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A Genome-Wide Association Study Identifies <i>LIPA</i> as a Susceptibility Gene for Coronary Artery Disease

2011· review· en· W2125746244 on OpenAlexfundno aff
Philipp S. Wild, Tanja Zeller, Arne Schillert, Silke Szymczak, Christoph Sinning, Arne Deiseroth, Renate B. Schnabel, Edith Lubos, Till Keller, Medea Eleftheriadis, Christoph Bickel, Hans J. Rupprecht, Sandra Wilde, Heidi Rossmann, Patrick Diemert, L. Adrienne Cupples, Claire Perret, Jeanette Erdmann, Klaus Stark, Marcus E. Kleber, Stephen E. Epstein, Benjamin F. Voight, Kari Kuulasmaa, Mingyao Li, Arne Schäfer, Norman Klopp, Peter S. Braund, Hendrik B. Sager, Serkalem Demissie, Carole Proust, Inke R. König, H.-Erich Wichmann, Wibke Reinhard, Michael M. Hoffmann, Jarmo Virtamo, Mary Susan Burnett, David S. Siscovick, Per Wiklund, Liming Qu, Nour Eddine El Mokthari, John R. Thompson, Annette Peters, Albert V. Smith, Emmanuelle Yon, Jens Baumert, Christian Hengstenberg, Winfried März, Philippe Amouyel, Joseph M. Devaney, Stephen M. Schwartz, Olli Saarela, Nehal N. Mehta, Deborah C. Rubin, Kaisa Silander, Alistair S. Hall, Jean Ferrières, Tamara B. Harris, Olle Melander, Frank Kee, Håkon Håkonarson, Juergen Schrezenmeir, Vilmundur Guðnason, Roberto Elosúa, Dominique Arveiler, Alun Evans, Daniel J. Rader, Thomas Illig, Stefan Schreiber, Joshua C. Bis, David Altshuler, Maryam Kavousi, J. C. M. Witteman, André G. Uitterlinden, Albert Hofman, Aaron R. Folsom, Maja Barbalić, Eric Boerwinkle, Sekar Kathiresan, Muredach P. Reilly, Christopher J. O’Donnell, Nilesh J. Samani, Heribert Schunkert, François Cambien, Karl J. Lackner, Laurence Tiret, Veikko Salomaa, Thomas Münzel, Andreas Ziegler, Stefan Blankenberg

Bibliographic record

VenueCirculation Cardiovascular Genetics · 2011
Typereview
Languageen
FieldBiochemistry, Genetics and Molecular Biology
TopicGenetic Associations and Epidemiology
Canadian institutionsnot available
FundersInstitute of GeneticsNational Heart, Lung, and Blood InstituteUniversitätsmedizin der Johannes Gutenberg-Universität MainzEconomic and Social Research CouncilMedical Research CouncilNational Institutes of HealthTerveyden ja hyvinvoinnin laitosHáskóli ÍslandsUniversität zu LübeckHjartaverndUniversitätsklinikum RegensburgChildren's Hospital of PhiladelphiaLunds UniversitetErasmus Medisch CentrumMedStar Health Research InstituteChristian-Albrechts-Universität zu KielUniversity of LeicesterInstitute Pasteur De LilleUmeå UniversitetBundesministerium für Bildung und ForschungUniversity of LeedsUniversity of MinnesotaMassachusetts General HospitalNational Institute on AgingNational Institute for Health and Care ResearchBroad InstituteUnited Kingdom Clinical Research CollaborationInstitut National de la Santé et de la Recherche MédicaleUniversity of WashingtonBritish Heart FoundationWellcome TrustUniversity of Pennsylvania
KeywordsSingle-nucleotide polymorphismGenome-wide association studyExpression quantitative trait lociLocus (genetics)GeneticsBiologyGenetic associationGeneOdds ratioCoronary artery diseaseGenotypeMedicineInternal medicine

