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Record W2181894304

The paternal sperm epigenome: implications for development and disease

2015· article· en· W2181894304 on OpenAlexaff
Sarah Kimmins

Bibliographic record

VenueAnimal Reproduction · 2015
Typearticle
Languageen
FieldBiochemistry, Genetics and Molecular Biology
TopicEpigenetics and DNA Methylation
Canadian institutionsMcGill University
Fundersnot available
KeywordsBiologyEpigeneticsEpigenomeGeneticsHistoneDNA methylationSpermGenomic imprintingOffspringDemethylaseHistone methylationGeneGene expressionPregnancy
DOInot available

Abstract

fetched live from OpenAlex

Despite the father transmitting half the heritable information to the embryo the focus on preconception health has largely been on the mother. New studies highlight the role of the father in disease transmission via non-genetic inheritance, through epigenetic mechanisms. Epigenetic mechanisms include, DNA methylation, post-translational modifications of histones and noncoding RNAs. Paternal effects have been linked to developmental abnormalities and complex diseases such as cancer, diabetes and obesity. Studies in humans and animals have linked epigenetic inheritance to the transmission of environmentally induced phenotypic traits from the father to the developing embryo and these have been associated with altered gene expression and developmental abnormalities in first and second offspring generationsThe genome in mature spermatozoa is packaged in a very unique manner. Whereas the majority of the genome is packaged by sperm-specific nucleoproteins, the protamines, regulatory regions of many genes retain nucleosomes. Moreover, such nucleosomes carry posttranslational modifications in a manner that is highly suggestive of a function in embryo development. In our previous study we show that paternal folate deficiency (FD) is associated with increased birth defects, minor alterations to DNA methylation in the sperm including at genes implicated in development and chronic disease (Lambrot et al ., 2013. Nat Commun ). We also showed folate deficiency altered global levels of histone methylation in sperm, suggesting their involvement in epigenetic inheritance. To address the role of modified histones in sperm for embryonic development we generated transgenic mice over-expressing the human histone demethylase LSD1/KDM1A. Offspring sired by such transgenic mice suffered from major developmental abnormalities and increased neonatal death. Remarkably, overexpression of KDM1A in heterozygote fathers affected wild type offspring as well, even for three following generations. ChIP-Seq studies of sperm of transgenic fathers revealed that H3 lysine 4 dimethylation levels were reduced at promoters of over 2300 genes, many of which serve important functions during embryonic development. In contrast, nucleosome occupancy levels were unaltered at such genes, indicating that KDM1A overexpression affects homeostasis of H3K4me2 during spermatogenesis. Together, our data from environmental exposures and transgenicsstrongly support the idea that disruption of sperm epigenome homeostasis is extremely detrimental for embryonic fitness of offspring. Our data reveals the potential of genetic mutations in chromatin modifiers and of environmental-induced alterations to the sperm epigenome as an underlying cause of birth defects and disease that may be traceable to the father.

Fetched live from OpenAlex and de-inverted. Abstracts are not stored in this database: the inverted indexes are 8.6 GB of the frame’s 9.3 GB of text, and the host has 13 GB free.

How this classification was reachedexpand

Full frame distilled prediction

Teacher imitation

Not calibrated prevalence, not ground truth. Human validation pending. Learned from the 10,348 direct Codex labels and 10,348 direct Gemma labels. Candidate is the union of thresholded teacher heads; consensus is their intersection. These outputs are machine_predicted_unvalidated and are not human labels or direct frontier model labels.

metaresearch head score (Codex)0.000
metaresearch head score (Gemma)0.000
Version: codex-gemma-dda1882f352aValidation status: machine_predicted_unvalidated
Candidate categoriesnone
Consensus categoriesnone
DomainCandidate signal: none · Consensus signal: none
Study designCandidate signal: Bench or experimental · Consensus signal: none
GenreCandidate signal: Empirical · Consensus signal: Empirical
Teacher disagreement score0.577
Threshold uncertainty score0.170

Codex and Gemma teacher scores by category

CategoryCodexGemma
Metaresearch0.0000.000
Meta-epidemiology (narrow)0.0000.000
Meta-epidemiology (broad)0.0000.000
Bibliometrics0.0000.000
Science and technology studies0.0000.000
Scholarly communication0.0000.000
Open science0.0000.000
Research integrity0.0000.000
Insufficient payload (model declined to judge)0.0000.000

Machine scores (provisional)

The two teacher heads of the student model, read on this work. A score orders the frame for review; it never asserts a category, and the validation status ships verbatim with every row.

Baseline scores from an immature model (maturity gate not passed, 7 training rounds). Scores rank; they never assert a category.

Opus teacher head0.050
GPT teacher head0.307
Teacher spread0.257 · how far apart the two teachers sit on this one work
Validation statusscore_only:v0-immature-baseline · verbatim from the scoring run: score_only means the number may rank works, and no category label ships from it

Classification

machine, unvalidated

Machine predicted; a candidate call from one teacher head, not a consensus.

The models applied no category: nothing in the taxonomy fit this work.
Study designBench or experimental
Domainnot available
GenreEmpirical

How this classification was reached, model by model and score by score, is at the end of the page under "How this classification was reached".

Quick stats

Citations0
Published2015
Admission routes1
Has abstractyes

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