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Record W2046947683 · doi:10.1158/1538-7445.am2011-2880

Abstract 2880: Role of Akt isoforms in chemoresistance of endometrial carcinoma cells

2011· article· en· W2046947683 on OpenAlexaff
Marie-Judith Lafleur, Julie Girouard, Éric Asselin

Bibliographic record

VenueCancer Research · 2011
Typearticle
Languageen
FieldBiochemistry, Genetics and Molecular Biology
TopicKruppel-like factors research
Canadian institutionsUniversité du Québec à Trois-RivièresInnovation and Economic Development Trois Rivières
Fundersnot available
KeywordsProtein kinase BAKT2AKT1Cancer researchCisplatinApoptosisXIAPCell growthWestern blotTransfectionAKT3BiologyChemistryCell cultureMedicineInternal medicineProgrammed cell deathCaspaseChemotherapyBiochemistry

Abstract

fetched live from OpenAlex

Abstract Akt (PKB) is a crucial survival kinase that is normally activated by a variety of growth factors. In tumours, its upstream regulation is affected by oncogenic events, which leads to the constitutive activation of the protein. Since studies have demonstrated that the three Akt isoforms exhibit different physiological functions, our hypothesis is that Akt isoforms may contribute differently in chemoresistance of endometrial carcinoma cells. The aim of this study is to determine the effect of a loss of function of each of Akt isoform in chemoresistant KLE endometrial carcinoma cells. In order to determine the specific role of each Akt isoform in cell survival/resistance to apoptosis and proliferation, we stably transfected KLE cells, which normally express Akt1, Akt2 and Akt3, with specific shRNA for each Akt isoform. We treated Akt isoform-specific transfectants for a period of 48 hour with different chemotherapeutics drugs and we performed western blot analysis for pro-apoptotic and anti-apoptotic factors. In this study, we have demonstrated that Akt1 and Akt2 downregulation sensitize KLE cells to cisplatin, doxorubicin and taxol. After treatment of KLE transfectants with all three drugs, we were able to see that activation pro-apoptotic factors such as cleaved caspase-3, -6 and -9 as well as cleaved PARP were induced. Moreover, the expression of anti-apoptotic factor XIAP was downregulated after drug treatment in these cells. Finally, Bcl-2 expression was inhibited after cisplatin treatment in Akt2 negative cells. We also performed MTT proliferation assay after a 72 hours treatment with chemotherapeutic drugs. Results gained from this study show that each Akt isoform is specifically implicated in cell survival. Akt1 and Akt2 shRNA clones were more sensitive to cisplatin, doxorubicin and taxol treatment. Akt3 shRNA clones were also more sensitive to cisplatin and doxorubicin but were less affected by taxol treatment. Our findings highlight the contribution of Akt isoforms in the molecular mechanisms that confer KLE cells resistance to chemotherapeutics drugs-induced apoptosis. According to our hypothesis, results show the differential implication of each Akt isoform in cell resistance to different chemotherapeutics drugs. We also showed that each isoform have a distinct impact on resistance to apoptosis induced by a given drug. Thus, the results confirm that Akt isoforms could be putative targets for therapy of endometrial cancers. Citation Format: {Authors}. {Abstract title} [abstract]. In: Proceedings of the 102nd Annual Meeting of the American Association for Cancer Research; 2011 Apr 2-6; Orlando, FL. Philadelphia (PA): AACR; Cancer Res 2011;71(8 Suppl):Abstract nr 2880. doi:10.1158/1538-7445.AM2011-2880

Fetched live from OpenAlex and de-inverted. Abstracts are not stored in this database: the inverted indexes are 8.6 GB of the frame’s 9.3 GB of text, and the host has 13 GB free.

How this classification was reachedexpand

Full frame distilled prediction

Teacher imitation

Not calibrated prevalence, not ground truth. Human validation pending. Learned from the 10,348 direct Codex labels and 10,348 direct Gemma labels. Candidate is the union of thresholded teacher heads; consensus is their intersection. These outputs are machine_predicted_unvalidated and are not human labels or direct frontier model labels.

metaresearch head score (Codex)0.001
metaresearch head score (Gemma)0.000
Version: codex-gemma-dda1882f352aValidation status: machine_predicted_unvalidated
Candidate categoriesnone
Consensus categoriesnone
DomainCandidate signal: none · Consensus signal: none
Study designCandidate signal: Bench or experimental · Consensus signal: Bench or experimental
GenreCandidate signal: Empirical · Consensus signal: Empirical
Teacher disagreement score0.007
Threshold uncertainty score0.553

Codex and Gemma teacher scores by category

CategoryCodexGemma
Metaresearch0.0010.000
Meta-epidemiology (narrow)0.0000.000
Meta-epidemiology (broad)0.0000.000
Bibliometrics0.0000.001
Science and technology studies0.0000.000
Scholarly communication0.0000.000
Open science0.0010.000
Research integrity0.0000.000
Insufficient payload (model declined to judge)0.0000.000

Machine scores (provisional)

The two teacher heads of the student model, read on this work. A score orders the frame for review; it never asserts a category, and the validation status ships verbatim with every row.

Baseline scores from an immature model (maturity gate not passed, 7 training rounds). Scores rank; they never assert a category.

Opus teacher head0.067
GPT teacher head0.357
Teacher spread0.290 · how far apart the two teachers sit on this one work
Validation statusscore_only:v0-immature-baseline · verbatim from the scoring run: score_only means the number may rank works, and no category label ships from it

Classification

machine, unvalidated

Machine predicted; a candidate call from one teacher head, not a consensus.

The models applied no category: nothing in the taxonomy fit this work.
Study designBench or experimental
Domainnot available
GenreEmpirical

How this classification was reached, model by model and score by score, is at the end of the page under "How this classification was reached".

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Citations0
Published2011
Admission routes1
Has abstractyes

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