{"id":"W2122091775","doi":"10.1038/nature13909","title":"Ischaemic accumulation of succinate controls reperfusion injury through mitochondrial ROS","year":2014,"lang":"en","type":"article","venue":"Nature","topic":"Mitochondrial Function and Pathology","field":"Biochemistry, Genetics and Molecular Biology","cited_by":2804,"is_retracted":false,"has_abstract":false,"ca_institutions":"","funders":"Biotechnology and Biological Sciences Research Council; National Heart, Lung, and Blood Institute; Medical Research Council; Canadian Institutes of Health Research; British Heart Foundation","keywords":"Reactive oxygen species; Mitochondrial ROS; Reperfusion injury; Mitochondrion; Ischemia; Citric acid cycle; Cell biology; Succinate dehydrogenase; Biology; In vivo; Biochemistry; Chemistry; Pharmacology; Metabolism; Medicine; Internal medicine","routes":{"ca_aff":false,"ca_fund":true,"ca_venue":false,"about_ca":false,"invisible_to_affiliation_only":true},"retraction":null,"screen":null}