.0001525543,0.0003738981,0.0001587423,0.00006824295,0.00003352019,0.0007621721,0.00002392925,0.00002274818],"category_scores_gemma":[0.001783188,0.0000878147,0.00002840837,0.0006278811,0.0004082058,0.0001678637,0.00038398,0.00017414,5.93422e-7],"about_ca_system_candidate":false,"about_ca_system_consensus":false,"about_ca_system_score_codex":0.000002600995,"about_ca_system_score_gemma":0.00006869659,"about_ca_topic_candidate":false,"about_ca_topic_consensus":false,"about_ca_topic_score_codex":0.0001441968,"about_ca_topic_score_gemma":0.0004495859,"domain_scores_codex":[0.9972041,0.00131839,0.0003652275,0.0005475453,0.0003265177,0.0002382294],"domain_scores_gemma":[0.9976336,0.001047967,0.000219776,0.00096958,0.00002998283,0.00009909822],"domain_codex":null,"domain_gemma":null,"domain_candidate":null,"domain_consensus":null,"study_design_codex":"observational","study_design_gemma":"observational","study_design_scores_codex":[0.0008466062,0.0004838788,0.9956569,0.00003599111,0.00001778581,0.00002353023,0.0006464504,0.0006427902,0.001374579,0.00009809461,0.00005346169,0.0001199394],"study_design_scores_gemma":[0.0006253449,0.0002377656,0.9921672,0.000008067701,0.0001961972,4.337922e-7,0.00004939456,0.006255513,0.000175575,0.00019575,0.00001825333,0.00007052309],"study_design_candidate":"observational","study_design_consensus":"observational","genre_codex":"empirical","genre_gemma":"empirical","genre_scores_codex":[0.9960127,0.0000813782,0.000002107418,0.002420848,0.00003952277,0.001067528,0.000354028,0.00001106476,0.00001082552],"genre_scores_gemma":[0.9988431,0.00005486708,0.0000108239,0.0009064432,0.00003322869,0.0001054138,0.00002271668,0.00001091794,0.00001243666],"genre_candidate":"empirical","genre_consensus":"empirical","teacher_disagreement_score":0.005612723,"threshold_uncertainty_score":0.3580979,"prediction_status":"machine_predicted_unvalidated"},"labels":[],"label_agreement":null},{"id":"W2163671213","doi":"10.1111/j.1399-0004.2004.00241.x","title":"A new model for prediction of the age of onset and penetrance for Huntington's disease based on CAG length","year":2004,"lang":"en","type":"article","venue":"Clinical Genetics","topic":"Genetic Neurodegenerative Diseases","field":"Neuroscience","cited_by":827,"is_retracted":false,"has_abstract":true,"routes":{"ca_aff":true,"ca_fund":false,"ca_venue":false,"about_ca":false},"ca_institutions":"University of British Columbia","funders":"National Institute of Neurological Disorders and Stroke","keywords":"Penetrance; Age of onset; Cohort; Trinucleotide repeat expansion; Confidence interval; Disease; Huntington's disease; Medicine; Predictive testing; Internal medicine; Pediatrics; Psychology; Genetics; Biology; Allele; Phenotype","retraction":null,"screen_n_in":null,"score":{"opus":0.1074478642820187,"gpt":0.3513736233857898,"spread":0.2439257591037711,"validation_status":"score_only:v0-immature-baseline"},"prediction":{"model_version":"codex-gemma-dda1882f352a","candidate_categories":[],"consensus_categories":[],"category_scores_codex":[0.000159901,0.0001306554,0.0002077821,0.00002812117,0.0000790041,0.00001178521,0.0002519054,0.00005671971,0.000001570814],"category_scores_gemma":[0.002607502,0.0001038302,0.000206327,0.0000961679,0.0002794772,0.00001627664,0.00004592034,0.00007587141,2.877379e-7],"about_ca_system_candidate":false,"about_ca_system_consensus":false,"about_ca_system_score_codex":0.000008221504,"about_ca_system_score_gemma":0.0004151181,"about_ca_topic_candidate":false,"about_ca_topic_consensus":false,"about_ca_topic_score_codex":0.00000148772,"about_ca_topic_score_gemma":0.000005049957,"domain_scores_codex":[0.9986337,0.00008657862,0.0004680206,0.0004227478,0.0002166382,0.000172245],"domain_scores_gemma":[0.9983604,0.0007750181,0.0001927632,0.0004401864,0.00007848035,0.000153165],"domain_codex":null,"domain_gemma":null,"domain_candidate":null,"domain_consensus":null,"study_design_codex":"simulation_or_modeling","study_design_gemma":"bench_or_experimental","study_design_scores_codex":[0.001870964,0.001480029,0.05621383,0.0004472459,0.0000171465,0.000001111936,0.0001574068,0.7558195,0.1754884,0.001097681,0.001219404,0.00618732],"study_design_scores_gemma":[0.003917218,0.001013623,0.2495123,0.0000893778,0.0001489777,1.82433e-7,0.000004646205,0.2910928,0.4480098,0.005810738,0.0002475704,0.000152884],"study_design_candidate":"simulation_or_modeling","study_design_consensus":null,"genre_codex":"empirical","genre_gemma":"empirical","genre_scores_codex":[0.9204741,0.00003967362,0.0766148,0.0004044397,0.0002220793,0.0009616928,0.001254691,0.00001194463,0.00001655592],"genre_scores_gemma":[0.9904083,0.00006066651,0.008196045,0.001063561,0.00007477654,0.0000431536,0.000009199253,0.00002344857,0.0001208302],"genre_candidate":"empirical","genre_consensus":"empirical","teacher_disagreement_score":0.4647268,"threshold_uncertainty_score":0.4234074,"prediction_status":"machine_predicted_unvalidated"},"labels":[],"label_agreement":null},{"id":"W2162229008","doi":"10.1136/jnnp.2007.128728","title":"Detection of Huntington's disease decades before diagnosis: the Predict-HD study","year":2007,"lang":"en","type":"article","venue":"Journal of Neurology Neurosurgery & Psychiatry","topic":"Genetic Neurodegenerative Diseases","field":"Neuroscience","cited_by":812,"is_retracted":false,"has_abstract":true,"routes":{"ca_aff":true,"ca_fund":false,"ca_venue":false,"about_ca":false},"ca_institutions":"University of British Columbia; Centre for Movement Disorders","funders":"National Institute of Neurological Disorders and Stroke; Hereditary Disease Foundation; National Institutes of Health; Cure Huntington's Disease Initiative; Roy J. Carver Charitable Trust; U.S. Public Health Service; Howard Hughes Medical Institute","keywords":"Huntington's disease; Disease; Neuroimaging; Medicine; Psychology; Internal medicine; Psychiatry","retraction":null,"screen_n_in":null,"score":{"opus":0.01628312546467445,"gpt":0.2723356917751127,"spread":0.2560525663104382,"validation_status":"score_only:v0-immature-baseline"},"prediction":{"model_version":"codex-gemma-dda1882f352a","candidate_categories":["metaepi_narrow"],"consensus_categories":[],"category_scores_codex":[0.001181767,0.0003775531,0.0005472838,0.0005661431,0.0003530181,0.00005792051,0.0008513994,0.00009878014,0.00002450349],"category_scores_gemma":[0.002305517,0.0002753429,0.0005073899,0.0008522741,0.0004576042,0.0002670506,0.0001598515,0.0009116259,0.00000666827],"about_ca_system_candidate":false,"about_ca_system_consensus":false,"about_ca_system_score_codex":0.00001183895,"about_ca_system_score_gemma":0.0002398456,"about_ca_topic_candidate":false,"about_ca_topic_consensus":false,"about_ca_topic_score_codex":0.00000325406,"about_ca_topic_score_gemma":0.00003540199,"domain_scores_codex":[0.9950179,0.001253691,0.001547274,0.0006155595,0.0009749148,0.0005905904],"domain_scores_gemma":[0.9954518,0.0015796,0.001660899,0.0006849141,0.0002358488,0.0003869346],"domain_codex":null,"domain_gemma":null,"domain_candidate":null,"domain_consensus":null,"study_design_codex":"observational","study_design_gemma":"observational","study_design_scores_codex":[0.001447088,0.001882621,0.9108248,0.00002832148,0.00003075998,0.0001811114,0.0001494939,0.0003933374,0.0828213,0.00003970403,0.0006915536,0.001509863],"study_design_scores_gemma":[0.0006909679,0.002358019,0.8802174,0.00001963645,0.0002344468,0.0001635114,0.00009172262,0.00002357332,0.114926,0.0004380558,0.0006804487,0.000156181],"study_design_candidate":"observational","study_design_consensus":"observational","genre_codex":"empirical","genre_gemma":"empirical","genre_scores_codex":[0.987672,0.0002394769,0.0001131963,0.003689593,0.00764829,0.0005429849,0.00001931675,0.000044286,0.00003086344],"genre_scores_gemma":[0.9925039,0.0001024946,0.00001491816,0.006381247,0.0008924821,0.0000234657,3.102662e-7,0.00006489841,0.00001630881],"genre_candidate":"empirical","genre_consensus":"empirical","teacher_disagreement_score":0.03210472,"threshold_uncertainty_score":0.9999699,"prediction_status":"machine_predicted_unvalidated"},"labels":[],"label_agreement":null},{"id":"W2170256902","doi":"10.1093/hmg/ddg169","title":"Selective striatal neuronal loss in a YAC128 mouse model of Huntington disease","year":2003,"lang":"en","type":"article","venue":"Human Molecular Genetics","topic":"Genetic Neurodegenerative Diseases","field":"Neuroscience","cited_by":802,"is_retracted":false,"has_abstract":true,"routes":{"ca_aff":true,"ca_fund":true,"ca_venue":false,"about_ca":false},"ca_institutions":"University of British Columbia","funders":"Canadian Institutes of Health Research; Emory University; Hereditary Disease Foundation; Michael Smith Health Research BC; National Science Foundation","keywords":"Huntington's disease; Neurodegeneration; Biology; Neuroscience; Huntingtin; Atrophy; Neuroprotection; Medium spiny neuron; Rotarod performance test; Spinal muscular atrophy; Genetic model; Huntingtin Protein; Phenotype; Striatum; Disease; Basal ganglia; Genetics; Pathology; Central nervous system; Endocrinology; Gene; Medicine; Dopamine; Motor activity","retraction":null,"screen_n_in":null,"score":{"opus":0.02938679871690934,"gpt":0.2696491236805703,"spread":0.2402623249636609,"validation_status":"score_only:v0-immature-baseline"},"prediction":{"model_version":"codex-gemma-dda1882f352a","candidate_categories":["metaepi_narrow"],"consensus_categories":[],"category_scores_codex":[0.00009628237,0.0002895354,0.0002504583,0.0001964176,0.00009839388,0.00003756542,0.0003584269,0.00005606755,0.00002935559],"category_scores_gemma":[0.0004761885,0.0003345033,0.0001392533,0.0003422455,0.0002247642,0.00004886885,0.0001130488,0.0001969521,0.00000977748],"about_ca_system_candidate":false,"about_ca_system_consensus":false,"about_ca_system_score_codex":0.00003725517,"about_ca_system_score_gemma":0.0002339203,"about_ca_topic_candidate":false,"about_ca_topic_consensus":false,"about_ca_topic_score_codex":0.000004282029,"about_ca_topic_score_gemma":0.00000825227,"domain_scores_codex":[0.9974201,0.0004324651,0.0004238993,0.0007356619,0.0005338052,0.0004540526],"domain_scores_gemma":[0.9989139,0.00005161961,0.0001570657,0.0005483261,0.00009297552,0.0002361138],"domain_codex":null,"domain_gemma":null,"domain_candidate":null,"domain_consensus":null,"study_design_codex":"bench_or_experimental","study_design_gemma":"bench_or_experimental","study_design_scores_codex":[0.00003054192,0.000324865,0.004518582,0.00002463387,0.000003946835,0.000007667009,0.00009057439,0.08234312,0.9079452,0.004662431,0.0000155785,0.00003285929],"study_design_scores_gemma":[0.0007975985,0.0001116634,0.007940301,0.00001167875,0.00002683834,3.801935e-7,0.00000939026,0.005149918,0.9821879,0.003467462,0.00001334376,0.0002835104],"study_design_candidate":"bench_or_experimental","study_design_consensus":"bench_or_experimental","genre_codex":"empirical","genre_gemma":"empirical","genre_scores_codex":[0.9961117,0.00005584432,0.002593851,0.0000165022,0.00004904215,0.0004464861,0.0001064673,0.00003140571,0.000588686],"genre_scores_gemma":[0.9986916,0.00002914763,0.000648771,0.0003611326,0.00001429827,0.00003621381,0.000008027396,0.00006596067,0.000144804],"genre_candidate":"empirical","genre_consensus":"empirical","teacher_disagreement_score":0.0771932,"threshold_uncertainty_score":0.9999107,"prediction_status":"machine_predicted_unvalidated"},"labels":[],"label_agreement":null},{"id":"W2592378632","doi":"10.1016/j.jalz.2017.01.014","title":"Consensus classification of posterior cortical atrophy","year":2017,"lang":"en","type":"article","venue":"Alzheimer s & Dementia","topic":"Genetic Neurodegenerative Diseases","field":"Neuroscience","cited_by":662,"is_retracted":false,"has_abstract":true,"routes":{"ca_aff":true,"ca_fund":false,"ca_venue":false,"about_ca":false},"ca_institutions":"University of Toronto; University Health Network","funders":"National Institutes of Health; NIHR Oxford Biomedical Research Centre; Alzheimer Nederland; Brain Research Trust; American College of Radiology; Engineering and Physical Sciences Research Council; National Institute of Neurological Disorders and Stroke; National Institute for Health and Care Research; Economic and Social Research Council; Alzheimer’s Research UK; Medical Research Council; National Institute on Aging; Alzheimer's Association","keywords":"Posterior cortical atrophy; Atrophy; Medicine; Computer science; Pathology; Disease; Dementia","retraction":null,"screen_n_in":null,"score":{"opus":0.08267005740463565,"gpt":0.3191259279106369,"spread":0.2364558705060013,"validation_status":"score_only:v0-immature-baseline"},"prediction":{"model_version":"codex-gemma-dda1882f352a","candidate_categories":[],"consensus_categories":[],"category_scores_codex":[0.00006293401,0.0001246804,0.0001409344,0.00003672404,0.000339448,0.00008300531,0.0004240966,0.00003264632,0.000155666],"category_scores_gemma":[0.0003949265,0.0001202058,0.00007642328,0.0000379985,0.0004456267,0.00006773537,0.0001474041,0.00006854523,0.0001345279],"about_ca_system_candidate":false,"about_ca_system_consensus":false,"about_ca_system_score_codex":0.00000151107,"about_ca_system_score_gemma":0.00004774249,"about_ca_topic_candidate":false,"about_ca_topic_consensus":false,"about_ca_topic_score_codex":0.00001397541,"about_ca_topic_score_gemma":0.000004760552,"domain_scores_codex":[0.998765,0.0001394806,0.0002666951,0.0003715161,0.000253148,0.0002041399],"domain_scores_gemma":[0.9987109,0.00008412098,0.0002643452,0.0007699261,0.00007300585,0.00009767243],"domain_codex":null,"domain_gemma":null,"domain_candidate":null,"domain_consensus":null,"study_design_codex":"bench_or_experimental","study_design_gemma":"bench_or_experimental","study_design_scores_codex":[0.00003668847,0.00007912695,0.009632169,0.000001855004,0.0001735764,0.000008002215,0.00002350575,0.000002094452,0.9835391,0.001450775,0.0003362303,0.004716897],"study_design_scores_gemma":[0.0002281309,0.00006326608,0.2737003,0.000003088523,0.001171616,0.000004266867,0.000007058859,0.0001447345,0.7241996,0.00009307056,0.0002974433,0.00008741684],"study_design_candidate":"bench_or_experimental","study_design_consensus":"bench_or_experimental","genre_codex":"empirical","genre_gemma":"empirical","genre_scores_codex":[0.9960347,0.001083877,0.00004409248,0.0005460418,0.000383271,0.0002231748,0.00003538884,0.00002523188,0.001624186],"genre_scores_gemma":[0.9991869,0.00001096871,0.0004622635,0.0002513767,0.00004311794,0.00001650761,0.000002519607,0.00001692235,0.000009433198],"genre_candidate":"empirical","genre_consensus":"empirical","teacher_disagreement_score":0.2640682,"threshold_uncertainty_score":0.4901852,"prediction_status":"machine_predicted_unvalidated"},"labels":[],"label_agreement":null},{"id":"W2135732941","doi":"10.1016/j.cell.2006.04.026","title":"Cleavage at the Caspase-6 Site Is Required for Neuronal Dysfunction and Degeneration Due to Mutant Huntingtin","year":2006,"lang":"en","type":"article","venue":"Cell","topic":"Genetic Neurodegenerative Diseases","field":"Neuroscience","cited_by":649,"is_retracted":false,"has_abstract":false,"routes":{"ca_aff":true,"ca_fund":false,"ca_venue":false,"about_ca":false},"ca_institutions":"Child and Family Research Institute; University of British Columbia","funders":"","keywords":"Huntingtin; Neurodegeneration; Biology; Excitotoxicity; Proteolysis; Caspase; Cell biology; Huntington's disease; Cleavage (geology); Huntingtin Protein; Caspase 3; Mutant; Molecular biology; Biochemistry; Apoptosis; Programmed cell death; Internal