Dysfunctional HDL containing L159R ApoA-I leads to exacerbation of atherosclerosis in hyperlipidemic mice
Why is this work in the frame?
A frame that forgets how it found something cannot be audited. These are the routes that admitted this work.
No Canadian affiliation. An affiliation-only frame — the usual design — would never have seen this work. It is one of the works that make the case for inverting the frame.
The three-model screen
all 1,000 screened works →All three models called this out of scope.
Lipid biology of dysfunctional HDL in atherosclerosis; abstract absent but the title is unambiguous.
It studies lipid biology and atherosclerosis in hyperlipidemic mice.
Title is clear domain atherosclerosis/lipid research despite missing abstract.
Abstract
No abstract. This is not a gap in this database — OpenAlex has none either. 23.3% of the frame is in this state, and the screen finds HALF as much metaresearch here, so the absence is a measured bias rather than a missing field.
The record
- Venue
- Biochimica et Biophysica Acta (BBA) - Molecular and Cell Biology of Lipids
- Topic
- Cholesterol and Lipid Metabolism
- Field
- Medicine
- Canadian institutions
- —
- Funders
- National Center for Research ResourcesNational Heart, Lung, and Blood InstituteGladstone InstitutesNational Cancer InstituteNational Institutes of HealthWake Forest UniversityUniversity of British ColumbiaNorth Carolina Biotechnology Center
- Keywords
- Dysfunctional familyExacerbationInternal medicineMedicineCardiologyPsychiatry
- Has abstract in OpenAlex
- no