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Lipin-1 and lipin-3 together determine adiposity in vivo

2013· article· en· 63 citations· W2169276971 on OpenAlex· 10.1016/j.molmet.2013.11.008

Why is this work in the frame?

A frame that forgets how it found something cannot be audited. These are the routes that admitted this work.

Canadian affiliationAn author listed a Canadian institution. This is the only route the usual frame has.

The three-model screen

all 1,000 screened works →

All three models called this out of scope.

stratum: aff_core · design weight: 5595.24 (the sample is stratified; any rate computed without the weight is wrong)
Claude Opus 4.8OUT
genre: empirical
about Canada: no
confidence: high

Lipin proteins and adiposity in mice; metabolic biology.

GPT-5.6 (high)OUT
genre: empirical
about Canada: no
confidence: high

This studies lipin proteins and adiposity in mice, not research itself.

Grok 4.5OUT
genre: empirical
about Canada: no
confidence: high

Mouse genetics of lipin enzymes and adiposity; domain metabolism biology.

Abstract

The lipin protein family of phosphatidate phosphatases has an established role in triacylglycerol synthesis and storage. Physiological roles for lipin-1 and lipin-2 have been identified, but the role of lipin-3 has remained mysterious. Using lipin single- and double-knockout models we identified a cooperative relationship between lipin-3 and lipin-1 that influences adipogenesis in vitro and adiposity in vivo. Furthermore, natural genetic variations in Lpin1 and Lpin3 expression levels across 100 mouse strains correlate with adiposity. Analysis of PAP activity in additional metabolic tissues from lipin single- and double-knockout mice also revealed roles for lipin-1 and lipin-3 in spleen, kidney, and liver, for lipin-1 alone in heart and skeletal muscle, and for lipin-1 and lipin-2 in lung and brain. Our findings establish that lipin-1 and lipin-3 cooperate in vivo to determine adipose tissue PAP activity and adiposity, and may have implications in understanding the protection of lipin-1-deficient humans from overt lipodystrophy.

Stored with the screening record, where it is evidence for the labels above.

The record

Venue
Molecular Metabolism
Topic
Lipid metabolism and biosynthesis
Field
Biochemistry, Genetics and Molecular Biology
Canadian institutions
University of Alberta
Funders
National Institute of General Medical SciencesNational Human Genome Research InstituteNational Heart, Lung, and Blood InstituteNational Institutes of Health
Keywords
Adipose tissueIn vivoLipodystrophyAdipogenesisInternal medicineEndocrinologyBiologyCell biologyMedicineImmunologyBiotechnology
Has abstract in OpenAlex
yes