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Interaction between maternal 5-HTT genotype and prenatal stress exposure, confirmation in two independent samples (P2.229)

2015· article· en· W1016824312 on OpenAlexaff
David Q. Beversdorf, Patrick Hecht, Michael R. Tilley, Melissa M. Hudson, Susan L. Connors, Xudong Liu

Bibliographic record

VenueNeurology · 2015
Typearticle
Languageen
FieldNeuroscience
TopicAutism Spectrum Disorder Research
Canadian institutionsQueen's University
Fundersnot available
KeywordsGenotypePrenatal stressMedicinePregnancyBiologyGeneticsGestationGene

Abstract

fetched live from OpenAlex

OBJECTIVE: Examine the interaction between maternal 5-HTT genotype and prenatal stress exposure in autism. BACKGROUND: Recent studies suggest prenatal psychosocial stressors during specific prenatal epochs increase the risk of autism. Absence of a 44bp segment of the serotonin transporter gene (5-HTT) promoter region is related to stress responsivity, and has inconsistently been associated with autism. If maternal presence of at least one copy this stress-associated short allele (S-allele) of 5-HTT increases risk of autism with prenatal stress exposure, one would expect the prenatal stress exposure in autism to be more frequently observed in mothers with the S-allele. DESIGN/METHODS: Sixty mothers of children with autism at University of Missouri, and 99 at Queen's University were genotyped, accompanied by surveys regarding prenatal psychosocial stress exposure, and timing of these stressors, as with our previous work. Stress exposure incidence was compared between mothers carrying the S-allele and mothers lacking the S-allele using Chi-square, performed separately for the previously reported critical window of months 5-6 of gestation, and for the entire pregnancy. RESULTS: For both samples, stress exposure during months 5-6 of gestation was significantly higher among mothers with the S-allele (Missouri-p=0007, Queen's-p=0.037), with no relationship for the entire pregnancy (p=0.13) for Missouri, but a significant relationship for the entire pregnancy (0.018) for Queen's. To determine whether the maternal S-allele might increase recall of prenatal stress, the stress surveys were also completed for unaffected siblings pregnancies. Reported stress exposure was not higher in S-allele mothers for unaffected sibling pregnancies. CONCLUSIONS: These data suggest that maternal genotype for stress susceptibility genes is a critical factor in the effect of prenatal stress exposure on autism. Further understanding of the mechanism may eventually allow the possibility of prevention in some cases. Study Supported by: Mizzou Advantage, University of Missouri Mission Enhancement Funds, Ongwanada Resource Center

Fetched live from OpenAlex and de-inverted. Abstracts are not stored in this database: the inverted indexes are 8.6 GB of the frame’s 9.3 GB of text, and the host has 13 GB free.

How this classification was reachedexpand

Full frame machine prediction

Teacher imitation

Not calibrated prevalence, not ground truth. Human validation pending. The Gemma side is a direct model label for every work in the frame, read from the title-only record. The Codex side is a classifier learned from the 10,348 direct Codex labels and calibrated to design-weighted sample rates; fields without enough sample support carry no Codex call. Candidate is the union of the two sides; consensus is their intersection. These outputs are machine_predicted_unvalidated and are not human labels.

metaresearch head score (Codex)0.004
metaresearch head score (Gemma)0.007
Version: metacan-v3-hybrid-931329e0061cValidation status: machine_predicted_unvalidated
Candidate categoriesnone
Consensus categoriesnone
DomainCandidate signal: none · Consensus signal: none
Study designCandidate signal: Observational · Consensus signal: Observational
GenreCandidate signal: Empirical · Consensus signal: Empirical
Teacher disagreement score0.008
Threshold uncertainty score0.019

Distilled classifier scores by category (both heads)

CategoryCodexGemma
Metaresearch0.0040.007
Meta-epidemiology (narrow)0.0010.000
Meta-epidemiology (broad)0.0010.001
Bibliometrics0.0010.000
Science and technology studies0.0010.001
Scholarly communication0.0010.000
Open science0.0010.001
Research integrity0.0010.001
Insufficient payload (model declined to judge)0.0050.000

Machine scores (provisional)

The two teacher heads of the student model, read on this work. A score orders the frame for review; it never asserts a category, and the validation status ships verbatim with every row.

Baseline scores from an immature model (maturity gate not passed, 7 training rounds). Scores rank; they never assert a category.

Opus teacher head0.068
GPT teacher head0.340
Teacher spread0.272 · how far apart the two teachers sit on this one work
Validation statusscore_only:v0-immature-baseline · verbatim from the scoring run: score_only means the number may rank works, and no category label ships from it

Classification

machine, unvalidated

Machine predicted; a candidate call from one source (direct Gemma or distilled Codex), not a consensus.

The models applied no category: nothing in the taxonomy fit this work.
Study designObservational
Domainnot available
GenreEmpirical

How this classification was reached, model by model and score by score, is at the end of the page under "How this classification was reached".

Quick stats

Citations0
Published2015
Admission routes1
Has abstractyes

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