{"id":"W2766300480","doi":"10.1016/j.radonc.2017.09.043","title":"ATR/CHK1 inhibitors and cancer therapy","year":2017,"lang":"en","type":"review","venue":"Radiotherapy and Oncology","topic":"DNA Repair Mechanisms","field":"Biochemistry, Genetics and Molecular Biology","cited_by":388,"is_retracted":false,"has_abstract":false,"ca_institutions":"","funders":"National Cancer Institute; School of Medicine, Case Western Reserve University; School of Medicine; Western University; Case Comprehensive Cancer Center, Case Western Reserve University; Comprehensive Cancer Center, City of Hope","keywords":"CHEK1; DNA damage; Synthetic lethality; Cancer research; DNA repair; Ataxia-telangiectasia; Cancer cell; G2-M DNA damage checkpoint; Mitosis; Cell cycle checkpoint; Cell cycle; Cancer; Biology; Kinase; DNA; Cell biology; Genetics","routes":{"ca_aff":false,"ca_fund":true,"ca_venue":false,"about_ca":false,"invisible_to_affiliation_only":true},"retraction":null,"screen":null,"machine_scores":{"provisional":true,"baseline":true,"maturity_gate_passed":false,"score_opus":0.04608454608422507,"score_gpt":0.3793831738759061,"score_spread":0.3332986277916811,"validation_status":"score_only:v0-immature-baseline","note":"Baseline scores from an immature model (maturity gate not passed). Scores rank; they never assert a category."}}