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Record W1579041659

Immune Modulation By Targeted Therapies Increases Oncolytic Viral Therapeutic Activity In Lung Cancer

2013· article· en· W1579041659 on OpenAlexaffvenue
Peter Liu

Bibliographic record

VenueJournal of undergraduate research in Alberta · 2013
Typearticle
Languageen
FieldBiochemistry, Genetics and Molecular Biology
TopicVirus-based gene therapy research
Canadian institutionsUniversity of Calgary
Fundersnot available
KeywordsLung cancerOncolytic virusImmune systemCancer researchTumor microenvironmentMedicineCytotoxic T cellImmunologyCancerImmunotherapyOncologyBiologyInternal medicineIn vitro
DOInot available

Abstract

fetched live from OpenAlex

BACKGROUND Lung cancer is the leading cause of cancer mortality worldwide, and non-small cell lung cancer (NSCLC) accounts for 85% of its diagnoses [1]. Despite paradigm shifting targeted (personalized/precision) opportunities for a select group of patients, current treatments have failed to improve the five-year survival rate of only 16.3% [2]. Enhancing the host immune systems’ ability to combat NSCLC has been an unrealized goal for decades. OBJECTIVE Conventional radiotherapy and chemotherapy have a low therapeutic window, causing detrimental effects to both tumor and healthy cells alike. Furthermore, an increasing level of immunosuppressive regulatory cells in the tumor microenvironment and inadequate cytotoxic T-lymphocytes mediated anti-tumor activity characterize tumor progression, making the enhancement of the host immune system’s ability to combat NSCLC crucial [3]. With recent studies indicating sunitnib’s role in inhibiting immunosuppressive cells in the tumor microenvironment and reovirus’ oncolytic, immunostimulating properties in targeting tumor cells, this investigation aims to explore the potential of combining reovirus and sunitnib as a novel therapy for NSCLC [4,5]. METHODS Reovirus and sunitnib effective dose 50 (ED50) were determined for human NSCLC cell lines A549, H460, H1299, and H1975 by exposing each to varying concentrations of reovirus or sunitnib for 48 hours, and quantifying absorbance with WST-1 reagent at 450nm. % viability was used to generate dose response curves, and Calcusyn software was utilized to calculate ED50. Each cell line was then exposed to reovirus, sunitnib, or combination at ED50, ½ ED50, or ¼ ED50 for 48 hours. 20µM was used as ED50 for sunitnib resistant cell lines. Absorbance at 450nm upon WST-1 exposure was read, and combination index (CI) was calculated using Calcusyn. Each experiment contained at least 3 replicates and was repeated 3 times. CI 1 indicates antagonistic effects. RESULTS Reovirus and sunitnib ED50 values for A549, H460, H1299, and H1975 cells were 15.29±0.99MOI and 17.12±2.62µM, 238.02±41.29MOI and 11.27±1.52µM, 6.85±2.39MOI and resistant, and 19.80±20.35µM and not determinable, respectively. CI values for all cell lines were < 1at ED50. DISCUSSION AND CONCLUSIONS In the present in vitro study, we have demonstrated that sunitnib and reovirus combination therapy results in synergy in NSCLC cells. This promising investigation establishes the preclinical foundation for further investigations into reovirus and sunitnib as a novel combination therapy to treat NSCLC. Following ex vivo and in vivo investigations, it is anticipated that this study will culminate into a human clinical trial.

Fetched live from OpenAlex and de-inverted. Abstracts are not stored in this database: the inverted indexes are 8.6 GB of the frame’s 9.3 GB of text, and the host has 13 GB free.

How this classification was reachedexpand

Full frame machine prediction

Teacher imitation

Not calibrated prevalence, not ground truth. Human validation pending. The Gemma side is a direct model label for every work in the frame, read from the title-only record. The Codex side is a classifier learned from the 10,348 direct Codex labels and calibrated to design-weighted sample rates; fields without enough sample support carry no Codex call. Candidate is the union of the two sides; consensus is their intersection. These outputs are machine_predicted_unvalidated and are not human labels.

metaresearch head score (Codex)0.000
metaresearch head score (Gemma)0.000
Version: metacan-v3-hybrid-931329e0061cValidation status: machine_predicted_unvalidated
Candidate categoriesnone
Consensus categoriesnone
DomainCandidate signal: none · Consensus signal: none
Study designCandidate signal: Bench or experimental · Consensus signal: Bench or experimental
GenreCandidate signal: Empirical · Consensus signal: Empirical
Teacher disagreement score0.002
Threshold uncertainty score0.006

Distilled classifier scores by category (both heads)

CategoryCodexGemma
Metaresearch0.0000.000
Meta-epidemiology (narrow)0.0000.000
Meta-epidemiology (broad)0.0000.000
Bibliometrics0.0000.000
Science and technology studies0.0000.000
Scholarly communication0.0000.000
Open science0.0000.000
Research integrity0.0000.001
Insufficient payload (model declined to judge)0.0020.000

Machine scores (provisional)

The two teacher heads of the student model, read on this work. A score orders the frame for review; it never asserts a category, and the validation status ships verbatim with every row.

Baseline scores from an immature model (maturity gate not passed, 7 training rounds). Scores rank; they never assert a category.

Opus teacher head0.029
GPT teacher head0.362
Teacher spread0.333 · how far apart the two teachers sit on this one work
Validation statusscore_only:v0-immature-baseline · verbatim from the scoring run: score_only means the number may rank works, and no category label ships from it

Classification

machine, unvalidated

Machine predicted; a candidate call from one source (direct Gemma or distilled Codex), not a consensus.

The models applied no category: nothing in the taxonomy fit this work.
Study designBench or experimental
Domainnot available
GenreEmpirical

How this classification was reached, model by model and score by score, is at the end of the page under "How this classification was reached".

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Citations0
Published2013
Admission routes2
Has abstractyes

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