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Sorafenib Treatment Following Hematopoietic Stem Cell Transplant In Pediatric FLT3/ITD+ AML

2013· article· en· W2292463206 on OpenAlexaff
Jessica A. Pollard, Bill H. Chang, Todd M. Cooper, Thomas Groß, Sumit Gupta, Phoenix A. Ho, Suzanne McGoldrick, Tanya Watt, India Sisler, Blythe Thomson, Ann E. Woolfrey, Soheil Meshinchi

Bibliographic record

VenueBlood · 2013
Typearticle
Languageen
FieldMedicine
TopicAcute Myeloid Leukemia Research
Canadian institutionsHospital for Sick Children
Fundersnot available
KeywordsMedicineSorafenibTolerabilityInternal medicineHematopoietic stem cell transplantationOncologyRetrospective cohort studyTransplantationContext (archaeology)PediatricsSurgeryAdverse effectHepatocellular carcinoma

Abstract

fetched live from OpenAlex

Abstract Internal tandem duplications of the FLT3 gene (FLT3/ITD) portend poor outcome in both adult and pediatric AML and hematopoietic stem cell transplantation (HSCT) is often performed. Unfortunately, approximately 35% of patients will have disease recurrence despite HSCT and curative options are limited in that context. Sorafenib is an oral multi-target tyrosine kinase with demonstrable efficacy in this AML subtype. Moreover, retrospective studies in adults with FLT3/ITD+ AML suggest tolerability and potential clinical benefit of sorafenib when used in the post HSCT setting for treatment of early relapse or minimal residual disease (MRD) emergence. There is limited published data regarding sorafenib’s tolerability and efficacy following HSCT in pediatric FLT3/ITD+ AML. We report here a retrospective review of our experience. We conducted a retrospective study of pediatric FLT3/ITD+ patients treated with allogeneic HSCT followed by sorafenib in the first 18 months following HSCT. Between March 2008 and March 2012, 13 FLT3/ITD+ pediatric patients at 7 HSCT centers met criteria for study inclusion. Median age at time of treatment was 14 years (range 6-21 years) and 6/14 (46%) were male. 9 patients underwent matched family donor HSCT and 4 had a matched unrelated donor. Sorafenib was initiated early after HSCT (median: 66 days, range 44-170 days) as prophylaxis in 5 patients who were considered to have very high risk features including failure to achieve clinical remission (CR) following first course of chemotherapy (N=2), high allelic ratio FLT3/ITD at diagnosis (N=1) and MRD at time of HSCT (N=2). For the remainder, sorafenib was given after sign of relapse (MRD or morphologic) at a median of 90 days (range 45-480 days) following HSCT. 11/13 patients received single agent therapy; 2 patients with morphologic relapse received additional chemotherapy (N=1) or radiotherapy (N=1). Among the entire group, post HSCT sorafenib therapy was initiated a median of 80 days following HSCT (range 44-480 days) at a median dose of 150 mg/m2/day (range 75-340 mg/m2/day) and continued for a median of 12 months (range 0.5-52 months). 9/13 patients (69%) experienced toxicity which was felt to be medically significant including thrombocytopenia (N=2), rash (N=3), anorexia (N=1), myelosuppression (N=3), infection (N=2) and life threatening cardiac dysfunction (N=1 in patient treated concomitantly with mitoxantrone). In 8/13 cases (61%) the toxicity resulted in reduction or temporary discontinuation of sorafenib yet all patients tolerated retrial of drug at the same or reduced dosing. One additional patient experienced dose limiting marrow hypoplasia at dosing of 340 mg/m2/day. Sorafenib did not appear to exacerbate GVHD. 8/13 (62%) patients had controlled GHVD at time of sorafenib initiation and only 2 patients had subsequent skin flare that was temporally associated with wean of immune suppression and did not preclude use of further sorafenib. Overall, 10/13 (77%) patients remain alive and 7/13 (54%) are disease free. Of 7 patients in continued CR, the median survival is 3.6 years from HSCT (range 1.75-5.6 years); 6/7 are off sorafenib therapy for a median of 10.5 months (range 7-36 months) after receiving treatment for a median of 19 months (range 8-52 months). 6/13 patients (46%) experienced progressive or recurrent disease while on therapy, 3 of whom have died: 2 from rapid disease progression despite sorafenib initiation and 1 from wild-type FLT3 recurrence. 3 additional patients with recurrent disease are receiving salvage therapy, 1 of whom has achieved a 2nd CR. Of particular interest is the outcome of patients who received sorafenib for MRD in the peri transplant period. All 5 remain alive and disease-free a median of 3.6 years from HSCT (range 1.75-5.6 years) and have been off sorafenib for a median of 12 months (range 7-36 months). Conversely, of the 5 patients who started treatment for morphologic recurrence, only 1/5 (20%) remain in CR. Of 3 additional patients who received prophylactic treatment for high AR or poor induction response, 2/3 have recurred, 1 of whom is now in 2ndCR with higher dose sorafenib in combination with conventional chemotherapy. Our study suggests that sorafenib is tolerable and may improve survival for pediatric patients who undergo HSCT for FLT3/ITD+ AML. Prospective study is necessary to further confirm tolerability and to determine the scope of clinical benefit for this high risk population. Disclosures: Off Label Use: Sorafenib use in AML will be discussed.