medicine; Gene; Medicine","retraction":null,"screen_n_in":null,"score":{"opus":0.02182544077842376,"gpt":0.2345491712376026,"spread":0.2127237304591788,"validation_status":"score_only:v0-immature-baseline"},"prediction":{"model_version":"codex-gemma-dda1882f352a","candidate_categories":[],"consensus_categories":[],"category_scores_codex":[0.00009108386,0.0001485056,0.000099549,0.00004330256,0.000428316,0.00009972622,0.00009854724,0.00003144898,0.00004786508],"category_scores_gemma":[0.0001351812,0.0001199313,0.00004992358,0.0001434796,0.00006766734,0.00006897169,0.00008814257,0.00004806595,0.00009731786],"about_ca_system_candidate":false,"about_ca_system_consensus":false,"about_ca_system_score_codex":0.00002763861,"about_ca_system_score_gemma":0.00002021157,"about_ca_topic_candidate":false,"about_ca_topic_consensus":false,"about_ca_topic_score_codex":0.00002689551,"about_ca_topic_score_gemma":0.0001067083,"domain_scores_codex":[0.998742,0.0001096314,0.0001847565,0.0005214546,0.0001939467,0.0002481952],"domain_scores_gemma":[0.9993907,0.0001764928,0.000077107,0.0002432221,0.00003759141,0.00007495422],"domain_codex":null,"domain_gemma":null,"domain_candidate":null,"domain_consensus":null,"study_design_codex":"bench_or_experimental","study_design_gemma":"bench_or_experimental","study_design_scores_codex":[0.000056068,0.00003721197,0.0002971455,0.00001134481,4.055851e-7,0.00000535609,0.00008091264,0.0005114706,0.9864817,0.00007118096,0.0121753,0.0002719499],"study_design_scores_gemma":[0.0003732329,0.0001254865,0.006435769,0.00000254895,0.00001766323,0.00001118638,0.00001083057,0.001560328,0.9845886,0.00008653689,0.006657579,0.0001302337],"study_design_candidate":"bench_or_experimental","study_design_consensus":"bench_or_experimental","genre_codex":"empirical","genre_gemma":"empirical","genre_scores_codex":[0.9957616,0.0000397659,0.001384904,0.001030784,0.0002950471,0.0005970668,0.0001246344,0.00003877078,0.0007274264],"genre_scores_gemma":[0.9925066,0.000004240138,0.0001609894,0.003565392,0.0002389931,0.00007559305,0.00001889952,0.00002806919,0.003401253],"genre_candidate":"empirical","genre_consensus":"empirical","teacher_disagreement_score":0.006138623,"threshold_uncertainty_score":0.4890656,"prediction_status":"machine_predicted_unvalidated"},"labels":[],"label_agreement":null},{"id":"W2115999215","doi":"10.1084/jem.20080178","title":"A novel pathogenic pathway of immune activation detectable before clinical onset in Huntington's disease","year":2008,"lang":"en","type":"article","venue":"The Journal of Experimental Medicine","topic":"Genetic Neurodegenerative Diseases","field":"Neuroscience","cited_by":641,"is_retracted":false,"has_abstract":true,"routes":{"ca_aff":true,"ca_fund":false,"ca_venue":false,"about_ca":false},"ca_institutions":"Child and Family Research Institute; University of British Columbia","funders":"National Institute for Health and Care Research; Wellcome Trust","keywords":"Immune system; Huntington's disease; Microglia; Huntingtin; Biology; Immunology; Innate immune system; Neuroimmunology; Inflammation; Disease; Medicine; Pathology","retraction":null,"screen_n_in":null,"score":{"opus":0.05972563675676124,"gpt":0.3328210045367076,"spread":0.2730953677799464,"validation_status":"score_only:v0-immature-baseline"},"prediction":{"model_version":"codex-gemma-dda1882f352a","candidate_categories":[],"consensus_categories":[],"category_scores_codex":[0.0006521677,0.0001494547,0.0003605174,0.0001354612,0.0001002654,0.00000318224,0.0003663305,0.00002954895,0.00008313968],"category_scores_gemma":[0.001983918,0.00009183028,0.0001265602,0.0002895328,0.0005413472,0.0001313534,0.0000829022,0.0002494493,0.000002385993],"about_ca_system_candidate":false,"about_ca_system_consensus":false,"about_ca_system_score_codex":0.00004353565,"about_ca_system_score_gemma":0.0001669753,"about_ca_topic_candidate":false,"about_ca_topic_consensus":false,"about_ca_topic_score_codex":0.00002350646,"about_ca_topic_score_gemma":0.000001681491,"domain_scores_codex":[0.9977341,0.0003923234,0.0009609042,0.000160795,0.0005589755,0.000192874],"domain_scores_gemma":[0.9983715,0.0004364606,0.0006849976,0.0002671668,0.00007834698,0.0001615361],"domain_codex":null,"domain_gemma":null,"domain_candidate":null,"domain_consensus":null,"study_design_codex":"bench_or_experimental","study_design_gemma":"bench_or_experimental","study_design_scores_codex":[0.0007359892,0.0006136277,0.005637667,0.000006119576,0.000006027369,0.00001919885,0.001351031,0.00005210599,0.9912707,0.00001817994,0.00004026259,0.0002491412],"study_design_scores_gemma":[0.002029427,0.001135783,0.1079868,0.000135595,0.00001779913,0.00008011321,0.0004818781,0.00006119149,0.8879452,0.00003725214,0.0000240506,0.00006485414],"study_design_candidate":"bench_or_experimental","study_design_consensus":"bench_or_experimental","genre_codex":"empirical","genre_gemma":"empirical","genre_scores_codex":[0.9983003,0.0005503359,0.0001362125,0.000430059,0.0002998235,0.0002166748,0.000009127582,0.000005555139,0.00005192428],"genre_scores_gemma":[0.9990841,0.0001148556,0.0001021081,0.0004529618,0.0001843528,0.000004863427,0.000001061631,0.00001766369,0.00003801227],"genre_candidate":"empirical","genre_consensus":"empirical","teacher_disagreement_score":0.1033254,"threshold_uncertainty_score":0.374473,"prediction_status":"machine_predicted_unvalidated"},"labels":[],"label_agreement":null},{"id":"W2129250133","doi":"10.1016/s1474-4422(10)70276-3","title":"Biological and clinical changes in premanifest and early stage Huntington's disease in the TRACK-HD study: the 12-month longitudinal analysis","year":2010,"lang":"en","type":"article","venue":"The Lancet Neurology","topic":"Genetic Neurodegenerative Diseases","field":"Neuroscience","cited_by":600,"is_retracted":false,"has_abstract":false,"routes":{"ca_aff":true,"ca_fund":false,"ca_venue":false,"about_ca":false},"ca_institutions":"University of British Columbia","funders":"Cardiff University; National Institute for Health and Care Research","keywords":"Medicine; Huntington's disease; Demographics; Atrophy; Observational study; Internal medicine; Disease; Longitudinal study; Demography; Pathology","retraction":null,"screen_n_in":null,"score":{"opus":0.1044159682265204,"gpt":0.3500308996249813,"spread":0.2456149313984609,"validation_status":"score_only:v0-immature-baseline"},"prediction":{"model_version":"codex-gemma-dda1882f352a","candidate_categories":[],"consensus_categories":[],"category_scores_codex":[0.001414139,0.0002001973,0.0004160168,0.00009848327,0.0002063982,0.0001042121,0.0007508512,0.00006622174,0.00002421218],"category_scores_gemma":[0.001613332,0.00009471773,0.00006475027,0.0004193424,0.00100748,0.00004873304,0.0002983183,0.0008427583,0.00000380357],"about_ca_system_candidate":false,"about_ca_system_consensus":false,"about_ca_system_score_codex":0.000001794876,"about_ca_system_score_gemma":0.00001904326,"about_ca_topic_candidate":false,"about_ca_topic_consensus":false,"about_ca_topic_score_codex":0.0001103358,"about_ca_topic_score_gemma":0.01147579,"domain_scores_codex":[0.99549,0.002839084,0.0003031167,0.0007406722,0.0002097293,0.000417419],"domain_scores_gemma":[0.9964876,0.002550711,0.0001332457,0.0007292267,0.00001518762,0.00008408735],"domain_codex":null,"domain_gemma":null,"domain_candidate":null,"domain_consensus":null,"study_design_codex":"observational","study_design_gemma":"observational","study_design_scores_codex":[0.0003732319,0.000300311,0.9959996,0.000002439546,0.00001311119,0.000124698,0.0005012303,0.0000386545,0.002074619,0.0002544265,0.00001088157,0.0003068048],"study_design_scores_gemma":[0.00063983,0.0006412103,0.9974132,7.947982e-7,0.0001116174,0.000006825417,0.00006627299,0.0003910624,0.0002186262,0.0002143346,0.0001981463,0.00009808163],"study_design_candidate":"observational","study_design_consensus":"observational","genre_codex":"empirical","genre_gemma":"empirical","genre_scores_codex":[0.9856387,0.00004745422,0.000001421796,0.01331628,0.0001458611,0.0007455208,0.00002577123,0.0000184962,0.00006044728],"genre_scores_gemma":[0.9957772,0.0001927305,0.000003199689,0.003684079,0.0002302183,0.0000832959,9.411299e-7,0.00001048076,0.0000178938],"genre_candidate":"empirical","genre_consensus":"empirical","teacher_disagreement_score":0.01136545,"threshold_uncertainty_score":0.6403759,"prediction_status":"machine_predicted_unvalidated"},"labels":[],"label_agreement":null},{"id":"W1996592124","doi":"10.1016/s0896-6273(02)00615-3","title":"Increased Sensitivity to N-Methyl-D-Aspartate Receptor-Mediated Excitotoxicity in a Mouse Model of Huntington's Disease","year":2002,"lang":"en","type":"article","venue":"Neuron","topic":"Genetic Neurodegenerative Diseases","field":"Neuroscience","cited_by":594,"is_retracted":false,"has_abstract":false,"routes":{"ca_aff":true,"ca_fund":false,"ca_venue":false,"about_ca":false},"ca_institutions":"University of British Columbia; Pacific Centre for Reproductive Medicine","funders":"","keywords":"Excitotoxicity; NMDA receptor; Huntington's disease; Huntingtin; Glutamate receptor; AMPA receptor; Neuroscience; Programmed cell death; Biology; Quinolinate; Genetically modified mouse; Neurodegeneration; Medium spiny neuron; Quinolinic acid; Neuron; Receptor; Transgene; Cell biology; Internal medicine; Central nervous system; Basal ganglia; Apoptosis; Biochemistry; Medicine; Disease; Gene","retraction":null,"screen_n_in":null,"score":{"opus":0.03779730953330144,"gpt":0.2517387210980842,"spread":0.2139414115647828,"validation_status":"score_only:v0-immature-baseline"},"prediction":{"model_version":"codex-gemma-dda1882f352a","candidate_categories":["metaepi_narrow"],"consensus_categories":[],"category_scores_codex":[0.0001840086,0.0003025072,0.0003400175,0.0002675781,0.00008281498,0.00003522205,0.0002557734,0.00005183707,0.00008060045],"category_scores_gemma":[0.004574401,0.000323689,0.0001073293,0.0006131039,0.0001325965,0.0001633535,0.0001666666,0.0002190639,0.0001668889],"about_ca_system_candidate":false,"about_ca_system_consensus":false,"about_ca_system_score_codex":0.00004059521,"about_ca_system_score_gemma":0.00005144559,"about_ca_topic_candidate":false,"about_ca_topic_consensus":false,"about_ca_topic_score_codex":0.00007878988,"about_ca_topic_score_gemma":0.00004874005,"domain_scores_codex":[0.996805,0.0008548354,0.0004082482,0.0008850095,0.0005387351,0.0005081946],"domain_scores_gemma":[0.9981548,0.0005123174,0.0001509441,0.0005947503,0.00007544777,0.0005116949],"domain_codex":null,"domain_gemma":null,"domain_candidate":null,"domain_consensus":null,"study_design_codex":"bench_or_experimental","study_design_gemma":"bench_or_experimental","study_design_scores_codex":[0.0001522565,0.0005327764,0.004179507,0.00002869781,0.000001222395,0.00001584231,0.0001591794,0.02046661,0.9738952,0.00003086976,0.0003127501,0.0002250852],"study_design_scores_gemma":[0.0005573086,0.00009208213,0.01160942,0.00001623924,0.000004990251,2.906167e-7,0.000006416755,0.09054378,0.8968498,0.00002592249,0.00004734553,0.0002464186],"study_design_candidate":"bench_or_experimental","study_design_consensus":"bench_or_experimental","genre_codex":"empirical","genre_gemma":"empirical","genre_scores_codex":[0.9979692,0.000005933088,0.0001424301,0.0003248572,0.0001110082,0.0006422296,0.0004627054,0.0001146459,0.0002270173],"genre_scores_gemma":[0.9970713,0.00003356092,0.0002298016,0.002163625,0.00003493538,0.00005768569,0.000006487905,0.00005345807,0.0003491093],"genre_candidate":"empirical","genre_consensus":"empirical","teacher_disagreement_score":0.07704541,"threshold_uncertainty_score":0.9999215,"prediction_status":"machine_predicted_unvalidated"},"labels":[],"label_agreement":null},{"id":"W2087157508","doi":"10.1002/mds.25075","title":"The incidence and prevalence of Huntington's disease: A systematic review and meta‐analysis","year":2012,"lang":"en","type":"review","venue":"Movement Disorders","topic":"Genetic Neurodegenerative Diseases","field":"Neuroscience","cited_by":587,"is_retracted":false,"has_abstract":true,"routes":{"ca_aff":true,"ca_fund":false,"ca_venue":false,"about_ca":false},"ca_institutions":"University of Toronto; University of Calgary","funders":"","keywords":"Incidence (geometry); Meta-analysis; Medicine; Epidemiology; Chorea; Confidence interval; Dementia; Prevalence; Demography; Huntington's disease; Disease; Pediatrics; Internal medicine","retraction":null,"screen_n_in":null,"score":{"opus":0.0516033668269791,"gpt":0.3207641677742643,"spread":0.2691608009472852,"validation_status":"score_only:v0-immature-baseline"},"prediction":{"model_version":"codex-gemma-dda1882f352a","candidate_categories":["metaepi_narrow"],"consensus_categories":[],"category_scores_codex":[0.0006088207,0.0004929611,0.002738584,0.0001335131,0.0002268664,0.00008025445,0.0005842892,0.0000401173,0.0000667426],"category_scores_gemma":[0.001008358,0.0002828781,0.0009243134,0.0007040941,0.0003612529,0.0001037796,0.0003804244,0.0001288518,0.000005356884],"about_ca_system_candidate":false,"about_ca_system_consensus":false,"about_ca_system_score_codex":0.00001817361,"about_ca_system_score_gemma":0.00006921418,"about_ca_topic_candidate":false,"about_ca_topic_consensus":false,"about_ca_topic_score_codex":0.000008054396,"about_ca_topic_score_gemma":0.00001506371,"domain_scores_codex":[0.9962794,0.00122762,0.0008868921,0.000707685,0.0005560216,0.0003423901],"domain_scores_gemma":[0.9968374,0.00102287,0.0009642315,0.0009362187,0.00004422685,0.0001950426],"domain_codex":null,"domain_gemma":null,"domain_candidate":null,"domain_consensus":null,"study_design_codex":"systematic_review","study_design_gemma":"meta_analysis","study_design_scores_codex":[8.597071e-7,0.00004827199,0.00004155402,0.9920452,0.00607269,2.758606e-7,0.00001540162,9.242824e-7,0.000002017073,0.0003309369,0.00002623598,0.001415635],"study_design_scores_gemma":[0.00003812275,0.00002120743,0.00006469958,0.01901647,0.9771105,1.331287e-7,0.000009656107,0.000005027098,0.00002218665,0.0002795008,0.003079621,0.000352881],"study_design_candidate":"systematic_review","study_design_consensus":null,"genre_codex":"review","genre_gemma":"review","genre_scores_codex":[0.00001989836,0.9956578,0.00002357401,0.0002297784,0.00002837456,0.003778907,0.0002138968,0.0000177177,0.00003006398],"genre_scores_gemma":[0.0004538397,0.9963121,0.00001085851,0.001170398,0.0000051467,0.001457213,0.000004162236,0.0000313557,0.0005548786],"genre_candidate":"review","genre_consensus":"review","teacher_disagreement_score":0.9730287,"threshold_uncertainty_score":0.9999623,"prediction_status":"machine_predicted_unvalidated"},"labels":[],"label_agreement":null},{"id":"W2008584420","doi":"10.1038/nm.2313","title":"Mutant huntingtin binds the mitochondrial fission GTPase dynamin-related protein-1 and increases its enzymatic activity","year":2011,"lang":"en","type":"article","venue":"Nature Medicine","topic":"Genetic Neurodegenerative Diseases","field":"Neuroscience","cited_by":544,"is_retracted":false,"has_abstract":false,"routes":{"ca_aff":true,"ca_fund":true,"ca_venue":false,"about_ca":false},"ca_institutions":"McGill University; University of British Columbia","funders":"National Eye Institute; Canadian Institutes of Health Research; University of California, Los Angeles; National Institutes of Health; National Center for Research Resources; National Institute of Neurological Disorders and Stroke; University of Ottawa; University of California, Irvine; CHDI Foundation","keywords":"Huntingtin; Huntingtin Protein; Mitochondrial fission; Neurodegeneration; Huntington's disease; Biology; Cell biology; Mitochondrion; GTPase; Mutant; Genetics; Disease; Medicine; Internal medicine; Gene","retraction":null,"screen_n_in":null,"score":{"opus":0.02328208117488196,"gpt":0.263803529589483,"spread":0.240521448414601,"validation_status":"score_only:v0-immature-baseline"},"prediction":{"model_version":"codex-gemma-dda1882f352a","candidate_categories":[],"consensus_categories":[],"category_scores_codex":[0.0002288602,0.0002937581,0.0002920589,0.000131325,0.0003107794,0.00002318464,0.000344278,0.0001981864,0.0002994832],"category_scores_gemma":[0.0074998,0.0001650052,0.0000509121,0.0004708887,0.0004160189,0.0001132054,0.0001501892,0.0007660689,0.00002060768],"about_ca_system_candidate":false,"about_ca_system_consensus":false,"about_ca_system_score_codex":0.00002253687,"about_ca_system_score_gemma":0.00006847045,"about_ca_topic_candidate":false,"about_ca_topic_consensus":false,"about_ca_topic_score_codex":0.0000501022,"about_ca_topic_score_gemma":0.00001535966,"domain_scores_codex":[0.9976627,0.0005228096,0.0002838156,0.0006027921,0.0005991847,0.0003286725],"domain_scores_gemma":[0.9986073,0.0004304649,0.0002197758,0.0004370587,0.00008622147,0.0002191542],"domain_codex":null,"domain_gemma":null,"domain_candidate":null,"domain_consensus":null,"study_design_codex":"bench_or_experimental","study_design_gemma":"bench_or_experimental","study_design_scores_codex":[0.0001717753,0.0001619005,0.0001887795,0.00004185022,0.000006796103,0.00006138131,0.0004808592,7.044519e-7,0.9964308,0.0004484832,0.0002340089,0.001772711],"study_design_scores_gemma":[0.001267235,0.0003613753,0.01366451,0.0001869253,0.00007461936,0.00005223375,0.00006801442,0.0005651735,0.982878,0.0004407775,0.0002571872,0.0001839357],"study_design_candidate":"bench_or_experimental","study_design_consensus":"bench_or_experimental","genre_codex":"empirical","genre_gemma":"empirical","genre_scores_codex":[0.9950862,0.0004764728,0.00002612763,0.001651003,0.0005721203,0.001034001,0.00002417881,0.00009174483,0.001038147],"genre_scores_gemma":[0.9980965,0.00002948507,0.00008739355,0.0009206262,0.0002390066,0.00006382332,0.000004141252,0.00003068827,0.0005282859],"genre_candidate":"empirical","genre_consensus":"empirical","teacher_disagreement_score":0.01355274,"threshold_uncertainty_score":0.8978503,"prediction_status":"machine_predicted_unvalidated"},"labels":[],"label_agreement":null},{"id":"W2032120877","doi":"10.1016/s1474-4422(11)70263-0","title":"Potential