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Record W2147995007

An Overview of Hashimoto’s Encephalopathy

2009· article· en· W2147995007 on OpenAlexaff
FJ Muzaffar, Talal Hussain Muzaffar

Bibliographic record

VenueIranian Red Crescent Medical Journal · 2009
Typearticle
Languageen
FieldMedicine
TopicAutoimmune Neurological Disorders and Treatments
Canadian institutionsMcMaster University
Fundersnot available
KeywordsMedicineEncephalopathyAutoimmune thyroiditisMyoclonusThyroiditisThyroid functionSubclinical infectionAtaxiaGastroenterologyPathologyVasculitisThyroid peroxidaseInternal medicineThyroidDiseaseAnesthesia
DOInot available

Abstract

fetched live from OpenAlex

: Hashimoto's encephalopathy is a rare complication of autoimmune thyroid disease with an estimated prevalence of 2.1/100,000.1 It is also known as steroid responsive encephalopathy associated with autoimmune thyroiditis, SREAT.2 The pathogenesis is still unknown, but the possible mechanism could be due to cerebral vasculitis with or without immune complex deposition.3 It is an inflammatory condition proposed by the presence of elevated TPO,4 elevated CSF protein,4 histological feature of vasculitis of venules, and lymphocytic perivascular cuff.1 Patients may present with encephalopathy (100%) which usually develop over 1 to 7 days, tremor (84%), transient aphasia (73%), seizure (66%), gait ataxia (63%), hypersomnolence (63%), myoclonus (38%), neuropsychiatric symptoms (36%), and stroke-like symptoms (27%).1,5 Variable thyroid function tests could be seen in patients with Hashimoto’s encephalopathy despite similar neurological findings. Goiter was found in 63% of the reported cases, subclinical hypothyroidism in 35%, normal thyroid function in 30%, overt hypothyroidism in 20%, and hyperthyroidism in 7% of cases.1,6 Lab tests in patients with Hashimoto’s encephalopathy usually show elevated TPO antibodies (100% of reported cases) and  elevated thyroglobulin antibodies in 73% of the reported cases.1,7Upon CSF examination, 78% of the reported cases had elevated protein, and normal leukocyte count in 76% of cases.1,7 The minority of patients, about 15%, had elevated ESR and CRP. Most of the patients (about 98%) with Hashimoto’s encephalopathy had abnormal EEG.7 EEG abnormalities could be generalized slowing, focal slowing, prominent triphasic waves, epileptiform abnormalities, and frontal intermittent rhythmic delta activity.7 50% of the patients with Hashimoto’s encephalopathy had abnormal imaging in CT and MRI, in the form of cerebral atrophy, abnormalities in the white matter and diffused subcortical or focal cortical abnormalities.7,8 Single photon emission computed tomography (SPECT) showed focal hypoperfusion in 73% of cases, global hypoperfusion in 9%, and 18% of cases had normal SPECT. Treatment options include steroids.6 Steroids could be given as oral prednisone (50-100 mg/day), or intravenous methylprednisolone (1 g/day). Thyroid hormone replacement therapy with steroid was administered for patients presenting with hypothyroidism and Hashimoto’s encephalopathy, and there was 92% improvement of reported cases using this combination therapy, and 67% improvement in patients taking levothyroxine alone. There has been only one case having been reported to recover from Hashimoto’s encephalopathy after thyroidectomy.8 Hashimot’s encephalopathy is a rare complication of autoimmune thyroid disease, which is most of the time underdiagnosed. The pathogenesis is still unknown. Steroid is considered the mainstay of treatment.

Fetched live from OpenAlex and de-inverted. Abstracts are not stored in this database: the inverted indexes are 8.6 GB of the frame’s 9.3 GB of text, and the host has 13 GB free.

How this classification was reachedexpand

Full frame distilled prediction

Teacher imitation

Not calibrated prevalence, not ground truth. Human validation pending. Learned from the 10,348 direct Codex labels and 10,348 direct Gemma labels. Candidate is the union of thresholded teacher heads; consensus is their intersection. These outputs are machine_predicted_unvalidated and are not human labels or direct frontier model labels.

metaresearch head score (Codex)0.000
metaresearch head score (Gemma)0.000
Version: codex-gemma-dda1882f352aValidation status: machine_predicted_unvalidated
Candidate categoriesInsufficient payload (model declined to judge)
Consensus categoriesnone
DomainCandidate signal: none · Consensus signal: none
Study designCandidate signal: Observational · Consensus signal: none
GenreCandidate signal: Empirical · Consensus signal: Empirical
Teacher disagreement score0.884
Threshold uncertainty score0.999

Codex and Gemma teacher scores by category

CategoryCodexGemma
Metaresearch0.0000.000
Meta-epidemiology (narrow)0.0000.000
Meta-epidemiology (broad)0.0000.000
Bibliometrics0.0000.000
Science and technology studies0.0000.000
Scholarly communication0.0000.000
Open science0.0000.000
Research integrity0.0000.001
Insufficient payload (model declined to judge)0.0020.000

Machine scores (provisional)

The two teacher heads of the student model, read on this work. A score orders the frame for review; it never asserts a category, and the validation status ships verbatim with every row.

Baseline scores from an immature model (maturity gate not passed, 7 training rounds). Scores rank; they never assert a category.

Opus teacher head0.051
GPT teacher head0.353
Teacher spread0.302 · how far apart the two teachers sit on this one work
Validation statusscore_only:v0-immature-baseline · verbatim from the scoring run: score_only means the number may rank works, and no category label ships from it

Classification

machine, unvalidated

Machine predicted; a candidate call from one teacher head, not a consensus.

Study designObservational
Domainnot available
GenreEmpirical

How this classification was reached, model by model and score by score, is at the end of the page under "How this classification was reached".

Quick stats

Citations0
Published2009
Admission routes1
Has abstractyes

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