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mTOR Signaling in Growth Control and Disease

2012· review· en· 8,483 citations· W2127063807 on OpenAlex· 10.1016/j.cell.2012.03.017

Why is this work in the frame?

A frame that forgets how it found something cannot be audited. These are the routes that admitted this work.

Canadian funderA Canadian agency funded it. The work may carry no Canadian affiliation at all.

No Canadian affiliation. An affiliation-only frame — the usual design — would never have seen this work. It is one of the works that make the case for inverting the frame.

Abstract

No abstract. This is not a gap in this database — OpenAlex has none either. 23.3% of the frame is in this state, and the screen finds HALF as much metaresearch here, so the absence is a measured bias rather than a missing field.

The record

Venue
Cell
Topic
PI3K/AKT/mTOR signaling in cancer
Field
Biochemistry, Genetics and Molecular Biology
Canadian institutions
Funders
National Cancer InstituteNational Institute of Allergy and Infectious DiseasesCanadian Institutes of Health ResearchNational Institutes of HealthHoward Hughes Medical Institute
Keywords
PI3K/AKT/mTOR pathwayBiologyNeurodegenerationMechanistic target of rapamycinSignal transductionDiseaseNeuroscienceProtein kinase BCell biologyBioinformaticsMedicineInternal medicine
Has abstract in OpenAlex
no