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Record W1526387215 · doi:10.1158/1538-7445.am2014-4349

Abstract 4349: Regulation of proliferation and platelet-derived growth factor expression in desmoid tumors by microRNA-29

2014· article· en· W1526387215 on OpenAlexaff
Mushriq Al‐Jazrawe, Raymond Poon, Jessica Liu, Benjamin A. Alman

Bibliographic record

VenueCancer Research · 2014
Typearticle
Languageen
FieldMedicine
TopicSoft tissue tumor case studies
Canadian institutionsHospital for Sick ChildrenUniversity of Toronto
Fundersnot available
KeywordsPlatelet-derived growth factor receptorWnt signaling pathwayGrowth factorCancer researchBiologyCTGFmicroRNAPlatelet-derived growth factorWNT3ASignal transductionMolecular biologyReceptorCell biologyGeneGenetics

Abstract

fetched live from OpenAlex

Abstract Desmoid tumors (DT), also called aggressive fibromatosis, are a rare, locally invasive soft tissue tumors. While DTs harbor mutations that activate β-catenin, a major effector molecule of canonical Wnt signaling, they also display elevated levels of both Platelet-Derived Growth Factor Receptor Beta (PDGFRβ) and its PDGF-B ligand. We hypothesize that active PDGFRβ signaling maintains DT progression. We studied the effect of PDGFRβ inhibition on DT growth. Primary cultures of DTs treated with the PDGFRβ inhibitor Sunitinib show a decrease in proliferation rate as measured by bromodeoxyuridine (BrdU) incorporation assay. In vivo, Sunitinib treatment of Apc1638N/+ mice, a mouse model of DT, also decreased the number and size of tumors. To investigate the relationship of β-catenin activation and PDGFRβ activation, we treated human skin fibroblasts with Wnt3a to activate canonical Wnt signaling, and utilized transgenic murine skin fibroblasts harboring an activated form of β-catenin. In both systems, PDGF-B ligand expression increased both at the mRNA and protein level. However, this increase was not observed immediately, suggesting an indirect mode of regulation. The PDGF-B gene contains a conserved microRNA-29 (miR29) binding site in its 3′ untranslated region. In silico miRacts analysis of five independent DT microarray studies predicted miR29 family deregulation. As such, we studied whether miR29 deregulation in DT leads to PDGF-B overexpression and tumor proliferation. By performing quantitative PCR on human DT samples, we observed that the levels of mature forms of all miR29 family members (MIR29A, B, and C) are significantly down-regulated. Overexpressing miR29 in DT cells decreases PDGF-B ligand levels as well as BrdU incorporation rate. Finally, β-catenin-induced modulation of the PDGF-B ligand is abrogated upon inhibiting miR29. Taken together, these data suggest that PDGFRβ signaling is a potentially important pathway in DT progression. PDGFRβ may be linked to β-catenin by the novel discovery of microRNA-29 down-regulation in DT tumors. This work will offer new perspectives on the molecular biology underlying DT development and help identify novel targets for therapy. Citation Format: Mushriq Al-Jazrawe, Raymond Poon, Jessica Liu, Benjamin Alman. Regulation of proliferation and platelet-derived growth factor expression in desmoid tumors by microRNA-29. [abstract]. In: Proceedings of the 105th Annual Meeting of the American Association for Cancer Research; 2014 Apr 5-9; San Diego, CA. Philadelphia (PA): AACR; Cancer Res 2014;74(19 Suppl):Abstract nr 4349. doi:10.1158/1538-7445.AM2014-4349

Fetched live from OpenAlex and de-inverted. Abstracts are not stored in this database: the inverted indexes are 8.6 GB of the frame’s 9.3 GB of text, and the host has 13 GB free.

How this classification was reachedexpand

Full frame distilled prediction

Teacher imitation

Not calibrated prevalence, not ground truth. Human validation pending. Learned from the 10,348 direct Codex labels and 10,348 direct Gemma labels. Candidate is the union of thresholded teacher heads; consensus is their intersection. These outputs are machine_predicted_unvalidated and are not human labels or direct frontier model labels.

metaresearch head score (Codex)0.000
metaresearch head score (Gemma)0.001
Version: codex-gemma-dda1882f352aValidation status: machine_predicted_unvalidated
Candidate categoriesnone
Consensus categoriesnone
DomainCandidate signal: none · Consensus signal: none
Study designCandidate signal: Bench or experimental · Consensus signal: Bench or experimental
GenreCandidate signal: Empirical · Consensus signal: Empirical
Teacher disagreement score0.331
Threshold uncertainty score0.380

Codex and Gemma teacher scores by category

CategoryCodexGemma
Metaresearch0.0000.001
Meta-epidemiology (narrow)0.0000.000
Meta-epidemiology (broad)0.0000.000
Bibliometrics0.0000.000
Science and technology studies0.0000.000
Scholarly communication0.0000.000
Open science0.0000.000
Research integrity0.0000.000
Insufficient payload (model declined to judge)0.0000.000

Machine scores (provisional)

The two teacher heads of the student model, read on this work. A score orders the frame for review; it never asserts a category, and the validation status ships verbatim with every row.

Baseline scores from an immature model (maturity gate not passed, 7 training rounds). Scores rank; they never assert a category.

Opus teacher head0.052
GPT teacher head0.367
Teacher spread0.315 · how far apart the two teachers sit on this one work
Validation statusscore_only:v0-immature-baseline · verbatim from the scoring run: score_only means the number may rank works, and no category label ships from it

Classification

machine, unvalidated

Machine predicted; a candidate call from one teacher head, not a consensus.

The models applied no category: nothing in the taxonomy fit this work.
Study designBench or experimental
Domainnot available
GenreEmpirical

How this classification was reached, model by model and score by score, is at the end of the page under "How this classification was reached".

Quick stats

Citations0
Published2014
Admission routes1
Has abstractyes

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