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Record W2315416109 · doi:10.1158/1538-7445.am10-994

Abstract 994: Role of Akt isoforms in regulating intermediate filaments expression in cancer progression

2010· article· en· W2315416109 on OpenAlexaff
Anne-Marie Fortier, Monique Cadrin, Éric Asselin

Bibliographic record

VenueCancer Research · 2010
Typearticle
Languageen
FieldBiochemistry, Genetics and Molecular Biology
TopicPI3K/AKT/mTOR signaling in cancer
Canadian institutionsUniversité du Québec à Trois-RivièresInnovation and Economic Development Trois Rivières
Fundersnot available
KeywordsProtein kinase BVimentinPI3K/AKT/mTOR pathwayCell biologyBiologyEpithelial–mesenchymal transitionKeratinIntermediate filamentCancer cellGene isoformCancer researchKeratin 8MetastasisSignal transductionCancerCytoskeletonCellImmunologyBiochemistryGeneGenetics

Abstract

fetched live from OpenAlex

Abstract Keratins are epithelial-specific intermediate filament (IF) proteins, which are expressed in a tissue-specific manner. As part of the cytoskeleton, keratins are important for the mechanical stability and integrity of epithelial cells and tissues. Moreover, a number of keratins are involved in intracellular signalling pathways which regulate the response to injuries and nonmechanical stresses, cell growth, cell death and cancer progression. Expression of keratins is maintained during malignant transformation of simple epithelial cells. However, vimentin, a mesenchymal-specific IF, is often co-expressed with keratins in late-stage cancer cells presenting a dedifferentiated phenotype. This is associated with an increased metastatic potential. Metastasis, the foremost cause of mortality in cancer patients, is a coordinated biological process involving multiple intracellular signalling pathways. PI3K/Akt pathway has been shown to play a role in invasion/metastasis. Moreover, the three Akt isoforms would play differential roles in this process. In the present study, we investigate the role of Akt isoforms in regulating the expression of keratins and vimentin in different epithelial carcinoma cell lines and the implication of IF in Akt-regulated cell invasion. Our results show that overexpression of constitutively active Akt isoforms up-regulates IF protein and mRNA levels, by increasing IF mRNA stability. On the contrary, down-regulation of Akt isoforms by shRNA decreases IF protein levels. To examine the regulation of IF expression under a more physiological condition, we treated cells with TGF-b1, which is known to induce Akt activation. We demonstrated that IF are up-regulated by TGF-b1 in PI3K-dependent manner. To our knowledge, these results represent the first indication that Akt activity is regulating IF expression. Overexpression of Akt isoforms also increases in vitro cell invasion. Interestingly, we showed that down-regulation of vimentin by siRNA decreases Akt2-induced cell invasion. These results support the hypothesis that modulation of IF expression plays an active role in cancer progression. Citation Format: {Authors}. {Abstract title} [abstract]. In: Proceedings of the 101st Annual Meeting of the American Association for Cancer Research; 2010 Apr 17-21; Washington, DC. Philadelphia (PA): AACR; Cancer Res 2010;70(8 Suppl):Abstract nr 994.

Fetched live from OpenAlex and de-inverted. Abstracts are not stored in this database: the inverted indexes are 8.6 GB of the frame’s 9.3 GB of text, and the host has 13 GB free.

How this classification was reachedexpand

Full frame machine prediction

Teacher imitation

Not calibrated prevalence, not ground truth. Human validation pending. The Gemma side is a direct model label for every work in the frame, read from the title-only record. The Codex side is a classifier learned from the 10,348 direct Codex labels and calibrated to design-weighted sample rates; fields without enough sample support carry no Codex call. Candidate is the union of the two sides; consensus is their intersection. These outputs are machine_predicted_unvalidated and are not human labels.

metaresearch head score (Codex)0.000
metaresearch head score (Gemma)0.000
Version: metacan-v3-hybrid-931329e0061cValidation status: machine_predicted_unvalidated
Candidate categoriesnone
Consensus categoriesnone
DomainCandidate signal: none · Consensus signal: none
Study designCandidate signal: Observational · Consensus signal: none
GenreCandidate signal: Empirical · Consensus signal: Empirical
Teacher disagreement score0.002
Threshold uncertainty score0.007

Distilled classifier scores by category (both heads)

CategoryCodexGemma
Metaresearch0.0000.000
Meta-epidemiology (narrow)0.0000.000
Meta-epidemiology (broad)0.0000.000
Bibliometrics0.0000.000
Science and technology studies0.0000.000
Scholarly communication0.0000.000
Open science0.0000.000
Research integrity0.0000.000
Insufficient payload (model declined to judge)0.0020.000

Machine scores (provisional)

The two teacher heads of the student model, read on this work. A score orders the frame for review; it never asserts a category, and the validation status ships verbatim with every row.

Baseline scores from an immature model (maturity gate not passed, 7 training rounds). Scores rank; they never assert a category.

Opus teacher head0.035
GPT teacher head0.422
Teacher spread0.387 · how far apart the two teachers sit on this one work
Validation statusscore_only:v0-immature-baseline · verbatim from the scoring run: score_only means the number may rank works, and no category label ships from it

Classification

machine, unvalidated

Machine predicted; a candidate call from one source (direct Gemma or distilled Codex), not a consensus.

The models applied no category: nothing in the taxonomy fit this work.
Study designObservational
Domainnot available
GenreEmpirical

How this classification was reached, model by model and score by score, is at the end of the page under "How this classification was reached".

Quick stats

Citations0
Published2010
Admission routes1
Has abstractyes

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