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Dynamic Reprogramming of the Kinome in Response to Targeted MEK Inhibition in Triple-Negative Breast Cancer
Why is this work in the frame?
A frame that forgets how it found something cannot be audited. These are the routes that admitted this work.
Canadian funderA Canadian agency funded it. The work may carry no Canadian affiliation at all.
No Canadian affiliation. An affiliation-only frame — the usual design — would never have seen this work. It is one of the works that make the case for inverting the frame.
Abstract
No abstract. This is not a gap in this database — OpenAlex has none either. 23.3% of the frame is in this state, and the screen finds HALF as much metaresearch here, so the absence is a measured bias rather than a missing field.
The record
- Venue
- Cell
- Topic
- Melanoma and MAPK Pathways
- Field
- Biochemistry, Genetics and Molecular Biology
- Canadian institutions
- —
- Funders
- National Institute of General Medical SciencesNational Institute of Diabetes and Digestive and Kidney DiseasesNational Cancer InstituteNational Institute of Environmental Health SciencesCanadian Institutes of Health Research
- Keywords
- KinomeTriple-negative breast cancerMEK inhibitorBiologyCancer researchMAPK/ERK pathwayReprogrammingReceptor tyrosine kinaseKinaseTrametinibGene knockdownCancerCell biologyBreast cancerApoptosisBiochemistryCellGenetics
- Has abstract in OpenAlex
- no