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Record W1969134353 · doi:10.1186/1753-6561-5-s1-p102

Role of the viral protein UL24 in nucleolar modifications induced by herpes simplex virus 1

2011· article· en· W1969134353 on OpenAlexaff
Angela Pearson, Amélie Bourget, Nawel Ben Abdeljelil, Maria H. Lymberopoulos

Bibliographic record

VenueBMC Proceedings · 2011
Typearticle
Languageen
FieldMedicine
TopicHerpesvirus Infections and Treatments
Canadian institutionsInstitut National de la Recherche Scientifique
Fundersnot available
KeywordsNucleolinNucleolusBiologyNucleophosminHerpes simplex virusFibrillarinTranscription (linguistics)Molecular biologyADARProtein subunitVirologyCell biologyNucleusRNAVirusGeneticsGene

Abstract

fetched live from OpenAlex

Herpes simplex virus 1 (HSV-1) infection induces multiple modifications to nucleoli. We and others have observed the redistribution of nucleolar proteins such as nucleolin, fibrillarin, B23 (nucleophosmin), upstream binding factor (UBF) and RNA polymerase I (RNAPI) during HSV-1 infection. UL24 is one of a limited number of HSV-1 proteins that localizes to nucleoli. In a murine model of ocular HSV-1 infection, viruses that do not express the UL24 protein exhibit reduced viral titers in the eye and in trigeminal ganglia, and a reduction in clinical signs of disease. UL24 is conserved among Herpesviridae. Specifically, the N-terminal region of the protein contains several stretches of highly conserved residues. Bioinformatics studies have identified an endonuclease motif in the N-terminal portion of UL24, although as yet, no nuclease activity has been demonstrated for the protein. Previously, we discovered that UL24 transiently localizes to nucleoli during infection, and is both necessary and sufficient to induce the dispersal of nucleolin from nucleoli throughout the nucleus. In contrast, the redistribution of fibrillarin is a UL24-independent event. Moreover, the largest subunit of RNAPI and the transcription factor UBF are redistributed to viral replication compartments and these effects are also UL24-independent. We recently investigated whether UL24 was implicated in other HSV-1-induced nucleolar modifications. We discovered that UL24 was involved in the redistribution of B23 during infection. Furthermore, expression of UL24 in the absence of other viral proteins was sufficient to induce the relocalisation of B23. Similar to what we found for nucleolin, the conserved N-terminal portion of UL24 was important for its effect on B23 redistribution. The impact of the mutant vUL24-E99A/K101A, in which the endonuclease motif has been altered, and of vUL24-G121A, which harbours an amino acid substitution outside of this motif, were tested. While the G121A mutation had only a modest effect on the ability the virus to induce the relocalisation of B23, the E99A/K101A mutation appeared to abolish this function. Interestingly, the E99A/K101A mutation was previously shown to have a large impact on viral pathogenesis in mice. We found that the effect of HSV-1 on the redistribution of B23 was similar in non-immortalized cells such as human foreskin fibroblasts, as in immortalized cell lines such as vero cells. The similarities with regard to the impact of UL24 on the spatial distribution of nucleolin and B23 suggest that these effects may be related to the same function of UL24 during HSV-1 infection.

Fetched live from OpenAlex and de-inverted. Abstracts are not stored in this database: the inverted indexes are 8.6 GB of the frame’s 9.3 GB of text, and the host has 13 GB free.

How this classification was reachedexpand

Full frame distilled prediction

Teacher imitation

Not calibrated prevalence, not ground truth. Human validation pending. Learned from the 10,348 direct Codex labels and 10,348 direct Gemma labels. Candidate is the union of thresholded teacher heads; consensus is their intersection. These outputs are machine_predicted_unvalidated and are not human labels or direct frontier model labels.

metaresearch head score (Codex)0.000
metaresearch head score (Gemma)0.000
Version: codex-gemma-dda1882f352aValidation status: machine_predicted_unvalidated
Candidate categoriesnone
Consensus categoriesnone
DomainCandidate signal: none · Consensus signal: none
Study designCandidate signal: Bench or experimental · Consensus signal: Bench or experimental
GenreCandidate signal: Empirical · Consensus signal: Empirical
Teacher disagreement score0.092
Threshold uncertainty score0.348

Codex and Gemma teacher scores by category

CategoryCodexGemma
Metaresearch0.0000.000
Meta-epidemiology (narrow)0.0000.000
Meta-epidemiology (broad)0.0000.000
Bibliometrics0.0000.000
Science and technology studies0.0000.000
Scholarly communication0.0000.000
Open science0.0000.000
Research integrity0.0000.000
Insufficient payload (model declined to judge)0.0000.000

Machine scores (provisional)

The two teacher heads of the student model, read on this work. A score orders the frame for review; it never asserts a category, and the validation status ships verbatim with every row.

Baseline scores from an immature model (maturity gate not passed, 7 training rounds). Scores rank; they never assert a category.

Opus teacher head0.037
GPT teacher head0.261
Teacher spread0.225 · how far apart the two teachers sit on this one work
Validation statusscore_only:v0-immature-baseline · verbatim from the scoring run: score_only means the number may rank works, and no category label ships from it

Classification

machine, unvalidated

Machine predicted; a candidate call from one teacher head, not a consensus.

The models applied no category: nothing in the taxonomy fit this work.
Study designBench or experimental
Domainnot available
GenreEmpirical

How this classification was reached, model by model and score by score, is at the end of the page under "How this classification was reached".

Quick stats

Citations5
Published2011
Admission routes1
Has abstractyes

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