Fetched live from OpenAlex and de-inverted. Abstracts are not stored in this database: the inverted indexes are 8.6 GB of the frame’s 9.3 GB of text, and the host has 13 GB free.

How this classification was reachedexpand

Full frame machine prediction

Teacher imitation

Not calibrated prevalence, not ground truth. Human validation pending. The Gemma side is a direct model label for every work in the frame, read from the title-only record. The Codex side is a classifier learned from the 10,348 direct Codex labels and calibrated to design-weighted sample rates; fields without enough sample support carry no Codex call. Candidate is the union of the two sides; consensus is their intersection. These outputs are machine_predicted_unvalidated and are not human labels.

metaresearch head score (Codex)0.000
metaresearch head score (Gemma)0.001
Version: metacan-v3-hybrid-931329e0061cValidation status: machine_predicted_unvalidated
Candidate categoriesnone
Consensus categoriesnone
DomainCandidate signal: none · Consensus signal: none
Study designCandidate signal: Observational · Consensus signal: Observational
GenreCandidate signal: Empirical · Consensus signal: Empirical
Teacher disagreement score0.001
Threshold uncertainty score0.002

Distilled classifier scores by category (both heads)

CategoryCodexGemma
Metaresearch0.0000.001
Meta-epidemiology (narrow)0.0000.000
Meta-epidemiology (broad)0.0000.000
Bibliometrics0.0000.000
Science and technology studies0.0000.000
Scholarly communication0.0000.000
Open science0.0000.000
Research integrity0.0000.000
Insufficient payload (model declined to judge)0.0000.000

Machine scores (provisional)

The two teacher heads of the student model, read on this work. A score orders the frame for review; it never asserts a category, and the validation status ships verbatim with every row.

Baseline scores from an immature model (maturity gate not passed, 7 training rounds). Scores rank; they never assert a category.

Opus teacher head0.015
GPT teacher head0.251
Teacher spread0.235 · how far apart the two teachers sit on this one work
Validation statusscore_only:v0-immature-baseline · verbatim from the scoring run: score_only means the number may rank works, and no category label ships from it

Classification

machine, unvalidated

Machine predicted; a candidate call from one source (direct Gemma or distilled Codex), not a consensus.

The models applied no category: nothing in the taxonomy fit this work.
Study designObservational
Domainnot available
GenreEmpirical

How this classification was reached, model by model and score by score, is at the end of the page under "How this classification was reached".

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Citations5
Published2013
Admission routes1
Has abstractyes

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