endpoints for clinical trials in premanifest and early Huntington's disease in the TRACK-HD study: analysis of 24 month observational data","year":2011,"lang":"en","type":"article","venue":"The Lancet Neurology","topic":"Genetic Neurodegenerative Diseases","field":"Neuroscience","cited_by":540,"is_retracted":false,"has_abstract":false,"routes":{"ca_aff":true,"ca_fund":false,"ca_venue":false,"about_ca":false},"ca_institutions":"University of British Columbia","funders":"National Institute for Health and Care Research","keywords":"Huntington's disease; Observational study; Medicine; Atrophy; Clinical trial; White matter; Biomarker; Internal medicine; Psychology; Disease; Magnetic resonance imaging; Biology","retraction":null,"screen_n_in":null,"score":{"opus":0.5240058223250217,"gpt":0.4476743832643977,"spread":0.076331439060624,"validation_status":"score_only:v0-immature-baseline"},"prediction":{"model_version":"codex-gemma-dda1882f352a","candidate_categories":["metaresearch"],"consensus_categories":[],"category_scores_codex":[0.005353115,0.0001437078,0.0007699545,0.0001459807,0.0000699213,0.00002835718,0.001220787,0.00004242896,0.00002000469],"category_scores_gemma":[0.01073087,0.00008964623,0.0001254365,0.0004456583,0.0002734972,0.0001076127,0.0002870446,0.0002353804,0.000001021819],"about_ca_system_candidate":false,"about_ca_system_consensus":false,"about_ca_system_score_codex":0.000001886929,"about_ca_system_score_gemma":0.00005233075,"about_ca_topic_candidate":false,"about_ca_topic_consensus":false,"about_ca_topic_score_codex":0.00009541765,"about_ca_topic_score_gemma":0.0004426284,"domain_scores_codex":[0.991686,0.006197295,0.0009038626,0.0006863661,0.000254252,0.0002722143],"domain_scores_gemma":[0.9935043,0.004930038,0.0003975158,0.001080382,0.00003374268,0.00005403103],"domain_codex":null,"domain_gemma":null,"domain_candidate":null,"domain_consensus":null,"study_design_codex":"observational","study_design_gemma":"observational","study_design_scores_codex":[0.002217524,0.0008453259,0.9931465,0.000009074882,0.00004493757,0.00003297909,0.000649207,0.000248283,0.001984148,0.0004246889,0.0001491679,0.0002481178],"study_design_scores_gemma":[0.001456562,0.0003990786,0.9926203,0.000001731509,0.0004695958,7.698823e-7,0.00003333862,0.003630138,0.0004578617,0.0008147362,0.00004348994,0.00007242068],"study_design_candidate":"observational","study_design_consensus":"observational","genre_codex":"empirical","genre_gemma":"empirical","genre_scores_codex":[0.996192,0.00003586778,0.00003871716,0.001897916,0.0002073635,0.00120823,0.0003856717,0.00001040602,0.00002377172],"genre_scores_gemma":[0.9973153,0.00006290236,0.00004422168,0.002280603,0.0001608591,0.00009957662,0.00001697257,0.00001180286,0.0000077353],"genre_candidate":"empirical","genre_consensus":"empirical","teacher_disagreement_score":0.005377756,"threshold_uncertainty_score":0.9976022,"prediction_status":"machine_predicted_unvalidated"},"labels":[],"label_agreement":null},{"id":"W2125766898","doi":"10.1016/s1474-4422(12)70327-7","title":"The natural history of multiple system atrophy: a prospective European cohort study","year":2013,"lang":"en","type":"article","venue":"The Lancet Neurology","topic":"Genetic Neurodegenerative Diseases","field":"Neuroscience","cited_by":535,"is_retracted":false,"has_abstract":true,"routes":{"ca_aff":false,"ca_fund":true,"ca_venue":false,"about_ca":false},"ca_institutions":"","funders":"Allergan; National Institutes of Health; Deutsche Parkinson Vereinigung; Ipsen; H. Lundbeck A/S; Uniklinikum Giessen und Marburg; Valeant Pharmaceuticals International; Alzheimer Forschung Initiative; Eli Lilly and Company; AstraZeneca; CSL Behring; Merz Pharmaceuticals; Teva Pharmaceutical Industries; Austrian Science Fund; International Parkinson and Movement Disorder Society; CSL Behring Foundation for Research and Advancement of Patient Health; Oesterreichische Nationalbank; GlaxoSmithKline","keywords":"Natural history; Prospective cohort study; Atrophy; Medicine; Natural (archaeology); Cohort; Cohort study; Natural history study; Geography; Pathology; Internal medicine; Archaeology","retraction":null,"screen_n_in":null,"score":{"opus":0.02275047277842667,"gpt":0.2331236195857679,"spread":0.2103731468073412,"validation_status":"score_only:v0-immature-baseline"},"prediction":{"model_version":"codex-gemma-dda1882f352a","candidate_categories":[],"consensus_categories":[],"category_scores_codex":[0.0003273902,0.0001881948,0.0003027473,0.000032469,0.0002179962,0.0000278126,0.0009728214,0.00001931166,0.0000168924],"category_scores_gemma":[0.0006943882,0.0001027104,0.00007597191,0.0001171838,0.0005673634,0.00005274911,0.000274762,0.000346545,0.0002102388],"about_ca_system_candidate":false,"about_ca_system_consensus":false,"about_ca_system_score_codex":0.00005330809,"about_ca_system_score_gemma":0.00005090812,"about_ca_topic_candidate":false,"about_ca_topic_consensus":false,"about_ca_topic_score_codex":0.00009372315,"about_ca_topic_score_gemma":0.00003362955,"domain_scores_codex":[0.9959408,0.002591291,0.0002825231,0.0005050093,0.0002887035,0.0003916785],"domain_scores_gemma":[0.9978628,0.0008409383,0.0002138919,0.0009326552,0.00009863449,0.00005113795],"domain_codex":null,"domain_gemma":null,"domain_candidate":null,"domain_consensus":null,"study_design_codex":"observational","study_design_gemma":"observational","study_design_scores_codex":[0.0005385294,0.0002862592,0.5787762,0.00002505813,0.0000474101,0.00008667541,0.001283533,0.0002542569,0.4053271,0.00204878,0.01086682,0.0004594362],"study_design_scores_gemma":[0.0008047908,0.000540736,0.9831907,0.000002190983,0.00003272959,0.00003753164,0.00009534995,0.001169233,0.01195427,0.00006231348,0.00199218,0.0001180062],"study_design_candidate":"observational","study_design_consensus":"observational","genre_codex":"empirical","genre_gemma":"empirical","genre_scores_codex":[0.9912851,0.0002041996,0.000005527176,0.0008831693,0.001393856,0.001835742,0.000007932971,0.0001109041,0.004273544],"genre_scores_gemma":[0.9973536,0.00001481508,0.000005421189,0.001485377,0.0002619622,0.0002248867,4.575274e-7,0.00003400757,0.0006194313],"genre_candidate":"empirical","genre_consensus":"empirical","teacher_disagreement_score":0.4044145,"threshold_uncertainty_score":0.4188407,"prediction_status":"machine_predicted_unvalidated"},"labels":[],"label_agreement":null},{"id":"W2013963839","doi":"10.1016/j.cell.2012.06.041","title":"Transcriptome-wide Regulation of Pre-mRNA Splicing and mRNA Localization by Muscleblind Proteins","year":2012,"lang":"en","type":"article","venue":"Cell","topic":"Genetic Neurodegenerative Diseases","field":"Neuroscience","cited_by":516,"is_retracted":false,"has_abstract":false,"routes":{"ca_aff":true,"ca_fund":false,"ca_venue":false,"about_ca":false},"ca_institutions":"Université de Montréal; Montreal Clinical Research Institute","funders":"National Institute of General Medical Sciences; National Human Genome Research Institute; Koch Institute for Integrative Cancer Research, Massachusetts Institute of Technology","keywords":"Biology; RNA splicing; Transcriptome; Alternative splicing; Exon; Messenger RNA; Cell biology; Myotonic dystrophy; RNA-binding protein; RNA; Translation (biology); Genetics; Gene; Gene expression","retraction":null,"screen_n_in":null,"score":{"opus":0.019080415069346,"gpt":0.2404734894536665,"spread":0.2213930743843205,"validation_status":"score_only:v0-immature-baseline"},"prediction":{"model_version":"codex-gemma-dda1882f352a","candidate_categories":[],"consensus_categories":[],"category_scores_codex":[0.00008110326,0.0001080594,0.0001038112,0.00005621596,0.00007809021,0.00001994294,0.00008172988,0.00003803484,0.00004682648],"category_scores_gemma":[0.0001144694,0.000105828,0.0000277591,0.0001703745,0.00008298555,0.0001871156,0.00002059684,0.00004822414,0.000007008225],"about_ca_system_candidate":false,"about_ca_system_consensus":false,"about_ca_system_score_codex":0.00001107395,"about_ca_system_score_gemma":0.00001322313,"about_ca_topic_candidate":false,"about_ca_topic_consensus":false,"about_ca_topic_score_codex":0.000009186495,"about_ca_topic_score_gemma":0.000001271656,"domain_scores_codex":[0.9990752,0.0001136108,0.0001873567,0.0002338112,0.0001972777,0.0001927572],"domain_scores_gemma":[0.9995332,0.00005904877,0.00009922041,0.0001735913,0.00002930938,0.000105624],"domain_codex":null,"domain_gemma":null,"domain_candidate":null,"domain_consensus":null,"study_design_codex":"bench_or_experimental","study_design_gemma":"bench_or_experimental","study_design_scores_codex":[0.00001795252,0.0001158356,0.005279144,0.00005183164,7.202306e-7,1.397697e-7,0.0005004458,0.0003111048,0.992916,0.00005912035,0.0004336429,0.00031408],"study_design_scores_gemma":[0.0003071048,0.00005270272,0.03195424,0.00001322139,0.00001511301,7.717057e-7,0.00001925592,0.001082551,0.9651898,0.00004837332,0.001220074,0.00009675263],"study_design_candidate":"bench_or_experimental","study_design_consensus":"bench_or_experimental","genre_codex":"empirical","genre_gemma":"empirical","genre_scores_codex":[0.9887051,0.0002903712,0.009610576,0.00004519109,0.0001049367,0.0004371958,0.00001772739,0.00002524437,0.0007636742],"genre_scores_gemma":[0.9990312,0.00003629036,0.0001774419,0.0002630709,0.00003873037,0.00001736522,0.000006606255,0.00001924529,0.0004100661],"genre_candidate":"empirical","genre_consensus":"empirical","teacher_disagreement_score":0.02772614,"threshold_uncertainty_score":0.4315539,"prediction_status":"machine_predicted_unvalidated"},"labels":[],"label_agreement":null},{"id":"W2063690295","doi":"10.1016/j.neuron.2010.01.008","title":"Early Increase in Extrasynaptic NMDA Receptor Signaling and Expression Contributes to Phenotype Onset in Huntington's Disease Mice","year":2010,"lang":"en","type":"article","venue":"Neuron","topic":"Genetic Neurodegenerative Diseases","field":"Neuroscience","cited_by":512,"is_retracted":false,"has_abstract":false,"routes":{"ca_aff":true,"ca_fund":true,"ca_venue":false,"about_ca":false},"ca_institutions":"Child and Family Research Institute; University of British Columbia","funders":"Canadian Institutes of Health Research; Michael Smith Health Research BC","keywords":"Excitotoxicity; Huntingtin; NMDA receptor; Memantine; Neuroprotection; CREB; Neuroscience; Biology; Phenotype; Pathogenesis; Transcription factor; Receptor; Mutant; Genetics; Gene; Immunology","retraction":null,"screen_n_in":null,"score":{"opus":0.01244927211205616,"gpt":0.2467401378587457,"spread":0.2342908657466896,"validation_status":"score_only:v0-immature-baseline"},"prediction":{"model_version":"codex-gemma-dda1882f352a","candidate_categories":[],"consensus_categories":[],"category_scores_codex":[0.0001628811,0.0002246527,0.0002053142,0.0002550657,0.00009497683,0.0001089064,0.0002628415,0.00005561916,0.00006139617],"category_scores_gemma":[0.004277341,0.0002283435,0.00003297061,0.0003915786,0.0001008075,0.0001632934,0.0001743289,0.0003552731,0.00005014236],"about_ca_system_candidate":false,"about_ca_system_consensus":false,"about_ca_system_score_codex":0.00001835774,"about_ca_system_score_gemma":0.00007000211,"about_ca_topic_candidate":false,"about_ca_topic_consensus":false,"about_ca_topic_score_codex":0.0001045193,"about_ca_topic_score_gemma":0.00009804625,"domain_scores_codex":[0.9979115,0.0003788747,0.000286871,0.0007762521,0.0002343296,0.0004121634],"domain_scores_gemma":[0.9985877,0.000645939,0.00007221197,0.0003178557,0.00003408563,0.0003421598],"domain_codex":null,"domain_gemma":null,"domain_candidate":null,"domain_consensus":null,"study_design_codex":"bench_or_experimental","study_design_gemma":"bench_or_experimental","study_design_scores_codex":[0.0003000915,0.000149651,0.08714255,0.0000170502,3.869086e-7,0.00001977495,0.0001016319,0.0001107137,0.9116744,0.00006542337,0.00007782519,0.0003405219],"study_design_scores_gemma":[0.0005415099,0.00006072156,0.3535842,0.00004676894,0.000004167263,3.84304e-7,0.000009979974,0.0001296271,0.6451873,0.0001202791,0.0001614497,0.000153694],"study_design_candidate":"bench_or_experimental","study_design_consensus":"bench_or_experimental","genre_codex":"empirical","genre_gemma":"empirical","genre_scores_codex":[0.9984409,0.0000292463,0.00001915587,0.0004746151,0.0003228782,0.0005622788,0.00005555917,0.00005071595,0.00004463374],"genre_scores_gemma":[0.9981264,0.00001060359,0.000205664,0.001446418,0.00004870535,0.00007433753,0.000004166554,0.0000360889,0.00004760237],"genre_candidate":"empirical","genre_consensus":"empirical","teacher_disagreement_score":0.2664871,"threshold_uncertainty_score":0.9311576,"prediction_status":"machine_predicted_unvalidated"},"labels":[],"label_agreement":null},{"id":"W2150852495","doi":"10.1128/mcb.24.18.8195-8209.2004","title":"Mutant Huntingtin Impairs Axonal Trafficking in Mammalian Neurons In Vivo and In Vitro","year":2004,"lang":"en","type":"article","venue":"Molecular and Cellular Biology","topic":"Genetic Neurodegenerative Diseases","field":"Neuroscience","cited_by":506,"is_retracted":false,"has_abstract":true,"routes":{"ca_aff":true,"ca_fund":true,"ca_venue":false,"about_ca":false},"ca_institutions":"University of British Columbia; McGill University; Montreal Neurological Institute and Hospital","funders":"National Institute of Diabetes and Digestive and Kidney Diseases; National Institute of Neurological Disorders and Stroke; National Institute of Mental Health; Genome Canada; Canadian Institutes of Health Research; Mayo Foundation for Medical Education and Research; National Science Foundation","keywords":"Huntingtin; Biology; Cell biology; Huntingtin Protein; Mitochondrion; Huntington's disease; Neurodegeneration; Axoplasmic transport; Mutant; Genetics; Gene; Disease","retraction":null,"screen_n_in":null,"score":{"opus":0.01141123364414159,"gpt":0.2306867063152468,"spread":0.2192754726711052,"validation_status":"score_only:v0-immature-baseline"},"prediction":{"model_version":"codex-gemma-dda1882f352a","candidate_categories":[],"consensus_categories":[],"category_scores_codex":[0.000129795,0.0001810493,0.0002161932,0.0002415535,0.00003948934,0.00002459163,0.0001204936,0.0000826776,0.000009039136],"category_scores_gemma":[0.0001499489,0.000184401,0.00003601871,0.0002789359,0.0001890845,0.00002978001,0.00009861307,0.0001875355,0.000001839405],"about_ca_system_candidate":false,"about_ca_system_consensus":false,"about_ca_system_score_codex":0.00002315732,"about_ca_system_score_gemma":0.00004445079,"about_ca_topic_candidate":false,"about_ca_topic_consensus":false,"about_ca_topic_score_codex":0.0001006332,"about_ca_topic_score_gemma":0.0001534487,"domain_scores_codex":[0.9983343,0.0003137881,0.0002580206,0.0006123962,0.00007639988,0.0004050556],"domain_scores_gemma":[0.9996762,0.0000482095,0.00004333833,0.0001426267,0.000006499837,0.00008308199],"domain_codex":null,"domain_gemma":null,"domain_candidate":null,"domain_consensus":null,"study_design_codex":"bench_or_experimental","study_design_gemma":"bench_or_experimental","study_design_scores_codex":[0.00005745032,0.00009887197,0.007816865,0.000009978616,7.278942e-7,0.0006429157,0.0001438051,0.0005593364,0.9898295,0.0004101021,4.324753e-7,0.0004300567],"study_design_scores_gemma":[0.001141002,0.00009222337,0.007426057,0.00001184189,0.000003378242,0.00003653948,0.00004707684,0.0003201738,0.9902316,0.0004575026,0.00005985545,0.0001726939],"study_design_candidate":"bench_or_experimental","study_design_consensus":"bench_or_experimental","genre_codex":"empirical","genre_gemma":"empirical","genre_scores_codex":[0.9987445,0.0003195367,0.0002226574,0.0002968267,0.00006275723,0.0002304505,0.0000142077,0.00001391569,0.00009516312],"genre_scores_gemma":[0.999067,0.00002500121,0.0001062548,0.0007436572,0.00001082931,0.00002085186,0.000004091462,0.00001737728,0.000004908607],"genre_candidate":"empirical","genre_consensus":"empirical","teacher_disagreement_score":0.001341904,"threshold_uncertainty_score":0.7519655,"prediction_status":"machine_predicted_unvalidated"},"labels":[],"label_agreement":null},{"id":"W2154778779","doi":"10.1016/s0896-6273(03)00366-0","title":"Huntingtin