← all works
Epistasis between mouse Klra and major histocompatibility complex class I loci is associated with a new mechanism of natural killer cell–mediated innate resistance to cytomegalovirus infection
Why is this work in the frame?
A frame that forgets how it found something cannot be audited. These are the routes that admitted this work.
Canadian affiliationAn author listed a Canadian institution. This is the only route the usual frame has.
The three-model screen
all 1,000 screened works →All three models called this out of scope.
stratum: aff_core · design weight: 5595.24 (the sample is stratified; any rate computed without the weight is wrong)
Claude Opus 4.8OUT
genre: empirical
about Canada: no
confidence: high
Immunogenetics of NK-cell resistance to cytomegalovirus in mice.
GPT-5.6 (high)OUT
genre: empirical
about Canada: no
confidence: high
The study concerns immune genetics and cytomegalovirus resistance, not research itself.
Grok 4.5OUT
genre: empirical
about Canada: no
confidence: high
Domain immunology genetics of viral resistance, not metaresearch.
Abstract
No abstract. This is not a gap in this database — OpenAlex has none either. 23.3% of the frame is in this state, and the screen finds HALF as much metaresearch here, so the absence is a measured bias rather than a missing field.
The record
- Venue
- Nature Genetics
- Topic
- Immune Cell Function and Interaction
- Field
- Immunology and Microbiology
- Canadian institutions
- Institut National de la Recherche ScientifiqueMontreal Clinical Research InstituteMcGill University
- Funders
- National Cancer Institute
- Keywords
- BiologyMajor histocompatibility complexInnate immune systemMechanism (biology)ImmunologyNatural killer cellEpistasisCytomegalovirusGeneticsVirologyImmune systemGeneVirusHerpesviridaeCytotoxic T cellViral diseaseIn vitro
- Has abstract in OpenAlex
- no