MétaCan
Menu
← Back to cohort
Record W2000451721 · doi:10.1158/1538-7445.am2011-96

Abstract 96: The functional role of DNA hypomethylation in liver cancer

2011· article· en· W2000451721 on OpenAlexaffabout
Barbara Stefañska, Bishnu Bhattacharyya, Matthew Suderman, Jian Huang, Michael Hallett, Ze‐Guang Han, Moshe Szyf

Bibliographic record

VenueCancer Research · 2011
Typearticle
Languageen
FieldBiochemistry, Genetics and Molecular Biology
TopicEpigenetics and DNA Methylation
Canadian institutionsMcGill University
Fundersnot available
KeywordsDNA methylationChromatin immunoprecipitationBiologyPromoterMethylationLiver cancerCancer researchMethylated DNA immunoprecipitationGeneMolecular biologyCancerMetastasisHepatocellular carcinomaGene expressionGenetics

Abstract

fetched live from OpenAlex

Abstract DNA hypomethylation, a process of losing methyl marks, may play an important role in cancer, especially through activating genes that promote cancer metastasis, the deadly aspect of cancer. We previously delineated using whole genome promoter methylation microarrays the landscape of hypomethylation in hepatocellular carcinoma which is one of the most common cancers worldwide. Our studies revealed that 50% of differentially methylated promoters in tumors are hypomethylated compared to adjacent normal tissue. In the present studies, using liver cancer cell lines as a model system, we evaluated the importance of the identified hypomethylated genes in liver cancer development and progression. We tested whether the hypomethylation observed in liver cancer is driven by MBD2, a protein reported previously to be implicated in DNA demethylation, which we found to be overexpressed in liver cancer patients. Following siRNA MBD2 depletion in HepG2 hepatocellular carcinoma cell line, we used methylated DNA immunoprecipitation, chromatin immunoprecipitation (ChIP) and 60K custom microarrays covering 12K bp upstream and 10K bp downstream of transcription start site to determine methylation and MBD2 binding in 300 top genes hypomethylated and/or induced in liver cancer patients and/or HepG2 cells. The array data were validated by quantitative ChIP and/or pyrosequencing. For functional analyses, we used siRNA transfection, soft agar and invasion assays. We discovered MBD2 binding peaks in 172 genes out of 300, mainly within their promoter regions. Using bioinformatic tools, we identified MBD2 binding motif and established putative transcription factors recognizing this sequence which may play a key role in recruiting MBD2 to target genes. 41% of those genes were hypermethylated in HepG2 cells after MBD2 depletion in a fragment corresponding to MBD2 binding site. In addition, expression analysis for some of the genes showed attenuation. In HepG2 cells, we depleted expression of several of the genes hypomethylated in cancer and regulated by MBD2 using siRNA and demonstrated a decrease in cell growth and invasive capacities. Our results establish a role for MBD2 in coordinating inhibition of a panel of genes involved in cancer growth and metastasis. This study was supported by a grant from the MDEIE program of the government of Quebec and the National Cancer Institute of Canada to MS. Citation Format: {Authors}. {Abstract title} [abstract]. In: Proceedings of the 102nd Annual Meeting of the American Association for Cancer Research; 2011 Apr 2-6; Orlando, FL. Philadelphia (PA): AACR; Cancer Res 2011;71(8 Suppl):Abstract nr 96. doi:10.1158/1538-7445.AM2011-96

Fetched live from OpenAlex and de-inverted. Abstracts are not stored in this database: the inverted indexes are 8.6 GB of the frame’s 9.3 GB of text, and the host has 13 GB free.

How this classification was reachedexpand

Full frame machine prediction

Teacher imitation

Not calibrated prevalence, not ground truth. Human validation pending. The Gemma side is a direct model label for every work in the frame, read from the title-only record. The Codex side is a classifier learned from the 10,348 direct Codex labels and calibrated to design-weighted sample rates; fields without enough sample support carry no Codex call. Candidate is the union of the two sides; consensus is their intersection. These outputs are machine_predicted_unvalidated and are not human labels.

metaresearch head score (Codex)0.000
metaresearch head score (Gemma)0.000
Version: metacan-v3-hybrid-931329e0061cValidation status: machine_predicted_unvalidated
Candidate categoriesnone
Consensus categoriesnone
DomainCandidate signal: none · Consensus signal: none
Study designCandidate signal: Observational · Consensus signal: none
GenreCandidate signal: Empirical · Consensus signal: Empirical
Teacher disagreement score0.005
Threshold uncertainty score0.015

Distilled classifier scores by category (both heads)

CategoryCodexGemma
Metaresearch0.0000.000
Meta-epidemiology (narrow)0.0000.000
Meta-epidemiology (broad)0.0000.000
Bibliometrics0.0000.000
Science and technology studies0.0000.000
Scholarly communication0.0000.000
Open science0.0000.000
Research integrity0.0000.000
Insufficient payload (model declined to judge)0.0050.001

Machine scores (provisional)

The two teacher heads of the student model, read on this work. A score orders the frame for review; it never asserts a category, and the validation status ships verbatim with every row.

Baseline scores from an immature model (maturity gate not passed, 7 training rounds). Scores rank; they never assert a category.

Opus teacher head0.109
GPT teacher head0.364
Teacher spread0.255 · how far apart the two teachers sit on this one work
Validation statusscore_only:v0-immature-baseline · verbatim from the scoring run: score_only means the number may rank works, and no category label ships from it

Classification

machine, unvalidated

Machine predicted; a candidate call from one source (direct Gemma or distilled Codex), not a consensus.

The models applied no category: nothing in the taxonomy fit this work.
Study designObservational
Domainnot available
GenreEmpirical

How this classification was reached, model by model and score by score, is at the end of the page under "How this classification was reached".

Quick stats

Citations0
Published2011
Admission routes2
Has abstractyes

Explore more

Same venueCancer Research→Same topicEpigenetics and DNA Methylation→French-language works237,207→