and Huntingtin-Associated Protein 1 Influence Neuronal Calcium Signaling Mediated by Inositol-(1,4,5) Triphosphate Receptor Type 1","year":2003,"lang":"en","type":"article","venue":"Neuron","topic":"Genetic Neurodegenerative Diseases","field":"Neuroscience","cited_by":476,"is_retracted":false,"has_abstract":false,"routes":{"ca_aff":true,"ca_fund":false,"ca_venue":false,"about_ca":false},"ca_institutions":"University of British Columbia; Children's & Women's Health Centre of British Columbia","funders":"National Institute of Neurological Disorders and Stroke","keywords":"Huntingtin; Cell biology; Inositol; Chemistry; Receptor; Cytosol; Diacylglycerol kinase; Calcium signaling; Neurodegeneration; HEK 293 cells; Biology; Signal transduction; Biochemistry; Protein kinase C; Internal medicine","retraction":null,"screen_n_in":null,"score":{"opus":0.02035345362674342,"gpt":0.2489654150928828,"spread":0.2286119614661394,"validation_status":"score_only:v0-immature-baseline"},"prediction":{"model_version":"codex-gemma-dda1882f352a","candidate_categories":["metaresearch","metaepi_narrow"],"consensus_categories":[],"category_scores_codex":[0.0002844081,0.0004538035,0.0003615878,0.0001230121,0.0004104842,0.0002122755,0.0003651084,0.000134281,0.0001713014],"category_scores_gemma":[0.01040716,0.0004738325,0.00007412491,0.0008584924,0.0002318171,0.0002690049,0.0001282654,0.0004738284,0.000137121],"about_ca_system_candidate":false,"about_ca_system_consensus":false,"about_ca_system_score_codex":0.00004131824,"about_ca_system_score_gemma":0.0001465349,"about_ca_topic_candidate":false,"about_ca_topic_consensus":false,"about_ca_topic_score_codex":0.00002987377,"about_ca_topic_score_gemma":0.000002443021,"domain_scores_codex":[0.9957917,0.001013306,0.0005557165,0.001177596,0.0006630735,0.0007986299],"domain_scores_gemma":[0.9982279,0.0005376165,0.0003675306,0.0003571107,0.0001776343,0.0003322402],"domain_codex":null,"domain_gemma":null,"domain_candidate":null,"domain_consensus":null,"study_design_codex":"bench_or_experimental","study_design_gemma":"bench_or_experimental","study_design_scores_codex":[0.00006016422,0.0001698294,0.008629576,0.0000220129,0.000004242535,0.00001285411,0.00006123722,0.00008250852,0.9895889,0.0001062663,0.001120315,0.0001420925],"study_design_scores_gemma":[0.0009046797,0.0003329402,0.009151702,0.00003890724,0.00002174211,0.000004959727,0.00001276829,0.0001729989,0.9825179,0.00003599812,0.006388279,0.000417114],"study_design_candidate":"bench_or_experimental","study_design_consensus":"bench_or_experimental","genre_codex":"empirical","genre_gemma":"empirical","genre_scores_codex":[0.9976324,0.0001325795,0.00001208348,0.0003179845,0.0004489439,0.0007715249,0.00007418052,0.0002483936,0.0003618876],"genre_scores_gemma":[0.9966387,0.00003100205,0.0001292327,0.002331047,0.00004290035,0.0000605455,0.00001910487,0.00009287416,0.0006546357],"genre_candidate":"empirical","genre_consensus":"empirical","teacher_disagreement_score":0.01012275,"threshold_uncertainty_score":0.9997714,"prediction_status":"machine_predicted_unvalidated"},"labels":[],"label_agreement":null},{"id":"W2121926054","doi":"10.1111/j.1460-9568.2008.06310.x","title":"Mechanisms of neurodegeneration in Huntington’s disease","year":2008,"lang":"en","type":"review","venue":"European Journal of Neuroscience","topic":"Genetic Neurodegenerative Diseases","field":"Neuroscience","cited_by":470,"is_retracted":false,"has_abstract":true,"routes":{"ca_aff":true,"ca_fund":false,"ca_venue":false,"about_ca":false},"ca_institutions":"University of Victoria","funders":"National Institute on Alcohol Abuse and Alcoholism; National Institutes of Health","keywords":"Huntingtin; Neurodegeneration; Huntington's disease; Excitotoxicity; Huntingtin Protein; Biology; Cell biology; Neuroscience; Autophagy; Polyglutamine tract; Apoptosis; Mutant; Genetics; Programmed cell death; Disease; Medicine; Gene; Internal medicine","retraction":null,"screen_n_in":null,"score":{"opus":0.08294353232152592,"gpt":0.3106552191218857,"spread":0.2277116868003598,"validation_status":"score_only:v0-immature-baseline"},"prediction":{"model_version":"codex-gemma-dda1882f352a","candidate_categories":["metaepi_narrow"],"consensus_categories":[],"category_scores_codex":[0.0006803912,0.0005944138,0.001403734,0.001054072,0.0001573344,0.000100913,0.002090019,0.00004098145,0.00001251403],"category_scores_gemma":[0.004822671,0.000495982,0.000740736,0.001582977,0.0004569039,0.0003957548,0.0003517845,0.0007231786,0.00003028849],"about_ca_system_candidate":false,"about_ca_system_consensus":false,"about_ca_system_score_codex":0.00005739984,"about_ca_system_score_gemma":0.0007927231,"about_ca_topic_candidate":false,"about_ca_topic_consensus":false,"about_ca_topic_score_codex":5.457414e-7,"about_ca_topic_score_gemma":2.828566e-7,"domain_scores_codex":[0.9910051,0.004086156,0.002208105,0.0009341679,0.001261562,0.0005049114],"domain_scores_gemma":[0.9956207,0.0003569084,0.002697706,0.00070119,0.0001690725,0.0004543795],"domain_codex":null,"domain_gemma":null,"domain_candidate":null,"domain_consensus":null,"study_design_codex":"bench_or_experimental","study_design_gemma":"not_applicable","study_design_scores_codex":[0.0001216749,0.001538642,0.00002676241,0.004246645,0.000009286939,0.008146846,0.0002396348,0.002797436,0.5447118,0.001669519,0.0007834248,0.4357083],"study_design_scores_gemma":[0.002226218,0.002791075,0.006382264,0.0157658,0.0006557118,0.003067615,0.00002159169,0.000431623,0.1042125,0.0003788496,0.8612877,0.002779093],"study_design_candidate":"not_applicable","study_design_consensus":null,"genre_codex":"review","genre_gemma":"review","genre_scores_codex":[0.01673153,0.9739065,0.002898534,0.00009977177,0.004117306,0.001160512,0.0001460352,0.00005497707,0.0008848754],"genre_scores_gemma":[0.03614398,0.9628175,0.0002446552,0.0004084093,0.0001444185,0.000004764391,0.000001563719,0.0001193854,0.0001153118],"genre_candidate":"review","genre_consensus":"review","teacher_disagreement_score":0.8605042,"threshold_uncertainty_score":0.9997492,"prediction_status":"machine_predicted_unvalidated"},"labels":[],"label_agreement":null},{"id":"W2027275070","doi":"10.1038/nm.2056","title":"Balance between synaptic versus extrasynaptic NMDA receptor activity influences inclusions and neurotoxicity of mutant huntingtin","year":2009,"lang":"en","type":"article","venue":"Nature Medicine","topic":"Genetic Neurodegenerative Diseases","field":"Neuroscience","cited_by":431,"is_retracted":false,"has_abstract":false,"routes":{"ca_aff":true,"ca_fund":true,"ca_venue":false,"about_ca":false},"ca_institutions":"University of British Columbia","funders":"Eunice Kennedy Shriver National Institute of Child Health and Human Development; National Institute of Neurological Disorders and Stroke; National Eye Institute; National Institute of Environmental Health Sciences; Canadian Institutes of Health Research","keywords":"Huntingtin; NMDA receptor; Memantine; Neuroprotection; Glutamate receptor; Excitotoxicity; CREB; Biology; Cell biology; Neuroscience; Huntingtin Protein; AMPA receptor; Receptor; Mutant; Genetics; Transcription factor; Gene","retraction":null,"screen_n_in":null,"score":{"opus":0.02636686660892221,"gpt":0.3159261404185545,"spread":0.2895592738096323,"validation_status":"score_only:v0-immature-baseline"},"prediction":{"model_version":"codex-gemma-dda1882f352a","candidate_categories":[],"consensus_categories":[],"category_scores_codex":[0.0002340178,0.0002636537,0.0004488576,0.0001523262,0.0002011705,0.00001321816,0.0003614644,0.0001730558,0.00006509582],"category_scores_gemma":[0.006921462,0.0002048367,0.00004846779,0.000569241,0.0005468732,0.0001228273,0.0001272902,0.0006792837,0.000004666877],"about_ca_system_candidate":false,"about_ca_system_consensus":false,"about_ca_system_score_codex":0.0000261855,"about_ca_system_score_gemma":0.00006220162,"about_ca_topic_candidate":false,"about_ca_topic_consensus":false,"about_ca_topic_score_codex":0.00001372726,"about_ca_topic_score_gemma":0.000005030932,"domain_scores_codex":[0.9978069,0.0002935103,0.0003220799,0.0006267501,0.000618586,0.0003321785],"domain_scores_gemma":[0.9978353,0.001245937,0.0002503572,0.0003679016,0.00008997868,0.0002105757],"domain_codex":null,"domain_gemma":null,"domain_candidate":null,"domain_consensus":null,"study_design_codex":"bench_or_experimental","study_design_gemma":"bench_or_experimental","study_design_scores_codex":[0.0001566982,0.00007614112,0.006258064,0.00002671036,0.000007747834,0.000007368169,0.000120793,0.00001031526,0.9897594,0.0002227211,0.00007465743,0.003279425],"study_design_scores_gemma":[0.001059473,0.000910057,0.1992125,0.00009172835,0.00004374306,0.000003489722,0.00002170327,0.0001417634,0.7980227,0.0001985576,0.0001401389,0.000154107],"study_design_candidate":"bench_or_experimental","study_design_consensus":"bench_or_experimental","genre_codex":"empirical","genre_gemma":"empirical","genre_scores_codex":[0.99551,0.0002759886,0.00004741386,0.002853766,0.0004147307,0.0004264188,0.00004193696,0.00006995932,0.0003597194],"genre_scores_gemma":[0.9983952,0.0001226854,0.0001415209,0.001056793,0.000241742,0.000008368919,0.000002851041,0.00001437954,0.00001646521],"genre_candidate":"empirical","genre_consensus":"empirical","teacher_disagreement_score":0.1929545,"threshold_uncertainty_score":0.8352997,"prediction_status":"machine_predicted_unvalidated"},"labels":[],"label_agreement":null},{"id":"W2037606715","doi":"10.1038/nrm2854","title":"Repeat instability as the basis for human diseases and as a potential target for therapy","year":2010,"lang":"en","type":"review","venue":"Nature Reviews Molecular Cell Biology","topic":"Genetic Neurodegenerative Diseases","field":"Neuroscience","cited_by":431,"is_retracted":false,"has_abstract":false,"routes":{"ca_aff":true,"ca_fund":true,"ca_venue":false,"about_ca":false},"ca_institutions":"SickKids Foundation; University of Toronto; Hospital for Sick Children","funders":"National Institute of Neurological Disorders and Stroke; Muscular Dystrophy Canada; National Institutes of Health; University of Rochester; Canadian Institutes of Health Research; Hospital for Sick Children","keywords":"Pathogenesis; Epigenetics; Disease; Biology; Genome instability; DNA; Trinucleotide repeat expansion; Transcription (linguistics); DNA methylation; Genetics; Bioinformatics; Neuroscience; Medicine; DNA damage; Gene; Immunology; Gene expression; Internal medicine","retraction":null,"screen_n_in":null,"score":{"opus":0.03504752321665539,"gpt":0.3596660601247794,"spread":0.324618536908124,"validation_status":"score_only:v0-immature-baseline"},"prediction":{"model_version":"codex-gemma-dda1882f352a","candidate_categories":["metaepi_narrow"],"consensus_categories":[],"category_scores_codex":[0.0005394148,0.0008705938,0.002127479,0.0001238287,0.0004628214,0.000094407,0.001030889,0.001013417,0.00007043927],"category_scores_gemma":[0.004227568,0.0004995547,0.001770938,0.0002706216,0.0005167085,0.00003464366,0.0002190636,0.001032316,0.00001916543],"about_ca_system_candidate":false,"about_ca_system_consensus":false,"about_ca_system_score_codex":0.00003492569,"about_ca_system_score_gemma":0.0003313062,"about_ca_topic_candidate":false,"about_ca_topic_consensus":false,"about_ca_topic_score_codex":0.000003198076,"about_ca_topic_score_gemma":0.000001668969,"domain_scores_codex":[0.9942462,0.002259663,0.0008746668,0.001861227,0.0001810177,0.000577167],"domain_scores_gemma":[0.9965714,0.0009244295,0.0007535284,0.001390042,0.0001371088,0.0002234545],"domain_codex":null,"domain_gemma":null,"domain_candidate":null,"domain_consensus":null,"study_design_codex":"design_other","study_design_gemma":"not_applicable","study_design_scores_codex":[0.00005052516,0.0002201901,0.00000245813,0.006825396,0.00003921992,0.00001177238,0.00001099976,2.812872e-7,0.08204351,0.002585021,0.0004310159,0.9077796],"study_design_scores_gemma":[0.0003357687,0.0004907379,0.000001162002,0.0003499723,0.0006268702,0.00002301487,0.00000124495,0.000001266263,0.02224859,0.002784949,0.9726593,0.0004771389],"study_design_candidate":"not_applicable","study_design_consensus":null,"genre_codex":"review","genre_gemma":"review","genre_scores_codex":[0.001012583,0.9856548,0.0002581378,0.0001685585,0.0007047103,0.01081481,0.001123383,0.00004110941,0.000221881],"genre_scores_gemma":[0.000879468,0.9911596,0.0005238499,0.002846733,0.0003010667,0.003694206,0.0003566357,0.0001113662,0.0001270358],"genre_candidate":"review","genre_consensus":"review","teacher_disagreement_score":0.9722283,"threshold_uncertainty_score":0.9997456,"prediction_status":"machine_predicted_unvalidated"},"labels":[],"label_agreement":null},{"id":"W2015789019","doi":"10.1212/01.wnl.0000132965.14653.d1","title":"Onset and rate of striatal atrophy in preclinical Huntington disease","year":2004,"lang":"en","type":"article","venue":"Neurology","topic":"Genetic Neurodegenerative Diseases","field":"Neuroscience","cited_by":401,"is_retracted":false,"has_abstract":true,"routes":{"ca_aff":false,"ca_fund":true,"ca_venue":false,"about_ca":false},"ca_institutions":"","funders":"National Center for Research Resources; University of British Columbia; National Institute of Neurological Disorders and Stroke; Johns Hopkins University; University of Washington","keywords":"Atrophy; Putamen; Huntington's disease; Medicine; Caudate nucleus; Internal medicine; Cardiology; Psychology; Disease","retraction":null,"screen_n_in":null,"score":{"opus":0.03494584068763029,"gpt":0.3042041987879837,"spread":0.2692583581003534,"validation_status":"score_only:v0-immature-baseline"},"prediction":{"model_version":"codex-gemma-dda1882f352a","candidate_categories":[],"consensus_categories":[],"category_scores_codex":[0.00008884607,0.0001168901,0.0001854333,0.00009117608,0.00003304956,0.000009822686,0.0001572904,0.00004536462,0.00001883871],"category_scores_gemma":[0.00158066,0.0001150584,0.00004511318,0.0001547666,0.000303837,0.00004976423,0.0001260802,0.0001698911,0.00001268302],"about_ca_system_candidate":false,"about_ca_system_consensus":false,"about_ca_system_score_codex":0.000003671574,"about_ca_system_score_gemma":0.00008455762,"about_ca_topic_candidate":false,"about_ca_topic_consensus":false,"about_ca_topic_score_codex":0.00003205286,"about_ca_topic_score_gemma":0.00002934573,"domain_scores_codex":[0.9984882,0.0004144266,0.0002706389,0.0005077883,0.00009267803,0.0002262219],"domain_scores_gemma":[0.9991329,0.0003904502,0.00008850117,0.000242154,0.00001521482,0.0001307839],"domain_codex":null,"domain_gemma":null,"domain_candidate":null,"domain_consensus":null,"study_design_codex":"bench_or_experimental","study_design_gemma":"observational","study_design_scores_codex":[0.003620301,0.0008576683,0.3725155,0.00005812708,0.000004373012,0.0002326785,0.0001329195,0.004883446,0.6134796,0.003316256,0.00004794469,0.0008512558],"study_design_scores_gemma":[0.001553424,0.0007686674,0.6880454,0.000004456434,0.00001201435,0.000004704995,0.000001030318,0.00007772574,0.3057619,0.003574882,0.00009648058,0.00009933606],"study_design_candidate":"observational","study_design_consensus":null,"genre_codex":"empirical","genre_gemma":"empirical","genre_scores_codex":[0.9979305,0.00002491591,0.00001520008,0.001527082,0.0001811137,0.0001986734,0.00005221405,0.00002036603,0.0000499282],"genre_scores_gemma":[0.9970924,0.00004727471,0.0000319384,0.002760839,0.0000323693,0.00001260906,0.00000212683,0.00001363481,0.000006759513],"genre_candidate":"empirical","genre_consensus":"empirical","teacher_disagreement_score":0.3155299,"threshold_uncertainty_score":0.4691946,"prediction_status":"machine_predicted_unvalidated"},"labels":[],"label_agreement":null},{"id":"W1998237974","doi":"10.1016/j.pneurobio.2006.11.003","title":"N-Methyl-d-aspartate (NMDA) receptor function and excitotoxicity in Huntington's