{"id":"W1966246090","doi":"10.1371/journal.pone.0094157","title":"Complex I Deficiency Due to Selective Loss of Ndufs4 in the Mouse Heart Results in Severe Hypertrophic Cardiomyopathy","year":2014,"lang":"en","type":"article","venue":"PLoS ONE","topic":"Mitochondrial Function and Pathology","field":"Biochemistry, Genetics and Molecular Biology","cited_by":53,"is_retracted":false,"has_abstract":true,"ca_institutions":"","funders":"Servier; Canadian Institutes of Health Research; Gates Cambridge Trust; Medical Research Council; Cambridge Trust; British Heart Foundation","keywords":"Hypertrophic cardiomyopathy; Mitochondrion; Cardiomyopathy; Internal medicine; Oxidative phosphorylation; Mitochondrial respiratory chain; Fibrosis; Reactive oxygen species; Endocrinology; Heart failure; Biology; Medicine; Cardiology; Cell biology; Biochemistry","routes":{"ca_aff":false,"ca_fund":true,"ca_venue":false,"about_ca":false,"invisible_to_affiliation_only":true},"retraction":null,"screen":{"n_in":0,"stratum":"fund_new","weight":1678.9,"opus":{"tier":"OUT","genre":"empirical","about_ca":false,"confidence":"high","reason":"Mouse genetic model of complex I deficiency and cardiomyopathy."},"gpt":{"tier":"OUT","genre":"empirical","about_ca":false,"confidence":"high","reason":"The study investigates mitochondrial dysfunction and cardiomyopathy in mice."},"grok":{"tier":"OUT","genre":"empirical","about_ca":false,"confidence":"high","reason":"Mouse model of cardiac complex I deficiency; biomedical pathophysiology."}}}