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Activation of p75NTR by proBDNF facilitates hippocampal long-term depression
Why is this work in the frame?
A frame that forgets how it found something cannot be audited. These are the routes that admitted this work.
Canadian funderA Canadian agency funded it. The work may carry no Canadian affiliation at all.
No Canadian affiliation. An affiliation-only frame — the usual design — would never have seen this work. It is one of the works that make the case for inverting the frame.
Abstract
No abstract. This is not a gap in this database — OpenAlex has none either. 23.3% of the frame is in this state, and the screen finds HALF as much metaresearch here, so the absence is a measured bias rather than a missing field.
The record
- Venue
- Nature Neuroscience
- Topic
- Nerve injury and regeneration
- Field
- Neuroscience
- Canadian institutions
- —
- Funders
- National Institute of Neurological Disorders and StrokeNational Heart, Lung, and Blood InstituteNatural Sciences and Engineering Research Council of CanadaNational Institutes of Health
- Keywords
- Long-term potentiationTropomyosin receptor kinase BNeuroscienceLow-affinity nerve growth factor receptorSynaptic plasticityHippocampal formationNeurotrophinBrain-derived neurotrophic factorNMDA receptorLong-term depressionNeurotrophic factorsMetaplasticitySynaptic fatigueHippocampusNeuroplasticityBiologyReceptorAMPA receptor
- Has abstract in OpenAlex
- no