FES kinases are required for oncogenic FLT3 signaling
Why is this work in the frame?
A frame that forgets how it found something cannot be audited. These are the routes that admitted this work.
No Canadian affiliation. An affiliation-only frame — the usual design — would never have seen this work. It is one of the works that make the case for inverting the frame.
The three-model screen
all 1,000 screened works →All three models called this out of scope.
FES kinases in oncogenic FLT3 signaling; cancer cell biology.
The title indicates a cancer-biology study, not research itself, but the abstract is missing.
Biomedical paper on FES kinases in oncogenic FLT3 signaling; title is unambiguous domain biology.
Abstract
No abstract. This is not a gap in this database — OpenAlex has none either. 23.3% of the frame is in this state, and the screen finds HALF as much metaresearch here, so the absence is a measured bias rather than a missing field.
The record
- Venue
- Leukemia
- Topic
- Multiple Myeloma Research and Treatments
- Field
- Medicine
- Canadian institutions
- —
- Funders
- Institut National Du CancerFondation de FranceLigue Contre le CancerInstitute of Cancer ResearchInstitut National de la Santé et de la Recherche Médicale
- Keywords
- KinaseReceptor tyrosine kinaseCancer researchHaematopoiesisBiologyFms-Like Tyrosine Kinase 3Myeloid leukemiaTyrosine kinaseCellCell cycleCell biologySignal transductionMutationStem cellGeneticsGene
- Has abstract in OpenAlex
- no