MétaCan
← tous les travaux

Quantifying the mechanisms for segmental duplications in mammalian genomes by statistical analysis and modeling

2005· article· en· 51 citations· W1972491078 sur OpenAlex· 10.1073/pnas.0407957102

Pourquoi ce travail est-il dans la base ?

Une base qui oublie comment elle a trouvé un travail ne peut pas être vérifiée. Voici les voies qui ont admis celui-ci.

Organisme subventionnaire canadienUn organisme canadien l'a financé. Le travail peut ne porter aucune affiliation canadienne.

Aucune affiliation canadienne. Une base fondée sur la seule affiliation (le devis habituel) n'aurait jamais vu ce travail. C'est l'un des travaux qui justifient l'inversion de la base.

Le tri à trois modèles

les 1 000 travaux triés →

Les trois modèles l'ont jugé hors champ.

strate : fund_new · poids de sondage : 1678.90 (l'échantillon est stratifié ; tout taux calculé sans le poids est faux)
Claude Opus 4.8OUT
genre : empirical
porte sur le Canada: non
confiance: high

Statistical modelling of the molecular mechanisms of segmental duplication in mammalian genomes.

GPT-5.6 (high)OUT
genre : empirical
porte sur le Canada: non
confiance: high

It models mechanisms of mammalian genomic duplication, not research methods or research governance.

Grok 4.5OUT
genre : empirical
porte sur le Canada: non
confiance: high

Genomics study of mechanisms of mammalian segmental duplications; domain biology.

Résumé

A large number of the segmental duplications in mammalian genomes have been cataloged by genome-wide sequence analyses. The molecular mechanisms involved in these duplications mostly remain a matter of speculation. To uncover, test, and further quantify the hypotheses on the mechanisms for the recent duplications in the mammalian genomes, we have performed a series of statistical analyses on the sequences flanking the duplicated segments and proposed a dynamic model for the duplication process. The model, when applied to the human duplication data, indicates that approximately 30% of the recent human segmental duplications were caused by a recombination-like mechanism, among which 12% were mediated by the most recently active repeat, Alu. But a significant proportion of the duplications are caused by some mechanism independent of the repeat distribution. A less sure but similar picture is found in the rodent genomes. A further analysis on the physical features of the flanking sequences suggests that one of the uncharacterized duplication mechanisms shared by the mammalian genomes is surprisingly well correlated with the physical instability in the DNA sequences.

Conservé avec la notice de tri, où il sert de preuve aux étiquettes ci-dessus.

La notice

Revue
Proceedings of the National Academy of Sciences
Thématique
Chromosomal and Genetic Variations
Domaine
Agricultural and Biological Sciences
Établissements canadiens
Organismes subventionnaires
U.S. Air ForceDefense Advanced Research Projects AgencyYork UniversityOffice of ScienceAir Force Research LaboratoryDivision of Mathematical SciencesAdvanced Research Projects AgencyNational Institutes of HealthNational Science Foundation
Mots-clés
Gene duplicationGenomeSegmental duplicationBiologyGeneticsGenome instabilityHuman genomeSequence (biology)Mechanism (biology)Evolutionary biologyComputational biologyDNAGeneGene familyDNA damage
Résumé présent dans OpenAlex
oui