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Record W2123655796 · doi:10.2337/db07-1578

β-Cell Replication by Loosening the Brakes of Glucagon-Like Peptide-1 Receptor Signaling

2008· article· en· W2123655796 on OpenAlexaff
Frans Schuit, Daniel J. Drucker

Bibliographic record

VenueDiabetes · 2008
Typearticle
Languageen
FieldMedicine
TopicPancreatic function and diabetes
Canadian institutionsLunenfeld-Tanenbaum Research InstituteUniversity of TorontoMount Sinai Hospital
Fundersnot available
KeywordsReceptorGlucagonCell biologyReplication (statistics)Signal transductionInternal medicineEndocrinologyBiologyMedicineChemistryVirologyInsulin

Abstract

fetched live from OpenAlex

A decrease in β-cell mass is a well-known key pathogenic event in diabetes, not only in human subjects with type 1 patients, where β-cells are destroyed by the immune system, but also in type 2 diabetes where reduced β-cell function results in hyperglycemia and associated metabolic abnormalities (1,2). These concepts have made the search for the set of rules that control pancreatic β-cell mass an important area of islet research. An interesting emerging topic with clinical relevance is the notion that the actions of glucagon-like peptide-1 (GLP-1) on islet β-cells could be harnessed to improve and preserve β-cell function and potentially reverse defects in β-cell mass (3). GLP-1 is a proglucagon-derived peptide secreted from gut endocrine cells that acts on β-cells at multiple levels, acutely stimulating insulin secretion while chronically promoting proinsulin biosynthesis and growth and survival of β-cells. During meals, GLP-1 is secreted and acts immediately as an incretin, acutely potentiating glucose-dependent insulin release. However, GLP-1 also enhances glucose competence of β-cells and restores glucose sensitivity to diabetic β-cells in vivo. These findings, taken together with the clinical development of GLP-1 receptor (GLP-1R) agonists and dipeptidyl peptidase-4 inhibitors, have focused on attention to the extent to which incretin-based agents may exert long-term beneficial effects on preservation of β-cell function in subjects with type 2 diabetes (4). In an exciting study published in this issue of Diabetes (5), two mechanisms by which GLP-1 causes β-cell replication have been explored. As the authors state, “the overall effect of GLP-1 on increasing β-cell mass in both in vivo and in vitro conditions is relatively small, and augmenting this effect would be beneficial for the treatment or prevention of both type 1 and type 2 diabetes.” The goal of the study by Klinger et al. was to elucidate molecular mechanisms …

Fetched live from OpenAlex and de-inverted. Abstracts are not stored in this database: the inverted indexes are 8.6 GB of the frame’s 9.3 GB of text, and the host has 13 GB free.

How this classification was reachedexpand

Direct model labels (unvalidated)

Per-model category and study-design labels from the labeling rounds. They are machine output, unvalidated, and the disagreement between models ships as data. No study design here is MEDLINE-validated yet.

Model armCategoriesStudy designConfidence
gemmano category
Domain: not available · Genre: Empirical
About the Canadian research system: no · About a Canadian topic: no
Bench or experimentallow
gptno category
Domain: not available · Genre: Empirical
About the Canadian research system: no · About a Canadian topic: no
Bench or experimentalhigh
models agreeAgreement compares identical category sets and study designs across arms.

Full frame machine prediction

Teacher imitation

Not calibrated prevalence, not ground truth. Human validation pending. The Gemma side is a direct model label for every work in the frame, read from the title-only record. The Codex side is a classifier learned from the 10,348 direct Codex labels and calibrated to design-weighted sample rates; fields without enough sample support carry no Codex call. Candidate is the union of the two sides; consensus is their intersection. These outputs are machine_predicted_unvalidated and are not human labels.

metaresearch head score (Codex)0.000
metaresearch head score (Gemma)0.001
Version: metacan-v3-hybrid-931329e0061cValidation status: machine_predicted_unvalidated
Candidate categoriesnone
Consensus categoriesnone
DomainCandidate signal: none · Consensus signal: none
Study designCandidate signal: Bench or experimental · Consensus signal: Bench or experimental
GenreCandidate signal: Empirical · Consensus signal: Empirical
Teacher disagreement score0.005
Threshold uncertainty score0.016

Distilled classifier scores by category (both heads)

CategoryCodexGemma
Metaresearch0.0000.001
Meta-epidemiology (narrow)0.0000.000
Meta-epidemiology (broad)0.0010.001
Bibliometrics0.0010.000
Science and technology studies0.0000.001
Scholarly communication0.0010.001
Open science0.0010.001
Research integrity0.0010.002
Insufficient payload (model declined to judge)0.0050.002

Machine scores (provisional)

The two teacher heads of the student model, read on this work. A score orders the frame for review; it never asserts a category, and the validation status ships verbatim with every row.

Baseline scores from an immature model (maturity gate not passed, 7 training rounds). Scores rank; they never assert a category.

Opus teacher head0.016
GPT teacher head0.231
Teacher spread0.215 · how far apart the two teachers sit on this one work
Validation statusscore_only:v0-immature-baseline · verbatim from the scoring run: score_only means the number may rank works, and no category label ships from it

Classification

machine, unvalidated

Labeled directly by 2 models reading the full record.

The models applied no category: nothing in the taxonomy fit this work.
Study designBench or experimental
Domainnot available
GenreEmpirical

How this classification was reached, model by model and score by score, is at the end of the page under "How this classification was reached".

Quick stats

Citations10
Published2008
Admission routes1
Has abstractyes

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