disease","year":2006,"lang":"en","type":"review","venue":"Progress in Neurobiology","topic":"Genetic Neurodegenerative Diseases","field":"Neuroscience","cited_by":399,"is_retracted":false,"has_abstract":false,"routes":{"ca_aff":true,"ca_fund":false,"ca_venue":false,"about_ca":false},"ca_institutions":"University of British Columbia","funders":"","keywords":"Excitotoxicity; Huntingtin; NMDA receptor; Glutamate receptor; Huntington's disease; Neuroscience; Neurodegeneration; Biology; Ionotropic effect; Striatum; Medium spiny neuron; Receptor; Internal medicine; Medicine; Disease; Biochemistry; Dopamine","retraction":null,"screen_n_in":null,"score":{"opus":0.04753627104307925,"gpt":0.3307021269686228,"spread":0.2831658559255436,"validation_status":"score_only:v0-immature-baseline"},"prediction":{"model_version":"codex-gemma-dda1882f352a","candidate_categories":["metaepi_narrow"],"consensus_categories":[],"category_scores_codex":[0.0002351747,0.0007020645,0.001378186,0.0006029939,0.00009638321,0.00007896035,0.0004754699,0.0002844254,0.00003280833],"category_scores_gemma":[0.0007347265,0.0006312614,0.0002085026,0.0007941524,0.0006670183,0.0001312928,0.0003596511,0.000734199,0.00005389558],"about_ca_system_candidate":false,"about_ca_system_consensus":false,"about_ca_system_score_codex":0.00006028951,"about_ca_system_score_gemma":0.0001721515,"about_ca_topic_candidate":false,"about_ca_topic_consensus":false,"about_ca_topic_score_codex":0.000006460085,"about_ca_topic_score_gemma":0.00002301276,"domain_scores_codex":[0.9939049,0.002401341,0.0008922495,0.001856491,0.0001814996,0.0007635166],"domain_scores_gemma":[0.9980358,0.0007116626,0.0004967983,0.00053226,0.00003065327,0.0001927721],"domain_codex":null,"domain_gemma":null,"domain_candidate":null,"domain_consensus":null,"study_design_codex":"design_other","study_design_gemma":"not_applicable","study_design_scores_codex":[0.0002455038,0.001280648,0.03549194,0.01100255,0.00001770439,0.0001161171,0.00002800452,0.00003788752,0.001905377,0.0009718,0.0003584469,0.948544],"study_design_scores_gemma":[0.001955206,0.0007510551,0.02627805,0.005381357,0.0002538723,0.00001936924,0.000005090717,0.00005277818,0.006282604,0.001202859,0.9553338,0.002484006],"study_design_candidate":"not_applicable","study_design_consensus":null,"genre_codex":"review","genre_gemma":"review","genre_scores_codex":[0.1787921,0.8178157,0.000004622273,0.00005355449,0.00105229,0.001924882,0.0001839544,0.0001067488,0.00006609005],"genre_scores_gemma":[0.03582012,0.9622537,0.00004715161,0.0002292142,0.0001796094,0.001165401,0.0001086205,0.0001071513,0.00008902279],"genre_candidate":"review","genre_consensus":"review","teacher_disagreement_score":0.9549753,"threshold_uncertainty_score":0.9996139,"prediction_status":"machine_predicted_unvalidated"},"labels":[],"label_agreement":null},{"id":"W1820767944","doi":"10.1523/jneurosci.20-10-03705.2000","title":"Wild-Type Huntingtin Protects from Apoptosis Upstream of Caspase-3","year":2000,"lang":"en","type":"article","venue":"Journal of Neuroscience","topic":"Genetic Neurodegenerative Diseases","field":"Neuroscience","cited_by":395,"is_retracted":false,"has_abstract":true,"routes":{"ca_aff":true,"ca_fund":false,"ca_venue":false,"about_ca":false},"ca_institutions":"University of British Columbia","funders":"National Institute of Neurological Disorders and Stroke; Hereditary Disease Foundation; National Institutes of Health; Consiglio Nazionale delle Ricerche; Strong; U.S. Public Health Service","keywords":"Huntingtin; Biology; Cell biology; Mutant; Huntingtin Protein; Wild type; Apoptosis; Programmed cell death; Caspase; Huntington's disease; Mutation; Molecular biology; Genetics; Gene; Disease","retraction":null,"screen_n_in":null,"score":{"opus":0.03637928876880873,"gpt":0.2728292860174387,"spread":0.2364499972486299,"validation_status":"score_only:v0-immature-baseline"},"prediction":{"model_version":"codex-gemma-dda1882f352a","candidate_categories":[],"consensus_categories":[],"category_scores_codex":[0.000193665,0.0001988342,0.0003201123,0.0001731441,0.0001607774,0.00008130255,0.0009065622,0.00003988854,0.0002193561],"category_scores_gemma":[0.002339415,0.0001650313,0.0001541893,0.001031459,0.0003834682,0.0003608303,0.00007166863,0.0002644676,0.00002546983],"about_ca_system_candidate":false,"about_ca_system_consensus":false,"about_ca_system_score_codex":0.00001914337,"about_ca_system_score_gemma":0.0002042459,"about_ca_topic_candidate":false,"about_ca_topic_consensus":false,"about_ca_topic_score_codex":0.00002175763,"about_ca_topic_score_gemma":0.000001229306,"domain_scores_codex":[0.9974077,0.0002969291,0.0006248212,0.0004408734,0.0008918219,0.0003378509],"domain_scores_gemma":[0.9983781,0.0002559807,0.0005844536,0.0003790778,0.0001730735,0.0002293177],"domain_codex":null,"domain_gemma":null,"domain_candidate":null,"domain_consensus":null,"study_design_codex":"bench_or_experimental","study_design_gemma":"bench_or_experimental","study_design_scores_codex":[0.00008573908,0.0002237058,0.001483751,0.000006227291,0.000001140205,0.00003709707,0.00009940661,0.001197936,0.9930534,0.00001561726,0.0002173495,0.003578647],"study_design_scores_gemma":[0.0003319664,0.0006101162,0.01595807,0.00004507545,0.00001925454,0.00007017652,0.00001683139,0.0002497495,0.9812417,0.0001758107,0.001144807,0.0001364245],"study_design_candidate":"bench_or_experimental","study_design_consensus":"bench_or_experimental","genre_codex":"empirical","genre_gemma":"empirical","genre_scores_codex":[0.9982648,0.00004063403,0.00005819007,0.0003123024,0.0005990323,0.0001815547,0.00002631598,0.00001791423,0.0004992957],"genre_scores_gemma":[0.9979007,0.0001393533,0.0004661609,0.001067624,0.0001244713,0.000002458321,1.7048e-7,0.00002051966,0.0002785247],"genre_candidate":"empirical","genre_consensus":"empirical","teacher_disagreement_score":0.01447432,"threshold_uncertainty_score":0.672978,"prediction_status":"machine_predicted_unvalidated"},"labels":[],"label_agreement":null},{"id":"W1991714330","doi":"10.1083/jcb.200909067","title":"IKK phosphorylates Huntingtin and targets it for degradation by the proteasome and lysosome","year":2009,"lang":"en","type":"article","venue":"The Journal of Cell Biology","topic":"Genetic Neurodegenerative Diseases","field":"Neuroscience","cited_by":393,"is_retracted":false,"has_abstract":true,"routes":{"ca_aff":true,"ca_fund":false,"ca_venue":false,"about_ca":false},"ca_institutions":"University of British Columbia","funders":"Eunice Kennedy Shriver National Institute of Child Health and Human Development; National Institute of Neurological Disorders and Stroke; National Institute of General Medical Sciences; National Institute on Aging; Hereditary Disease Foundation; National Institutes of Health; Fox Family Foundation; Cure Huntington's Disease Initiative","keywords":"Huntingtin; Lysosome; Cell biology; SUMO protein; Biology; Proteasome; Phosphorylation; Ubiquitin; Neurotoxicity; Mutant; Biochemistry; Chemistry; Gene","retraction":null,"screen_n_in":null,"score":{"opus":0.01824041131233203,"gpt":0.2595307368913448,"spread":0.2412903255790127,"validation_status":"score_only:v0-immature-baseline"},"prediction":{"model_version":"codex-gemma-dda1882f352a","candidate_categories":[],"consensus_categories":[],"category_scores_codex":[0.0003490949,0.00009061043,0.0001211849,0.00002669227,0.0001791809,0.00002574253,0.000187989,0.00003001134,0.000007757804],"category_scores_gemma":[0.0004022491,0.00004512616,0.00003017423,0.00005431614,0.0001695499,0.00005016917,0.00002596335,0.0001034962,0.000001104292],"about_ca_system_candidate":false,"about_ca_system_consensus":false,"about_ca_system_score_codex":0.000005246663,"about_ca_system_score_gemma":0.00002026685,"about_ca_topic_candidate":false,"about_ca_topic_consensus":false,"about_ca_topic_score_codex":0.000001711913,"about_ca_topic_score_gemma":5.18888e-7,"domain_scores_codex":[0.9992126,0.0002573956,0.0001993483,0.0001131225,0.00006776086,0.0001497539],"domain_scores_gemma":[0.9990739,0.0004839632,0.0002517868,0.00008932553,0.00005641497,0.00004459281],"domain_codex":null,"domain_gemma":null,"domain_candidate":null,"domain_consensus":null,"study_design_codex":"bench_or_experimental","study_design_gemma":"bench_or_experimental","study_design_scores_codex":[0.00009041564,0.00002746324,0.0001810781,0.000003298696,0.00000186434,3.632787e-7,0.0001462098,0.000007598805,0.9966165,0.0001007291,0.0005471409,0.002277309],"study_design_scores_gemma":[0.0003738985,0.0005724063,0.001597107,0.000003421747,0.00002507051,0.00003487721,0.00005169677,0.0001267024,0.9912869,0.00304398,0.002829867,0.00005401353],"study_design_candidate":"bench_or_experimental","study_design_consensus":"bench_or_experimental","genre_codex":"empirical","genre_gemma":"empirical","genre_scores_codex":[0.9929794,0.00165596,0.000270718,0.004645265,0.0000747938,0.000277402,0.0000109521,0.000003490221,0.00008203874],"genre_scores_gemma":[0.9972651,0.0004824229,0.0001412509,0.00186548,0.00005547502,0.00000329197,7.423538e-7,0.000005266308,0.0001809569],"genre_candidate":"empirical","genre_consensus":"empirical","teacher_disagreement_score":0.005329571,"threshold_uncertainty_score":0.1840191,"prediction_status":"machine_predicted_unvalidated"},"labels":[],"label_agreement":null},{"id":"W2123275897","doi":"10.1212/wnl.0b013e318249f683","title":"CAG repeat expansion in Huntington disease determines age at onset in a fully dominant fashion","year":2012,"lang":"en","type":"article","venue":"Neurology","topic":"Genetic Neurodegenerative Diseases","field":"Neuroscience","cited_by":391,"is_retracted":false,"has_abstract":true,"routes":{"ca_aff":true,"ca_fund":false,"ca_venue":false,"about_ca":false},"ca_institutions":"Hôtel-Dieu de Montréal; University of Alberta; University of Toronto; Centre for Addiction and Mental Health; Simon Fraser University; University of Calgary; University of British Columbia","funders":"National Center for Advancing Translational Sciences; National Center for Research Resources; National Institute of Neurological Disorders and Stroke; National Institutes of Health; National Institute for Health and Care Research; Wellcome Trust; Northwestern University","keywords":"Allele; Age of onset; Huntington's disease; Trinucleotide repeat expansion; Analysis of variance; Biology; Disease; Allele frequency; Genetics; Medicine; Internal medicine; Gene","retraction":null,"screen_n_in":null,"score":{"opus":0.02598145629466226,"gpt":0.2718766535527191,"spread":0.2458951972580569,"validation_status":"score_only:v0-immature-baseline"},"prediction":{"model_version":"codex-gemma-dda1882f352a","candidate_categories":[],"consensus_categories":[],"category_scores_codex":[0.0001637669,0.0002508806,0.0002718554,0.0002863151,0.00008903591,0.00001861912,0.0002664173,0.00008748462,0.00005782531],"category_scores_gemma":[0.001238469,0.000237842,0.0000726217,0.0003076501,0.0001629327,0.0001593247,0.0003032742,0.0002252654,0.0001036587],"about_ca_system_candidate":false,"about_ca_system_consensus":false,"about_ca_system_score_codex":0.00003523943,"about_ca_system_score_gemma":0.0000423941,"about_ca_topic_candidate":false,"about_ca_topic_consensus":false,"about_ca_topic_score_codex":0.00004166951,"about_ca_topic_score_gemma":0.0006479676,"domain_scores_codex":[0.99704,0.0008842349,0.0003753068,0.0007357608,0.0002314663,0.0007332092],"domain_scores_gemma":[0.9987509,0.0003657717,0.0001114284,0.0005020804,0.00001499412,0.000254791],"domain_codex":null,"domain_gemma":null,"domain_candidate":null,"domain_consensus":null,"study_design_codex":"bench_or_experimental","study_design_gemma":"observational","study_design_scores_codex":[0.0006081476,0.000243874,0.410742,0.00001547432,2.944887e-7,0.0005237305,0.0001973539,0.0001341931,0.5870837,0.00002859458,0.00007903232,0.0003436067],"study_design_scores_gemma":[0.0007374204,0.0002216563,0.7167193,0.000008187342,0.000008108696,0.00004422135,0.000004719604,0.0002737445,0.2812026,0.00004328657,0.0005484846,0.0001882352],"study_design_candidate":"observational","study_design_consensus":null,"genre_codex":"empirical","genre_gemma":"empirical","genre_scores_codex":[0.998081,0.00005355341,0.000005704135,0.0006311897,0.000635738,0.0004098394,0.00001849957,0.00004021376,0.0001243226],"genre_scores_gemma":[0.9958398,0.00004888463,0.00001425792,0.003730119,0.00008699344,0.0001055135,0.000007694261,0.00003618246,0.0001305846],"genre_candidate":"empirical","genre_consensus":"empirical","teacher_disagreement_score":0.3059773,"threshold_uncertainty_score":0.9698914,"prediction_status":"machine_predicted_unvalidated"},"labels":[],"label_agreement":null},{"id":"W2125054419","doi":"10.1093/hmg/ddm217","title":"Huntingtin has a membrane association signal that can modulate huntingtin aggregation, nuclear entry and toxicity","year":2007,"lang":"en","type":"article","venue":"Human Molecular Genetics","topic":"Genetic Neurodegenerative Diseases","field":"Neuroscience","cited_by":389,"is_retracted":false,"has_abstract":true,"routes":{"ca_aff":true,"ca_fund":true,"ca_venue":false,"about_ca":false},"ca_institutions":"McMaster University","funders":"Canadian Institutes of Health Research; University of Toronto; McMaster University; Cure Huntington's Disease Initiative","keywords":"Huntingtin; Polyglutamine tract; Biology; Huntingtin Protein; Cell biology; Endoplasmic reticulum; Endosome; Mutant; Biochemistry; Intracellular; Gene","retraction":null,"screen_n_in":null,"score":{"opus":0.02642567496643303,"gpt":0.2475578439840799,"spread":0.2211321690176469,"validation_status":"score_only:v0-immature-baseline"},"prediction":{"model_version":"codex-gemma-dda1882f352a","candidate_categories":["metaepi_narrow"],"consensus_categories":[],"category_scores_codex":[0.0004081018,0.0003081923,0.0002395394,0.0001542284,0.0005830311,0.0003189289,0.0002893292,0.0001404403,0.0001100863],"category_scores_gemma":[0.0004430244,0.0003613033,0.0001034959,0.0002584362,0.0001770652,0.00005973404,0.0001910846,0.0002255078,0.00002324117],"about_ca_system_candidate":false,"about_ca_system_consensus":false,"about_ca_system_score_codex":0.00009511163,"about_ca_system_score_gemma":0.00004936049,"about_ca_topic_candidate":false,"about_ca_topic_consensus":false,"about_ca_topic_score_codex":0.00002400148,"about_ca_topic_score_gemma":0.00004519838,"domain_scores_codex":[0.9972128,0.0002731278,0.0004031834,0.0007510577,0.0007439721,0.0006159015],"domain_scores_gemma":[0.998778,0.0001270487,0.0003570687,0.0003766488,0.0001268649,0.0002343901],"domain_codex":null,"domain_gemma":null,"domain_candidate":null,"domain_consensus":null,"study_design_codex":"bench_or_experimental","study_design_gemma":"bench_or_experimental","study_design_scores_codex":[0.00001011784,0.00009222373,0.01071431,0.00002080893,0.00001207743,0.00004175073,0.0003637772,0.0005567464,0.9868149,0.0004360067,0.0001065009,0.0008308364],"study_design_scores_gemma":[0.0005581027,0.00007937936,0.05646345,0.00002067179,0.00005057665,0.000009487899,0.00004445285,0.0008913096,0.9404977,0.0005283948,0.0005188,0.0003376555],"study_design_candidate":"bench_or_experimental","study_design_consensus":"bench_or_experimental","genre_codex":"empirical","genre_gemma":"empirical","genre_scores_codex":[0.9968534,0.0001137901,0.001022114,0.0002768934,0.0001157903,0.0003835363,0.00002251678,0.000111797,0.001100219],"genre_scores_gemma":[0.9968771,0.00003851857,0.0008439302,0.001772583,0.0001128542,0.000006470812,0.00001168493,0.00007780909,0.0002590947],"genre_candidate":"empirical","genre_consensus":"empirical","teacher_disagreement_score":0.04631712,"threshold_uncertainty_score":0.9998839,"prediction_status":"machine_predicted_unvalidated"},"labels":[],"label_agreement":null},{"id":"W2068816428","doi":"10.1212/wnl.56.3.336","title":"Assessment of a disease-specific muscular impairment rating scale in myotonic dystrophy","year":2001,"lang":"en","type":"article","venue":"Neurology","topic":"Genetic Neurodegenerative Diseases","field":"Neuroscience","cited_by":377,"is_retracted":false,"has_abstract":true,"routes":{"ca_aff":true,"ca_fund":false,"ca_venue":false,"about_ca":false},"ca_institutions":"Centre Hospitalier de l’Université de Montréal","funders":"","keywords":"Myotonic dystrophy; Muscular dystrophy; Rating scale; Physical medicine and rehabilitation; Medicine; Disease; Neuromuscular disease; Physical therapy; Psychology; Internal