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Dual ligand/receptor interactions activate urothelial defenses against uropathogenic E. coli

2015· article· en· 18 citations· W2414157519 sur OpenAlex· 10.1038/srep16234

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Claude Opus 4.8OUT
genre : empirical
porte sur le Canada: non
confiance: high

Molecular immunology of urothelial defense against uropathogenic E. coli.

GPT-5.6 (high)OUT
genre : empirical
porte sur le Canada: non
confiance: high

This is a biomedical study of urothelial defenses against infection.

Grok 4.5OUT
genre : empirical
porte sur le Canada: non
confiance: high

Host–pathogen immunology study of urothelial defenses against UPEC.

Résumé

During urinary tract infection (UTI), the second most common bacterial infection, dynamic interactions take place between uropathogenic E. coli (UPEC) and host urothelial cells. While significant strides have been made in the identification of the virulence factors of UPEC, our understanding of how the urothelial cells mobilize innate defenses against the invading UPEC remains rudimentary. Here we show that mouse urothelium responds to the adhesion of type 1-fimbriated UPEC by rapidly activating the canonical NF-κB selectively in terminally differentiated, superficial (umbrella) cells. This activation depends on a dual ligand/receptor system, one between FimH adhesin and uroplakin Ia and another between lipopolysaccharide and Toll-like receptor 4. When activated, all the nuclei (up to 11) of a multinucleated umbrella cell are affected, leading to significant amplification of proinflammatory signals. Intermediate and basal cells of the urothelium undergo NF-κB activation only if the umbrella cells are detached or if the UPEC persistently express type 1-fimbriae. Inhibition of NF-κB prevents the urothelium from clearing the intracellular bacterial communities, leading to prolonged bladder colonization by UPEC. Based on these data, we propose a model of dual ligand/receptor system in innate urothelial defenses against UPEC.

Conservé avec la notice de tri, où il sert de preuve aux étiquettes ci-dessus.

La notice

Revue
Scientific Reports
Thématique
Urinary Tract Infections Management
Domaine
Medicine
Établissements canadiens
Organismes subventionnaires
National Institute of Diabetes and Digestive and Kidney DiseasesNational Institute of Allergy and Infectious DiseasesYork UniversityBiomedical Laboratory Research and Development, VA Office of Research and DevelopmentSchool of Medicine, New York UniversityNational Institutes of HealthOffice of Research and DevelopmentU.S. Department of Veterans Affairs
Mots-clés
ReceptorLigand (biochemistry)Cancer researchCell biologyChemistryComputational biologyBiologyGenetics
Résumé présent dans OpenAlex
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