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Decline in skeletal muscle mitochondrial function with aging in humans

2005· article· en· 1,308 citations· W2002973721 on OpenAlex· 10.1073/pnas.0501559102

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A frame that forgets how it found something cannot be audited. These are the routes that admitted this work.

Canadian affiliationAn author listed a Canadian institution. This is the only route the usual frame has.

Machine scores (provisional)

Baseline scores from an immature model (maturity gate not passed, 7 training rounds). Scores rank; they never assert a category.

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Opus teacher head0.021
GPT teacher head0.284
Teacher spread
0.263 · how far apart the two teachers sit on this one work
Validation status
score_only:v0-immature-baseline · verbatim from the scoring run: score_only means the number may rank works, and no category label ships from it

Abstract

Cumulative mtDNA damage occurs in aging animals, and mtDNA mutations are reported to accelerate aging in mice. We determined whether aging results in increased DNA oxidative damage and reduced mtDNA abundance and mitochondrial function in skeletal muscle of human subjects. Studies performed in 146 healthy men and women aged 18-89 yr demonstrated that mtDNA and mRNA abundance and mitochondrial ATP production all declined with advancing age. Abundance of mtDNA was positively related to mitochondrial ATP production rate, which in turn, was closely associated with aerobic capacity and glucose tolerance. The content of several mitochondrial proteins was reduced in older muscles, whereas the level of the oxidative DNA lesion, 8-oxo-deoxyguanosine, was increased, supporting the oxidative damage theory of aging. These results demonstrate that age-related muscle mitochondrial dysfunction is related to reduced mtDNA and muscle functional changes that are common in the elderly.

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The record

Venue
Proceedings of the National Academy of Sciences
Topic
Mitochondrial Function and Pathology
Field
Biochemistry, Genetics and Molecular Biology
Canadian institutions
Institute of Nutrition, Metabolism and Diabetes
Funders
National Center for Research ResourcesNational Institute of Diabetes and Digestive and Kidney DiseasesNational Institute on Aging
Keywords
Mitochondrial DNAMitochondrionSkeletal muscleBiologyOxidative phosphorylationOxidative damage8-Hydroxy-2'-deoxyguanosineOxidative stressInternal medicineEndocrinologyGeneticsDNA oxidationBiochemistryGeneMedicine
Has abstract in OpenAlex
yes