medicine; Developmental psychology","retraction":null,"screen_n_in":null,"score":{"opus":0.02293268131362515,"gpt":0.2777772417186982,"spread":0.254844560405073,"validation_status":"score_only:v0-immature-baseline"},"prediction":{"model_version":"codex-gemma-dda1882f352a","candidate_categories":[],"consensus_categories":[],"category_scores_codex":[0.0001171606,0.0001997432,0.0002711526,0.0001910673,0.00007315211,0.00001886906,0.0003157238,0.00004643252,0.0001643709],"category_scores_gemma":[0.00009375529,0.00019702,0.0001151198,0.0004059028,0.000196062,0.00006501444,0.0001419539,0.0002212863,0.00001867919],"about_ca_system_candidate":false,"about_ca_system_consensus":false,"about_ca_system_score_codex":0.00002718337,"about_ca_system_score_gemma":0.0001026039,"about_ca_topic_candidate":false,"about_ca_topic_consensus":false,"about_ca_topic_score_codex":0.00001149183,"about_ca_topic_score_gemma":0.0000144025,"domain_scores_codex":[0.9974664,0.0006286306,0.000397968,0.0007235924,0.0003449453,0.0004383938],"domain_scores_gemma":[0.9989927,0.0001634189,0.0001356289,0.0005073045,0.00003469414,0.0001662814],"domain_codex":null,"domain_gemma":null,"domain_candidate":null,"domain_consensus":null,"study_design_codex":"bench_or_experimental","study_design_gemma":"observational","study_design_scores_codex":[0.0002517231,0.0006371695,0.1974587,0.00002502171,0.000002191608,0.0003169411,0.00004860289,0.00766946,0.7924834,0.0007452373,0.00004120768,0.0003202614],"study_design_scores_gemma":[0.001041992,0.001096389,0.9104839,0.000008047936,0.0000117929,0.0000245662,0.00000664428,0.004430752,0.08142542,0.0002920153,0.00100143,0.0001770681],"study_design_candidate":"observational","study_design_consensus":null,"genre_codex":"empirical","genre_gemma":"empirical","genre_scores_codex":[0.9976273,0.00007403791,0.0002273587,0.0006541648,0.0002682789,0.0004897764,0.00001524472,0.00004036854,0.0006035002],"genre_scores_gemma":[0.9982107,0.0001857966,0.0001725563,0.001222016,0.00005174729,0.0001053869,0.000002754262,0.00002771504,0.00002136228],"genre_candidate":"empirical","genre_consensus":"empirical","teacher_disagreement_score":0.7130252,"threshold_uncertainty_score":0.8034241,"prediction_status":"machine_predicted_unvalidated"},"labels":[],"label_agreement":null},{"id":"W1968304311","doi":"10.1037/a0020937","title":"Neurocognitive signs in prodromal Huntington disease.","year":2010,"lang":"en","type":"article","venue":"Neuropsychology","topic":"Genetic Neurodegenerative Diseases","field":"Neuroscience","cited_by":377,"is_retracted":false,"has_abstract":true,"routes":{"ca_aff":true,"ca_fund":false,"ca_venue":false,"about_ca":false},"ca_institutions":"Dalhousie University","funders":"National Center for Research Resources; National Institute of Neurological Disorders and Stroke; National Institutes of Health; CHDI Foundation","keywords":"Prodrome; Huntington's disease; Neurocognitive; Disease; Prodromal Stage; Stage (stratigraphy); Medicine; Psychology; Cognition; Cognitive impairment; Psychiatry; Internal medicine; Biology; Psychosis","retraction":null,"screen_n_in":null,"score":{"opus":0.02556689841939224,"gpt":0.2998209911343865,"spread":0.2742540927149943,"validation_status":"score_only:v0-immature-baseline"},"prediction":{"model_version":"codex-gemma-dda1882f352a","candidate_categories":["metaepi_narrow"],"consensus_categories":[],"category_scores_codex":[0.00009059424,0.0002671237,0.0002055377,0.0002423359,0.0001091668,0.00004937517,0.0004951123,0.00007165573,0.0003505263],"category_scores_gemma":[0.002398317,0.0002720742,0.00008175172,0.0005053781,0.0003599642,0.0001257027,0.0001317076,0.0006931568,0.0004665171],"about_ca_system_candidate":false,"about_ca_system_consensus":false,"about_ca_system_score_codex":0.000005604107,"about_ca_system_score_gemma":0.00006348189,"about_ca_topic_candidate":false,"about_ca_topic_consensus":false,"about_ca_topic_score_codex":0.000003687006,"about_ca_topic_score_gemma":0.00001953771,"domain_scores_codex":[0.9971428,0.0005107818,0.0003102353,0.001198081,0.0002560627,0.0005820632],"domain_scores_gemma":[0.99868,0.0003185425,0.0001021198,0.0005752714,0.00004992102,0.0002741699],"domain_codex":null,"domain_gemma":null,"domain_candidate":null,"domain_consensus":null,"study_design_codex":"bench_or_experimental","study_design_gemma":"observational","study_design_scores_codex":[0.0001173481,0.0003674188,0.02616013,0.000005737766,6.571715e-7,0.0002303777,0.00004515412,0.00001645934,0.9700911,0.001615433,0.0002350878,0.001115114],"study_design_scores_gemma":[0.0007633136,0.0003130197,0.5384419,0.000004701494,0.000009119383,0.00002592937,0.000005971654,0.00005895274,0.4565283,0.001251165,0.002317274,0.0002803141],"study_design_candidate":"bench_or_experimental","study_design_consensus":null,"genre_codex":"empirical","genre_gemma":"empirical","genre_scores_codex":[0.9875546,0.000003833015,0.00002559859,0.00121851,0.00174774,0.0005494393,0.00003558111,0.0001412714,0.008723393],"genre_scores_gemma":[0.9921757,0.0000128664,0.00005964245,0.007130881,0.0001909881,0.0001119749,0.000003885481,0.00006011484,0.0002539439],"genre_candidate":"empirical","genre_consensus":"empirical","teacher_disagreement_score":0.5135627,"threshold_uncertainty_score":0.9999731,"prediction_status":"machine_predicted_unvalidated"},"labels":[],"label_agreement":null},{"id":"W1991480442","doi":"10.1038/nrn3570","title":"Choosing an animal model for the study of Huntington's disease","year":2013,"lang":"en","type":"review","venue":"Nature reviews. Neuroscience","topic":"Genetic Neurodegenerative Diseases","field":"Neuroscience","cited_by":364,"is_retracted":false,"has_abstract":false,"routes":{"ca_aff":true,"ca_fund":true,"ca_venue":false,"about_ca":false},"ca_institutions":"Child and Family Research Institute; University of British Columbia","funders":"Canadian Institutes of Health Research","keywords":"Huntington's disease; Disease; Animal model; Context (archaeology); Identification (biology); Human disease; Clinical phenotype; Neuroscience; Selection (genetic algorithm); Psychology; Bioinformatics; Medicine; Biology; Computer science; Phenotype; Pathology; Genetics; Gene; Machine learning","retraction":null,"screen_n_in":null,"score":{"opus":0.1734014947664425,"gpt":0.4192176478085518,"spread":0.2458161530421093,"validation_status":"score_only:v0-immature-baseline"},"prediction":{"model_version":"codex-gemma-dda1882f352a","candidate_categories":["metaresearch","metaepi_narrow"],"consensus_categories":[],"category_scores_codex":[0.000837112,0.000989401,0.00216074,0.0002610937,0.0007526087,0.0003568094,0.003736655,0.0002296903,0.00001081584],"category_scores_gemma":[0.008547132,0.0005829139,0.0009057677,0.001603503,0.0004930013,0.0004942135,0.0005535843,0.001239567,0.00001786157],"about_ca_system_candidate":false,"about_ca_system_consensus":false,"about_ca_system_score_codex":0.00004856877,"about_ca_system_score_gemma":0.0006253005,"about_ca_topic_candidate":false,"about_ca_topic_consensus":false,"about_ca_topic_score_codex":0.000002877431,"about_ca_topic_score_gemma":0.000002903908,"domain_scores_codex":[0.9927104,0.001514233,0.001402303,0.002452655,0.001132793,0.0007876789],"domain_scores_gemma":[0.9944155,0.001034955,0.001555366,0.002324471,0.0001960775,0.0004736258],"domain_codex":null,"domain_gemma":null,"domain_candidate":null,"domain_consensus":null,"study_design_codex":"design_other","study_design_gemma":"not_applicable","study_design_scores_codex":[0.00002621362,0.001616124,0.00001359929,0.01233819,0.000006847918,0.000005927535,0.0001340184,0.0003809959,0.005483423,0.0005487727,0.001017493,0.9784284],"study_design_scores_gemma":[0.0003421673,0.000684357,0.00009028005,0.003203556,0.001645332,0.000009308787,0.00001616405,0.01549486,0.000893013,0.00005863773,0.9765077,0.001054625],"study_design_candidate":"design_other","study_design_consensus":null,"genre_codex":"review","genre_gemma":"review","genre_scores_codex":[0.001844527,0.9830555,0.0004066091,0.00004727912,0.0009378351,0.01338526,0.0002283397,0.0000737245,0.00002096137],"genre_scores_gemma":[0.009081424,0.986895,0.000182727,0.001087244,0.0002171289,0.002162552,0.000006619683,0.0001316902,0.0002356588],"genre_candidate":"review","genre_consensus":"review","teacher_disagreement_score":0.9773738,"threshold_uncertainty_score":0.9998043,"prediction_status":"machine_predicted_unvalidated"},"labels":[],"label_agreement":null},{"id":"W2016460575","doi":"10.1002/ajmg.b.30992","title":"CAG‐repeat length and the age of onset in Huntington disease (HD): A review and validation study of statistical approaches","year":2009,"lang":"en","type":"review","venue":"American Journal of Medical Genetics Part B Neuropsychiatric Genetics","topic":"Genetic Neurodegenerative Diseases","field":"Neuroscience","cited_by":364,"is_retracted":false,"has_abstract":true,"routes":{"ca_aff":true,"ca_fund":false,"ca_venue":false,"about_ca":false},"ca_institutions":"University of British Columbia","funders":"National Center for Advancing Translational Sciences; National Institute of Neurological Disorders and Stroke","keywords":"Psychology; Disease; Perspective (graphical); Survival analysis; Medicine; Computer science; Internal medicine; Artificial intelligence","retraction":null,"screen_n_in":null,"score":{"opus":0.06078828323692004,"gpt":0.3405133718963326,"spread":0.2797250886594126,"validation_status":"score_only:v0-immature-baseline"},"prediction":{"model_version":"codex-gemma-dda1882f352a","candidate_categories":["metaepi_narrow"],"consensus_categories":[],"category_scores_codex":[0.001692862,0.0006294851,0.003176355,0.0004413013,0.00008374554,0.00005022161,0.001098041,0.0001035814,0.00001583032],"category_scores_gemma":[0.003689035,0.0004275887,0.0003173728,0.00127898,0.001794318,0.00002840658,0.0003439408,0.0009271217,8.79839e-7],"about_ca_system_candidate":false,"about_ca_system_consensus":false,"about_ca_system_score_codex":0.00002024508,"about_ca_system_score_gemma":0.0007954066,"about_ca_topic_candidate":false,"about_ca_topic_consensus":false,"about_ca_topic_score_codex":0.00001138357,"about_ca_topic_score_gemma":0.000007273051,"domain_scores_codex":[0.9882667,0.005260637,0.003084094,0.0007919702,0.00217094,0.0004256653],"domain_scores_gemma":[0.9935405,0.002032621,0.002909183,0.0007624633,0.0001496776,0.0006056129],"domain_codex":null,"domain_gemma":null,"domain_candidate":null,"domain_consensus":null,"study_design_codex":"design_other","study_design_gemma":"not_applicable","study_design_scores_codex":[0.0002203281,0.002332515,0.002451351,0.01043822,0.0001341985,0.0001953413,0.0003511373,0.0001580235,0.000008781921,0.0000910321,0.0003355423,0.9832835],"study_design_scores_gemma":[0.06222064,0.09106591,0.1620532,0.1055849,0.1517816,0.006744,0.004078406,0.003882511,0.0008578772,0.003254711,0.3953032,0.01317294],"study_design_candidate":"design_other","study_design_consensus":null,"genre_codex":"review","genre_gemma":"review","genre_scores_codex":[0.1731666,0.8245126,0.000119215,0.0003322692,0.0002166591,0.001546686,0.00008769846,0.000005205451,0.00001303426],"genre_scores_gemma":[0.087426,0.9114547,0.0004385175,0.0004514359,0.000124669,0.0000275087,0.00000891118,0.0000630711,0.000005241731],"genre_candidate":"review","genre_consensus":"review","teacher_disagreement_score":0.9701106,"threshold_uncertainty_score":0.9998176,"prediction_status":"machine_predicted_unvalidated"},"labels":[],"label_agreement":null},{"id":"W2098163379","doi":"10.1074/jbc.m001475200","title":"Inhibiting Caspase Cleavage of Huntingtin Reduces Toxicity and Aggregate Formation in Neuronal and Nonneuronal Cells","year":2000,"lang":"en","type":"article","venue":"Journal of Biological Chemistry","topic":"Genetic Neurodegenerative Diseases","field":"Neuroscience","cited_by":363,"is_retracted":false,"has_abstract":true,"routes":{"ca_aff":true,"ca_fund":false,"ca_venue":false,"about_ca":false},"ca_institutions":"University of British Columbia","funders":"National Institute of Neurological Disorders and Stroke; National Cancer Institute; National Institute on Aging","keywords":"Huntingtin; Huntingtin Protein; Cleavage (geology); Polyglutamine tract; Huntington's disease; Cell biology; Toxicity; Caspase 3; Caspase; Chemistry; Biology; Apoptosis; Programmed cell death; Molecular biology; Mutant; Biochemistry; Medicine; Disease; Internal medicine; Gene","retraction":null,"screen_n_in":null,"score":{"opus":0.02847436676864426,"gpt":0.2479588458785815,"spread":0.2194844791099372,"validation_status":"score_only:v0-immature-baseline"},"prediction":{"model_version":"codex-gemma-dda1882f352a","candidate_categories":[],"consensus_categories":[],"category_scores_codex":[0.0001575434,0.0001135417,0.0001991771,0.00002877979,0.00004580356,0.00002518657,0.0001050652,0.00005773229,0.00006808622],"category_scores_gemma":[0.0006497785,0.00008745411,0.00004618529,0.00009518386,0.0002052276,0.0001104668,0.00005232884,0.0002149676,6.270104e-7],"about_ca_system_candidate":false,"about_ca_system_consensus":false,"about_ca_system_score_codex":0.000009146911,"about_ca_system_score_gemma":0.00001996766,"about_ca_topic_candidate":false,"about_ca_topic_consensus":false,"about_ca_topic_score_codex":8.8762e-7,"about_ca_topic_score_gemma":2.236158e-7,"domain_scores_codex":[0.9989362,0.0001133535,0.0004441421,0.0001923256,0.000158227,0.0001556955],"domain_scores_gemma":[0.9992735,0.0002139306,0.0003106545,0.00005056492,0.00003010798,0.0001212263],"domain_codex":null,"domain_gemma":null,"domain_candidate":null,"domain_consensus":null,"study_design_codex":"bench_or_experimental","study_design_gemma":"bench_or_experimental","study_design_scores_codex":[0.0001021045,0.0001009941,0.0007216618,0.00002969526,7.974688e-7,0.00005437793,0.00004124949,0.00004193362,0.9962639,0.000001333548,0.00001933577,0.002622625],"study_design_scores_gemma":[0.000443086,0.000152882,0.007394824,0.00004804203,0.000004385241,0.0002488675,0.00003949968,0.0001408334,0.9913702,0.0000622903,0.00001673606,0.00007834366],"study_design_candidate":"bench_or_experimental","study_design_consensus":"bench_or_experimental","genre_codex":"empirical","genre_gemma":"empirical","genre_scores_codex":[0.9993475,0.00008396461,0.000002750314,0.0001303624,0.00001860295,0.00004662802,0.00001715172,0.000004525854,0.0003485118],"genre_scores_gemma":[0.9993204,0.0002776962,0.0001197314,0.0001977738,0.00006090277,7.909661e-7,6.43525e-7,0.000004491191,0.00001750952],"genre_candidate":"empirical","genre_consensus":"empirical","teacher_disagreement_score":0.006673162,"threshold_uncertainty_score":0.3566275,"prediction_status":"machine_predicted_unvalidated"},"labels":[],"label_agreement":null},{"id":"W2081802283","doi":"10.1073/pnas.0409402102","title":"Disturbed Ca <sup>2+</sup> signaling and apoptosis of medium spiny neurons in Huntington's disease","year":2005,"lang":"en","type":"article","venue":"Proceedings of the National Academy of Sciences","topic":"Genetic Neurodegenerative Diseases","field":"Neuroscience","cited_by":360,"is_retracted":false,"has_abstract":true,"routes":{"ca_aff":false,"ca_fund":true,"ca_venue":false,"about_ca":false},"ca_institutions":"","funders":"National Institute of Neurological Disorders and Stroke; Cure Huntington's Disease Initiative; Hereditary Disease Foundation; Canadian Institutes of Health Research; Welch Foundation","keywords":"Mitochondrial permeability transition pore; MPTP; Medium spiny neuron; Cell biology; Glutamate receptor; Biology; Apoptosis; Huntington's disease; Cytochrome c; Chemistry; Mitochondrion; Biochemistry; Programmed cell death; Receptor; Neuroscience; Internal