Abstract

fetched live from OpenAlex

BACKGROUND: eQTL analyses are important to improve the understanding of genetic association results. We performed a genome-wide association and global gene expression study to identify functionally relevant variants affecting the risk of coronary artery disease (CAD). METHODS AND RESULTS: In a genome-wide association analysis of 2078 CAD cases and 2953 control subjects, we identified 950 single-nucleotide polymorphisms (SNPs) that were associated with CAD at P<10(-3). Subsequent in silico and wet-laboratory replication stages and a final meta-analysis of 21 428 CAD cases and 38 361 control subjects revealed a novel association signal at chromosome 10q23.31 within the LIPA (lysosomal acid lipase A) gene (P=3.7×10(-8); odds ratio, 1.1; 95% confidence interval, 1.07 to 1.14). The association of this locus with global gene expression was assessed by genome-wide expression analyses in the monocyte transcriptome of 1494 individuals. The results showed a strong association of this locus with expression of the LIPA transcript (P=1.3×10(-96)). An assessment of LIPA SNPs and transcript with cardiovascular phenotypes revealed an association of LIPA transcript levels with impaired endothelial function (P=4.4×10(-3)). CONCLUSIONS: The use of data on genetic variants and the addition of data on global monocytic gene expression led to the identification of the novel functional CAD susceptibility locus LIPA, located on chromosome 10q23.31. The respective eSNPs associated with CAD strongly affect LIPA gene expression level, which was related to endothelial dysfunction, a precursor of CAD.

Fetched live from OpenAlex and de-inverted. Abstracts are not stored in this database: the inverted indexes are 8.6 GB of the frame’s 9.3 GB of text, and the host has 13 GB free.

How this classification was reachedexpand

Full frame machine prediction

Teacher imitation

Not calibrated prevalence, not ground truth. Human validation pending. The Gemma side is a direct model label for every work in the frame, read from the title-only record. The Codex side is a classifier learned from the 10,348 direct Codex labels and calibrated to design-weighted sample rates; fields without enough sample support carry no Codex call. Candidate is the union of the two sides; consensus is their intersection. These outputs are machine_predicted_unvalidated and are not human labels.

metaresearch head score (Codex)0.001
metaresearch head score (Gemma)0.002
Version: metacan-v3-hybrid-931329e0061cValidation status: machine_predicted_unvalidated
Candidate categoriesnone
Consensus categoriesnone
DomainCandidate signal: none · Consensus signal: none
Study designCandidate signal: Observational · Consensus signal: Observational
GenreCandidate signal: Review · Consensus signal: none
Teacher disagreement score0.003
Threshold uncertainty score0.011

Distilled classifier scores by category (both heads)

CategoryCodexGemma
Metaresearch0.0010.002
Meta-epidemiology (narrow)0.0010.000
Meta-epidemiology (broad)0.0010.001
Bibliometrics0.0010.001
Science and technology studies0.0000.001
Scholarly communication0.0010.000
Open science0.0000.001
Research integrity0.0010.001
Insufficient payload (model declined to judge)0.0030.000

Machine scores (provisional)

The two teacher heads of the student model, read on this work. A score orders the frame for review; it never asserts a category, and the validation status ships verbatim with every row.

Baseline scores from an immature model (maturity gate not passed, 7 training rounds). Scores rank; they never assert a category.

Opus teacher head0.041
GPT teacher head0.293
Teacher spread0.253 · how far apart the two teachers sit on this one work
Validation statusscore_only:v0-immature-baseline · verbatim from the scoring run: score_only means the number may rank works, and no category label ships from it

Classification

machine, unvalidated

Machine predicted; a candidate call from one source (direct Gemma or distilled Codex), not a consensus.

The models applied no category: nothing in the taxonomy fit this work.
Study designObservational
Domainnot available
GenreReview

How this classification was reached, model by model and score by score, is at the end of the page under "How this classification was reached".

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Citations152
Published2011
Admission routes1
Has abstractyes

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