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Coding-Independent Regulation of the Tumor Suppressor PTEN by Competing Endogenous mRNAs
Why is this work in the frame?
A frame that forgets how it found something cannot be audited. These are the routes that admitted this work.
Canadian funderA Canadian agency funded it. The work may carry no Canadian affiliation at all.
No Canadian affiliation. An affiliation-only frame — the usual design — would never have seen this work. It is one of the works that make the case for inverting the frame.
Abstract
No abstract. This is not a gap in this database — OpenAlex has none either. 23.3% of the frame is in this state, and the screen finds HALF as much metaresearch here, so the absence is a measured bias rather than a missing field.
The record
- Venue
- Cell
- Topic
- Cancer-related molecular mechanisms research
- Field
- Biochemistry, Genetics and Molecular Biology
- Canadian institutions
- —
- Funders
- National Center for Research ResourcesCanadian Institutes of Health ResearchNational Cancer InstituteNational Institutes of HealthAssociazione Italiana per la Ricerca sul CancroThomas Jefferson UniversityHarvard UniversityNational Health and Medical Research CouncilHarvard CatalystFondazione per la Ricerca BiomedicaU.S. Department of Defense
- Keywords
- Competing endogenous RNABiologyPTENmicroRNACarcinogenesisSuppressorCrosstalkRNAGeneEndogenyComputational biologyLong non-coding RNAGeneticsCancer researchPI3K/AKT/mTOR pathwaySignal transduction
- Has abstract in OpenAlex
- no