medicine","retraction":null,"screen_n_in":null,"score":{"opus":0.03648751398554717,"gpt":0.2925185441667298,"spread":0.2560310301811827,"validation_status":"score_only:v0-immature-baseline"},"prediction":{"model_version":"codex-gemma-dda1882f352a","candidate_categories":[],"consensus_categories":[],"category_scores_codex":[0.0005573812,0.0001323883,0.0001881418,0.0002412549,0.0001596641,0.00002941144,0.0007607725,0.00003675915,0.00001475311],"category_scores_gemma":[0.003198315,0.00009820636,0.00007082149,0.0008729144,0.001290543,0.000415353,0.0002358612,0.0001406293,7.754105e-7],"about_ca_system_candidate":false,"about_ca_system_consensus":false,"about_ca_system_score_codex":0.0000185029,"about_ca_system_score_gemma":0.00006273062,"about_ca_topic_candidate":false,"about_ca_topic_consensus":false,"about_ca_topic_score_codex":0.000005349339,"about_ca_topic_score_gemma":7.603771e-8,"domain_scores_codex":[0.9976861,0.00002626925,0.0004173536,0.0004329934,0.00122836,0.0002088567],"domain_scores_gemma":[0.9990985,0.0002779739,0.0003769384,0.00001485994,0.0001408182,0.00009091318],"domain_codex":null,"domain_gemma":null,"domain_candidate":null,"domain_consensus":null,"study_design_codex":"bench_or_experimental","study_design_gemma":"bench_or_experimental","study_design_scores_codex":[0.00002525973,0.0001067614,0.02020826,0.00007502049,0.000002005438,6.47882e-9,0.0003387652,0.006274704,0.9691071,0.003603527,0.000121606,0.0001369821],"study_design_scores_gemma":[0.0001835154,0.00002987045,0.08978876,0.0000705456,0.00001277104,6.945257e-7,0.0001229723,0.006193019,0.9004559,0.003020003,0.00003756345,0.0000843662],"study_design_candidate":"bench_or_experimental","study_design_consensus":"bench_or_experimental","genre_codex":"empirical","genre_gemma":"empirical","genre_scores_codex":[0.9937667,0.00005880654,5.29448e-7,0.005380568,0.00001153407,0.0002198558,0.00004379125,0.000008194309,0.0005100635],"genre_scores_gemma":[0.9987661,0.00003573095,0.0004086998,0.000668725,0.00006175945,0.00001378175,8.786544e-8,0.000006322958,0.00003882162],"genre_candidate":"empirical","genre_consensus":"empirical","teacher_disagreement_score":0.0695805,"threshold_uncertainty_score":0.4755061,"prediction_status":"machine_predicted_unvalidated"},"labels":[],"label_agreement":null},{"id":"W2623948558","doi":"10.1016/s1474-4422(17)30124-2","title":"Neurofilament light protein in blood as a potential biomarker of neurodegeneration in Huntington's disease: a retrospective cohort analysis","year":2017,"lang":"en","type":"article","venue":"The Lancet Neurology","topic":"Genetic Neurodegenerative Diseases","field":"Neuroscience","cited_by":357,"is_retracted":false,"has_abstract":true,"routes":{"ca_aff":true,"ca_fund":false,"ca_venue":false,"about_ca":false},"ca_institutions":"University of British Columbia","funders":"Medical Research Council; Vetenskapsrådet; National Institutes of Health; Knut och Alice Wallenbergs Stiftelse; Research Councils UK; NIHR Biomedical Research Centre, Royal Marsden NHS Foundation Trust/Institute of Cancer Research","keywords":"Neurodegeneration; Huntington's disease; Biomarker; Internal medicine; Medicine; Cohort; Oncology; Retrospective cohort study; Disease; Biology; Genetics","retraction":null,"screen_n_in":null,"score":{"opus":0.02087487255600939,"gpt":0.2780571164582733,"spread":0.2571822439022639,"validation_status":"score_only:v0-immature-baseline"},"prediction":{"model_version":"codex-gemma-dda1882f352a","candidate_categories":[],"consensus_categories":[],"category_scores_codex":[0.0004289205,0.0002827134,0.0006541175,0.0003828067,0.0002553955,0.000113492,0.001100704,0.00007218751,0.00006164887],"category_scores_gemma":[0.001925059,0.000232632,0.0001766036,0.0006429516,0.000378458,0.0001621885,0.0003977052,0.0003727066,0.00001436839],"about_ca_system_candidate":false,"about_ca_system_consensus":false,"about_ca_system_score_codex":0.00001701776,"about_ca_system_score_gemma":0.00008584793,"about_ca_topic_candidate":false,"about_ca_topic_consensus":false,"about_ca_topic_score_codex":0.0002459612,"about_ca_topic_score_gemma":0.0003940119,"domain_scores_codex":[0.996143,0.001297732,0.0004994158,0.001030287,0.0004758925,0.0005536671],"domain_scores_gemma":[0.9976292,0.00009874178,0.0005005642,0.001603875,0.00006604678,0.0001015379],"domain_codex":null,"domain_gemma":null,"domain_candidate":null,"domain_consensus":null,"study_design_codex":"bench_or_experimental","study_design_gemma":"observational","study_design_scores_codex":[0.0004811656,0.0002216685,0.4641245,0.000009122871,0.00002378399,0.0001610753,0.0000405194,0.001049229,0.5329854,0.0008332882,0.00002225347,0.00004798023],"study_design_scores_gemma":[0.0008011376,0.0001488461,0.6916568,0.000004959011,0.0001522183,0.000004417124,0.000001441603,0.002046747,0.3044918,0.0005405294,0.00002804516,0.000123036],"study_design_candidate":"observational","study_design_consensus":null,"genre_codex":"empirical","genre_gemma":"empirical","genre_scores_codex":[0.9884593,0.00002936392,0.00001909144,0.00959358,0.0002299459,0.001052188,0.00004562394,0.00003453243,0.0005363048],"genre_scores_gemma":[0.997692,0.00006698813,0.00001496785,0.00175678,0.0001179526,0.0002191517,0.000005837813,0.00003104831,0.0000952894],"genre_candidate":"empirical","genre_consensus":"empirical","teacher_disagreement_score":0.2284936,"threshold_uncertainty_score":0.9486457,"prediction_status":"machine_predicted_unvalidated"},"labels":[],"label_agreement":null},{"id":"W2919539529","doi":"10.1016/j.neuron.2019.01.039","title":"Huntingtin Lowering Strategies for Disease Modification in Huntington’s Disease","year":2019,"lang":"en","type":"review","venue":"Neuron","topic":"Genetic Neurodegenerative Diseases","field":"Neuroscience","cited_by":348,"is_retracted":false,"has_abstract":false,"routes":{"ca_aff":true,"ca_fund":true,"ca_venue":false,"about_ca":false},"ca_institutions":"BC Children's Hospital; University of British Columbia","funders":"Rosetrees Trust; Wellcome Trust; Canadian Institutes of Health Research; UK Dementia Research Institute; Teva Pharmaceutical Industries; Medical Research Council; Huntington Society of Canada; CHDI Foundation","keywords":"Huntingtin; Huntington's disease; Disease; Neuroscience; Huntingtin Protein; Psychology; Biology; Medicine; Internal medicine","retraction":null,"screen_n_in":null,"score":{"opus":0.1318124163774413,"gpt":0.3684555437186128,"spread":0.2366431273411715,"validation_status":"score_only:v0-immature-baseline"},"prediction":{"model_version":"codex-gemma-dda1882f352a","candidate_categories":["metaepi_narrow"],"consensus_categories":[],"category_scores_codex":[0.0001423559,0.0006923505,0.001010881,0.0003888977,0.0001395668,0.000347445,0.0007929624,0.0001069086,0.00002871965],"category_scores_gemma":[0.001422197,0.0006793269,0.0005749634,0.0004653604,0.00009395082,0.0003014056,0.0001966633,0.0003613353,0.0001774714],"about_ca_system_candidate":false,"about_ca_system_consensus":false,"about_ca_system_score_codex":0.00009553046,"about_ca_system_score_gemma":0.0009128909,"about_ca_topic_candidate":false,"about_ca_topic_consensus":false,"about_ca_topic_score_codex":0.000004573413,"about_ca_topic_score_gemma":0.000002902871,"domain_scores_codex":[0.9959555,0.0003853462,0.0008019708,0.001752068,0.0004381202,0.000666975],"domain_scores_gemma":[0.99734,0.0006866254,0.0005126685,0.001058529,0.0000526336,0.0003496029],"domain_codex":null,"domain_gemma":null,"domain_candidate":null,"domain_consensus":null,"study_design_codex":"design_other","study_design_gemma":"not_applicable","study_design_scores_codex":[0.0003313159,0.001253512,0.0002733537,0.1075924,0.00002103314,0.00011024,0.0001199206,0.003229564,0.005877169,0.005406009,0.0008589353,0.8749266],"study_design_scores_gemma":[0.0008673938,0.0001394977,0.001165827,0.0072292,0.000969489,0.000001987524,0.00002137104,0.001712751,0.00185963,0.0007180547,0.9834776,0.001837228],"study_design_candidate":"not_applicable","study_design_consensus":null,"genre_codex":"review","genre_gemma":"review","genre_scores_codex":[0.01969567,0.9700052,0.0003767756,0.000112792,0.001663171,0.006582658,0.0007896058,0.0002762767,0.0004978661],"genre_scores_gemma":[0.03665629,0.9602158,0.00003505381,0.000306312,0.0003048611,0.001445928,0.0001528498,0.0002519574,0.0006309211],"genre_candidate":"review","genre_consensus":"review","teacher_disagreement_score":0.9826186,"threshold_uncertainty_score":0.9995658,"prediction_status":"machine_predicted_unvalidated"},"labels":[],"label_agreement":null},{"id":"W1987824870","doi":"10.1038/ng1927","title":"Mutations in SYNE1 lead to a newly discovered form of autosomal recessive cerebellar ataxia","year":2006,"lang":"en","type":"article","venue":"Nature Genetics","topic":"Genetic Neurodegenerative Diseases","field":"Neuroscience","cited_by":347,"is_retracted":false,"has_abstract":false,"routes":{"ca_aff":true,"ca_fund":true,"ca_venue":false,"about_ca":false},"ca_institutions":"Université Laval; Hôpital de l'Enfant-Jésus; Université de Montréal; Centre Hospitalier de l’Université de Montréal","funders":"McGill University","keywords":"Spinocerebellar ataxia; Ataxia; Genetics; Biology; Cerebellar ataxia; Ataxia-telangiectasia; Gene; Neuroscience","retraction":null,"screen_n_in":null,"score":{"opus":0.01340821935683464,"gpt":0.2746128036224669,"spread":0.2612045842656323,"validation_status":"score_only:v0-immature-baseline"},"prediction":{"model_version":"codex-gemma-dda1882f352a","candidate_categories":[],"consensus_categories":[],"category_scores_codex":[0.00004779069,0.0002181959,0.0002265568,0.0002088851,0.00007965677,0.00004313637,0.0004055651,0.0001757569,0.00002596069],"category_scores_gemma":[0.0005061848,0.0002141591,0.00007873437,0.0008124423,0.0001079317,0.00006981957,0.0001262702,0.0002999211,0.00002631576],"about_ca_system_candidate":false,"about_ca_system_consensus":false,"about_ca_system_score_codex":0.00005147929,"about_ca_system_score_gemma":0.0001539982,"about_ca_topic_candidate":false,"about_ca_topic_consensus":false,"about_ca_topic_score_codex":0.00003270706,"about_ca_topic_score_gemma":0.0003588257,"domain_scores_codex":[0.9981435,0.0001100948,0.0003755014,0.0005508778,0.0004581216,0.0003618867],"domain_scores_gemma":[0.9990813,0.0001488607,0.0001415422,0.0004244701,0.0001063327,0.00009747096],"domain_codex":null,"domain_gemma":null,"domain_candidate":null,"domain_consensus":null,"study_design_codex":"bench_or_experimental","study_design_gemma":"bench_or_experimental","study_design_scores_codex":[0.00005269956,0.0002380665,0.01783524,0.00003382444,0.000003573631,0.00003142164,0.0003314539,0.01386646,0.9641801,0.0006519904,0.00164733,0.001127912],"study_design_scores_gemma":[0.0004960531,0.0001266527,0.08989104,0.00002539331,0.00001792516,0.000006112703,0.00004527895,0.000695922,0.9049758,0.002325055,0.001183595,0.0002111379],"study_design_candidate":"bench_or_experimental","study_design_consensus":"bench_or_experimental","genre_codex":"empirical","genre_gemma":"empirical","genre_scores_codex":[0.996463,0.0004557394,0.0004019905,0.0004150176,0.0003154127,0.0005161373,0.0003774069,0.00002676977,0.001028516],"genre_scores_gemma":[0.9953734,0.00002768672,0.003205805,0.0006578037,0.0001097843,0.00003189435,0.0000298233,0.00003409844,0.0005296894],"genre_candidate":"empirical","genre_consensus":"empirical","teacher_disagreement_score":0.0720558,"threshold_uncertainty_score":0.8733153,"prediction_status":"machine_predicted_unvalidated"},"labels":[],"label_agreement":null},{"id":"W2084636668","doi":"10.1016/j.tins.2014.09.003","title":"Autophagy in Huntington disease and huntingtin in autophagy","year":2014,"lang":"en","type":"review","venue":"Trends in Neurosciences","topic":"Genetic Neurodegenerative Diseases","field":"Neuroscience","cited_by":341,"is_retracted":false,"has_abstract":false,"routes":{"ca_aff":true,"ca_fund":true,"ca_venue":false,"about_ca":false},"ca_institutions":"Child and Family Research Institute; University of British Columbia","funders":"Canadian Institutes of Health Research","keywords":"Autophagy; Huntingtin; Huntington's disease; Polyglutamine tract; Lysosome; Cell biology; Huntingtin Protein; Biology; Neurodegeneration; BAG3; Protein aggregation; Neuroscience; Disease; Genetics; Biochemistry; Medicine; Apoptosis","retraction":null,"screen_n_in":null,"score":{"opus":0.07152464519885082,"gpt":0.3645512117638758,"spread":0.293026566565025,"validation_status":"score_only:v0-immature-baseline"},"prediction":{"model_version":"codex-gemma-dda1882f352a","candidate_categories":["metaepi_narrow"],"consensus_categories":[],"category_scores_codex":[0.0006990616,0.0008003943,0.001687013,0.002765553,0.0001420039,0.0003002864,0.001310352,0.0001705785,0.00006372338],"category_scores_gemma":[0.002846103,0.0007114062,0.0002395612,0.004279287,0.00076452,0.0003086692,0.0005363959,0.0007472918,0.00002509781],"about_ca_system_candidate":false,"about_ca_system_consensus":false,"about_ca_system_score_codex":0.00008613441,"about_ca_system_score_gemma":0.0002984923,"about_ca_topic_candidate":false,"about_ca_topic_consensus":false,"about_ca_topic_score_codex":0.00007275322,"about_ca_topic_score_gemma":0.0002294237,"domain_scores_codex":[0.9926538,0.001643618,0.001323857,0.002578647,0.0007183368,0.001081762],"domain_scores_gemma":[0.9974715,0.0009822662,0.0005126873,0.0006923774,0.00001349596,0.0003276621],"domain_codex":null,"domain_gemma":null,"domain_candidate":null,"domain_consensus":null,"study_design_codex":"design_other","study_design_gemma":"not_applicable","study_design_scores_codex":[0.00001822546,0.0005850872,0.003081606,0.002964644,0.000001258244,0.0003900287,0.0001394811,0.0001919057,0.001317012,0.0008920812,0.00007187654,0.9903468],"study_design_scores_gemma":[0.003860683,0.0008964699,0.166525,0.0191803,0.0003560644,0.000100285,0.00006103115,0.007460821,0.01204046,0.001290054,0.7819086,0.0063202],"study_design_candidate":"design_other","study_design_consensus":null,"genre_codex":"review","genre_gemma":"review","genre_scores_codex":[0.2140737,0.7804659,0.000002693389,0.0001348807,0.002253781,0.001004163,0.00009217112,0.0001591056,0.00181354],"genre_scores_gemma":[0.1057272,0.8922148,0.00004327022,0.0004281072,0.0001123636,0.0002747145,0.000008582375,0.00008554116,0.001105399],"genre_candidate":"review","genre_consensus":"review","teacher_disagreement_score":0.9840266,"threshold_uncertainty_score":0.9995337,"prediction_status":"machine_predicted_unvalidated"},"labels":[],"label_agreement":null},{"id":"W2026062838","doi":"10.1523/jneurosci.1212-08.2008","title":"Growth of White Matter in the Adolescent Brain: Role of Testosterone and Androgen Receptor","year":2008,"lang":"en","type":"article","venue":"Journal of Neuroscience","topic":"Genetic Neurodegenerative Diseases","field":"Neuroscience","cited_by":339,"is_retracted":false,"has_abstract":true,"routes":{"ca_aff":true,"ca_fund":true,"ca_venue":false,"about_ca":false},"ca_institutions":"Université du Québec à Chicoutimi; Cégep de Jonquière; Centre Hospitalier de l’Université de Montréal; McGill University; Montreal Neurological Institute and Hospital","funders":"Canadian Institutes of Health Research; Heart And Stroke Foundation Of Quebec; Heart and Stroke Foundation of Canada","keywords":"White matter; Androgen receptor; Testosterone (patch); Endocrinology; Internal medicine; Sexual dimorphism; Androgen; Biology; Exon; Magnetic resonance imaging; Psychology; Gene; Medicine; Genetics; Hormone; Prostate cancer","retraction":null,"screen_n_in":null,"score":{"opus":0.03124548089200197,"gpt":0.2495839935736161,"spread":0.2183385126816141,"validation_status":"score_only:v0-immature-baseline"},"prediction":{"model_version":"codex-gemma-dda1882f352a","candidate_categories":[],"consensus_categories":[],"category_scores_codex":[0.0002517509,0.0001120137,0.0002128361,0.00016816,0.0001009057,0.00001536631,0.0005569498,0.0000190738,0.000009825904],"category_scores_gemma":[0.0007905825,0.00007557923,0.0000601009,0.0004907378,0.0005725208,0.0002127505,0.0001156063,0.0001618038,0.000001498317],"about_ca_system_candidate":false,"about_ca_system_consensus":false,"about_ca_system_score_codex":0.000007955512,"about_ca_system_score_gemma":0.00007807079,"about_ca_topic_candidate":false,"about_ca_topic_consensus":false,"about_ca_topic_score_codex":0.000001865659,"about_ca_topic_score_gemma":5.937532e-7,"domain_scores_codex":[0.9982272,0.0003118313,0.0004577123,0.0002262721,0.000592531,0.0001844878],"domain_scores_gemma":[0.999007,0.0001906697,0.0004318904,0.0001910022,0.00009615345,0.00008328988],"domain_codex":null,"domain_gemma":null,"domain_candidate":null,"domain_consensus":null,"study_design_codex":"bench_or_experimental","study_design_gemma":"bench_or_experimental","study_design_scores_codex":[0.00003139276,0.0001838196,0.1902226,0.0000132568,2.39356e-7,0.00002354779,0.0006249572,0.00003479557,0.8083904,0.00001730808,0.0004178754,0.00003970957],"study_design_scores_gemma":[0.0002259358,0.0002880918,0.4756823,0.00002513265,0.000003195092,0.0003994229,0.00004455878,0.0000385194,0.5230885,0.00004205198,0.0001170654,0.00004523322],"study_design_candidate":"bench_or_experimental","study_design_consensus":"bench_or_experimental","genre_codex":"empirical","genre_gemma":"empirical","genre_scores_codex":[0.9976398,0.00008701257,0.0000261096,0.001890019,0.0001290442,0.0001252939,0.00001137309,0.000001810175,0.0000894942],"genre_scores_gemma":[0.9961898,0.0001278494,0.00008491837,0.003533448,0.00002529806,0.000001376238,4.045978e-8,0.000007806792,0.0000294185],"genre_candidate":"empirical","genre_consensus":"empirical","teacher_disagreement_score":0.2854597,"threshold_uncertainty_score":0.3082032,"prediction_status":"machine_predicted_unvalidated"},"labels":[],"label_agreement":null},{"id":"W2722517739","doi":"10.1016/s1474-4422(17)30161-8","title":"Identification of genetic variants associated with Huntington's disease progression: a genome-wide association study","year":2017,"lang":"en","type":"article","venue":"The Lancet Neurology","topic":"Genetic Neurodegenerative Diseases","field":"Neuroscience","cited_by":332,"is_retracted":false,"has_abstract":false,"routes":{"ca_aff":true,"ca_fund":false,"ca_venue":false,"about_ca":false},"ca_institutions":"University of British Columbia","funders":"UCLH Biomedical Research Centre; Medical Research Council; Seventh Framework Programme; European Commission; Rosetrees Trust; Guarantors of Brain; Research Councils UK; Brain Research Trust; Novartis; National Institute for Health and Care Research; Roche; Wellcome Trust; Agence Nationale de la Recherche; CHDI Foundation","keywords":"Huntington's disease; Disease; Genetic association; Genome-wide association study; Trinucleotide repeat expansion; Genetics; Medicine; Oncology; Biology; Bioinformatics; Internal medicine; Allele; Gene; Genotype; Single-nucleotide polymorphism","retraction":null,"screen_n_in":null,"score":{"opus":0.03287868661296017,"gpt":0.298027486192952,"spread":0.2651487995799918,"validation_status":"score_only:v0-immature-baseline"},"prediction":{"model_version":"codex-gemma-dda1882f352a","candidate_categories":["metaresearch"],"consensus_categories":[],"category_scores_codex":[0.0005090791,0.0001769489,0.0003164267,0.0000511425,0.0006350132,0.0001419927,0.001044464,0.00004695915,0.0000211135],"category_scores_gemma":[0.008984892,0.000123686,0.00004951581,0.0001328374,0.0002362275,0.0001100479,0.000227947,0.0002076896,0.00002326776],"about_ca_system_candidate":false,"about_ca_system_consensus":false,"about_ca_system_score_codex":0.00002015923,"about_ca_system_score_gemma":0.00007664139,"about_ca_topic_candidate":false,"about_ca_topic_consensus":false,"about_ca_topic_score_codex":0.000005830817,"about_ca_topic_score_gemma":0.00002315222,"domain_scores_codex":[0.9970654,0.001149329,0.0003415845,0.0005570825,0.0004996014,0.0003870464],"domain_scores_gemma":[0.9965979,0.0006988299,0.001211598,0.001266359,0.0001397857,0.00008550902],"domain_codex":null,"domain_gemma":null,"domain_candidate":null,"domain_consensus":null,"study_design_codex":"observational","study_design_gemma":"observational","study_design_scores_codex":[0.0003239581,0.0004487979,0.9453396,0.000007843282,0.00002286685,0.00003297135,0.0001965341,0.0003034161,0.05302956,0.00003774684,0.0001196902,0.0001370804],"study_design_scores_gemma":[0.001030689,0.0003687281,0.9798402,0.000006090993,0.0001024595,8.019112e-7,0.000006536148,0.0002685198,0.01780339,0.0004085793,0.00005105802,0.0001129614],"study_design_candidate":"observational","study_design_consensus":"observational","genre_codex":"empirical","genre_gemma":"empirical","genre_scores_codex":[0.9934479,0.00001362331,0.00004431031,0.005067486,0.0002527378,0.0008966141,0.00005385715,0.00005941077,0.0001640706],"genre_scores_gemma":[0.9980258,0.0000173042,0.000007117727,0.001459935,0.0001424294,0.0001277898,0.000003182095,0.00002863264,0.0001877932],"genre_candidate":"empirical","genre_consensus":"empirical","teacher_disagreement_score":0.03522616,"threshold_uncertainty_score":0.9993628,"prediction_status":"machine_predicted_unvalidated"},"labels":[],"label_agreement":null},{"id":"W2041882228","doi":"10.1038/nn1702","title":"Palmitoylation of huntingtin by HIP14is essential for its trafficking and function","year":2006,"lang":"en","type":"article","venue":"Nature Neuroscience","topic":"Genetic Neurodegenerative Diseases","field":"Neuroscience","cited_by":332,"is_retracted":false,"has_abstract":false,"routes":{"ca_aff":true,"ca_fund":false,"ca_venue":false,"about_ca":false},"ca_institutions":"BC Children's Hospital; University of British Columbia","funders":"National Institute of Neurological Disorders and Stroke; Wellcome Trust","keywords":"Palmitoylation; Huntingtin; Polyglutamine tract; Cell biology; Mutant; Chemistry; Biology; Cysteine; Biochemistry; Enzyme; Gene","retraction":null,"screen_n_in":null,"score":{"opus":0.01102342915954623,"gpt":0.2583234966930781,"spread":0.2473000675335318,"validation_status":"score_only:v0-immature-baseline"},"prediction":{"model_version":"codex-gemma-dda1882f352a","candidate_categories":[],"consensus_categories":[],"category_scores_codex":[0.0001119743,0.0001481414,0.000128046,0.0001058761,0.0002439722,0.00007918473,0.0002013847,0.00009613564,0.000004240564],"category_scores_gemma":[0.001180563,0.0001435459,0.00005140966,0.0004690727,0.0001778306,0.0002269698,0.00004703727,0.0001709566,8.720231e-7],"about_ca_system_candidate":false,"about_ca_system_consensus":false,"about_ca_system_score_codex":0.000008337347,"about_ca_system_score_gemma":0.00003452767,"about_ca_topic_candidate":false,"about_ca_topic_consensus":false,"about_ca_topic_score_codex":0.000002355465,"about_ca_topic_score_gemma":0.000002368491,"domain_scores_codex":[0.9983652,0.00008218653,0.0002263288,0.0006636712,0.0004019689,0.000260644],"domain_scores_gemma":[0.9992931,0.0002307732,0.000180612,0.0001506706,0.00009007093,0.00005478976],"domain_codex":null,"domain_gemma":null,"domain_candidate":null,"domain_consensus":null,"study_design_codex":"bench_or_experimental","study_design_gemma":"bench_or_experimental","study_design_scores_codex":[0.00002921265,0.0000630928,0.0007219547,0.00002248504,1.689944e-7,6.502692e-7,0.00001070426,0.0004613445,0.9956236,0.001529689,0.0007041986,0.0008328793],"study_design_scores_gemma":[0.0003081112,0.000123099,0.01483974,0.000007075248,0.00001251296,0.00000444226,0.00000426293,0.005156436,0.9775215,0.0001962776,0.001704325,0.0001222222],"study_design_candidate":"bench_or_experimental","study_design_consensus":"bench_or_experimental","genre_codex":"empirical","genre_gemma":"empirical","genre_scores_codex":[0.9945758,0.0002035827,0.003513331,0.0001942329,0.0008224593,0.0003832525,0.00007087262,0.00005558827,0.0001808989],"genre_scores_gemma":[0.998895,0.00001300802,0.000110269,0.0006448395,0.0001172912,0.00001948527,0.000004838545,0.0000167574,0.0001784738],"genre_candidate":"empirical","genre_consensus":"empirical","teacher_disagreement_score":0.01810212,"threshold_uncertainty_score":0.5853633,"prediction_status":"machine_predicted_unvalidated"},"labels":[],"label_agreement":null},{"id":"W2168583567","doi":"10.1523/jneurosci.0590-05.2005","title":"Cognitive Dysfunction Precedes Neuropathology and Motor Abnormalities in the YAC128 Mouse Model of Huntington's Disease","year":2005,"lang":"en","type":"article","venue":"Journal of Neuroscience","topic":"Genetic Neurodegenerative Diseases","field":"Neuroscience","cited_by":328,"is_retracted":false,"has_abstract":true,"routes":{"ca_aff":true,"ca_fund":true,"ca_venue":false,"about_ca":false},"ca_institutions":"University of British Columbia","funders":"Canadian Institutes of Health Research; Cure Huntington's Disease Initiative; Killam Trusts; Michael Smith Health Research BC; Huntington Society of Canada; Hereditary Disease Foundation","keywords":"Neuropathology; Prepulse inhibition; Huntington's disease; Psychology; Neuroscience; Habituation; Cognition; Disease; Medicine; Internal medicine; Psychiatry; Schizophrenia (object-oriented programming)","retraction":null,"screen_n_in":null,"score":{"opus":0.04308505777376605,"gpt":0.2796219212640588,"spread":0.2365368634902928,"validation_status":"score_only:v0-immature-baseline"},"prediction":{"model_version":"codex-gemma-dda1882f352a","candidate_categories":[],"consensus_categories":[],"category_scores_codex":[0.0004210353,0.0001559895,0.0001974147,0.0002360807,0.0001382301,0.00007866088,0.0004475148,0.0000219147,0.000003268779],"category_scores_gemma":[0.0029966,0.0001117015,0.00007700866,0.0002966563,0.000636932,0.0005912181,0.00009743308,0.0002404668,0.000001298467],"about_ca_system_candidate":false,"about_ca_system_consensus":false,"about_ca_system_score_codex":0.00001224563,"about_ca_system_score_gemma":0.0001272523,"about_ca_topic_candidate":false,"about_ca_topic_consensus":false,"about_ca_topic_score_codex":0.000001440516,"about_ca_topic_score_gemma":0.000001755927,"domain_scores_codex":[0.9979274,0.0005331703,0.0004599502,0.0003222096,0.0004962532,0.000261067],"domain_scores_gemma":[0.9987067,0.0004463195,0.0004240452,0.0001747587,0.0001329875,0.0001151678],"domain_codex":null,"domain_gemma":null,"domain_candidate":null,"domain_consensus":null,"study_design_codex":"bench_or_experimental","study_design_gemma":"bench_or_experimental","study_design_scores_codex":[0.0001503372,0.0002150661,0.003301877,0.00001565318,3.508844e-7,0.00001254094,0.0004099132,0.00636067,0.9888004,0.0001519439,0.00001434408,0.0005668751],"study_design_scores_gemma":[0.0006699476,0.0005242918,0.1304944,0.00003782715,0.00003166035,0.00006582834,0.000191873,0.01700478,0.8506291,0.0001701277,0.00003875502,0.0001414644],"study_design_candidate":"bench_or_experimental","study_design_consensus":"bench_or_experimental","genre_codex":"empirical","genre_gemma":"empirical","genre_scores_codex":[0.9981802,0.00005601297,0.0007002759,0.0005368817,0.0001903363,0.0001965079,0.0000451479,0.000007980402,0.00008663148],"genre_scores_gemma":[0.9972965,0.000219623,0.0000865343,0.002182362,0.00005466531,0.000007349421,1.18267e-7,0.00001209485,0.0001407873],"genre_candidate":"empirical","genre_consensus":"empirical","teacher_disagreement_score":0.1381714,"threshold_uncertainty_score":0.4555054,"prediction_status":"machine_predicted_unvalidated"},"labels":[],"label_agreement":null},{"id":"W2130668838","doi":"10.1093/hmg/ddm133","title":"Mutant huntingtin's effects on striatal gene expression in mice recapitulate changes observed in human Huntington's disease brain and do not differ with mutant huntingtin length or wild-type huntingtin dosage","year":2007,"lang":"en","type":"article","venue":"Human Molecular Genetics","topic":"Genetic Neurodegenerative Diseases","field":"Neuroscience","cited_by":322,"is_retracted":false,"has_abstract":true,"routes":{"ca_aff":true,"ca_fund":false,"ca_venue":false,"about_ca":false},"ca_institutions":"Child and Family Research Institute; University of British Columbia","funders":"","keywords":"Huntingtin; Biology; Huntington's disease; Mutant; Huntingtin Protein; Molecular biology; Transgene; Gene expression; Polyglutamine tract; Wild type; Gene; Genetically modified mouse; Genetics; Internal medicine; Disease","retraction":null,"screen_n_in":null,"score":{"opus":0.02861731872987026,"gpt":0.2713400562446262,"spread":0.242722737514756,"validation_status":"score_only:v0-immature-baseline"},"prediction":{"model_version":"codex-gemma-dda1882f352a","candidate_categories":["metaepi_narrow"],"consensus_categories":[],"category_scores_codex":[0.0009791295,0.001069085,0.0008632248,0.000805199,0.0005495944,0.0003861263,0.0008193032,0.000261722,0.00004322296],"category_scores_gemma":[0.001701679,0.0009721175,0.0001400259,0.001106197,0.0003645683,0.0001402734,0.0006375376,0.0007864416,0.00001066539],"about_ca_system_candidate":false,"about_ca_system_consensus":false,"about_ca_system_score_codex":0.0001252871,"about_ca_system_score_gemma":0.0001231277,"about_ca_topic_candidate":false,"about_ca_topic_consensus":false,"about_ca_topic_score_codex":0.00004811737,"about_ca_topic_score_gemma":0.0004718189,"domain_scores_codex":[0.9926968,0.001005826,0.001056795,0.002383152,0.001227175,0.001630257],"domain_scores_gemma":[0.9967024,0.0007742174,0.0006421102,0.001160571,0.0001319805,0.000588733],"domain_codex":null,"domain_gemma":null,"domain_candidate":null,"domain_consensus":null,"study_design_codex":"bench_or_experimental","study_design_gemma":"bench_or_experimental","study_design_scores_codex":[0.000905257,0.0005429845,0.01969253,0.0002300866,0.00001502427,0.001255788,0.0006068984,0.0005880601,0.9746714,0.000120031,0.00001011446,0.001361856],"study_design_scores_gemma":[0.003488688,0.000880168,0.2001262,0.0006402112,0.00006742114,0.000009340718,0.00009578851,0.0001776271,0.7934166,0.00008650473,0.00007453569,0.0009369073],"study_design_candidate":"bench_or_experimental","study_design_consensus":"bench_or_experimental","genre_codex":"empirical","genre_gemma":"empirical","genre_scores_codex":[0.9969239,0.0002208753,0.0001353296,0.000252263,0.0002065803,0.001975994,0.00005203109,0.000154517,0.00007845986],"genre_scores_gemma":[0.9960709,0.00007962561,0.001393495,0.001711848,0.0002249491,0.00008633917,0.00005890913,0.0002710023,0.0001029928],"genre_candidate":"empirical","genre_consensus":"empirical","teacher_disagreement_score":0.1812547,"threshold_uncertainty_score":0.9992729,"prediction_status":"machine_predicted_unvalidated"},"labels":[],"label_agreement":null},{"id":"W2017901987","doi":"10.1016/j.neuroscience.2011.08.052","title":"Pathophysiology of Huntington's disease: time-dependent alterations in synaptic and receptor function","year":2011,"lang":"en","type":"review","venue":"Neuroscience","topic":"Genetic Neurodegenerative Diseases","field":"Neuroscience","cited_by":320,"is_retracted":false,"has_abstract":false,"routes":{"ca_aff":true,"ca_fund":true,"ca_venue":false,"about_ca":false},"ca_institutions":"University of British Columbia","funders":"National Institute of Neurological Disorders and Stroke; Canadian Institutes of Health Research","keywords":"Huntington's disease; Neuroscience; Excitotoxicity; Glutamate receptor; Striatum; Medium spiny neuron; Biology; NMDA receptor; Huntingtin; Neurodegeneration; Receptor; Internal medicine; Disease; Medicine; Genetics; Dopamine","retraction":null,"screen_n_in":null,"score":{"opus":0.04996941540106634,"gpt":0.294920815992814,"spread":0.2449514005917477,"validation_status":"score_only:v0-immature-baseline"},"prediction":{"model_version":"codex-gemma-dda1882f352a","candidate_categories":["metaepi_narrow"],"consensus_categories":[],"category_scores_codex":[0.0001597266,0.0003914642,0.0007909627,0.0003687032,0.0001459253,0.00005485462,0.0005518697,0.000095969,0.00007686383],"category_scores_gemma":[0.001706925,0.0003444219,0.0001562784,0.0007179749,0.0005634893,0.0001877126,0.0003060383,0.0002845747,0.00008891636],"about_ca_system_candidate":false,"about_ca_system_consensus":false,"about_ca_system_score_codex":0.00002865136,"about_ca_system_score_gemma":0.0002922144,"about_ca_topic_candidate":false,"about_ca_topic_consensus":false,"about_ca_topic_score_codex":0.000005267971,"about_ca_topic_score_gemma":0.000001441511,"domain_scores_codex":[0.9965293,0.0008301832,0.0006037827,0.001334577,0.0003096281,0.0003924761],"domain_scores_gemma":[0.9984075,0.0003318687,0.0004304922,0.0005780762,0.00004284328,0.0002091878],"domain_codex":null,"domain_gemma":null,"domain_candidate":null,"domain_consensus":null,"study_design_codex":"design_other","study_design_gemma":"not_applicable","study_design_scores_codex":[0.0001102753,0.001050577,0.0001817912,0.007179529,0.000007743693,0.00003837811,0.0001395075,0.0001461821,0.4136076,0.002770397,0.0002737157,0.5744943],"study_design_scores_gemma":[0.001958607,0.003834529,0.008327233,0.01406244,0.002020981,0.00007164205,0.00002770657,0.001898461,0.05827218,0.004339952,0.8995105,0.005675744],"study_design_candidate":"not_applicable","study_design_consensus":null,"genre_codex":"review","genre_gemma":"review","genre_scores_codex":[0.03766006,0.9570897,0.0001512161,0.00003702448,0.001966889,0.002178133,0.000449618,0.0001195712,0.0003478532],"genre_scores_gemma":[0.01069531,0.9882923,0.00003040602,0.0003079044,0.0000523357,0.0001738572,0.000008900041,0.00004737635,0.0003916441],"genre_candidate":"review","genre_consensus":"review","teacher_disagreement_score":0.8992368,"threshold_uncertainty_score":0.9999008,"prediction_status":"machine_predicted_unvalidated"},"labels":[],"label_agreement":null}]}