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125 RATE OF TELOMERE SHORTENING AND CARDIOVASCULAR PHENOTYPES

2013· article· en· W2335450375 on OpenAlexaff
Stefano Masi, Francesco D’Aiuto, Kathleen Kahn, Wkhs Wong, W J Woodside, Gargi Ghosh, W P Whincup, K D Kuh, Hugh Hughes, Thomas von Zglinicki, H R Hardy, D J Deanfield

Bibliographic record

VenueHeart · 2013
Typearticle
Languageen
FieldEnvironmental Science
TopicHealth, Environment, Cognitive Aging
Canadian institutionsInstitute of Aging
Fundersnot available
KeywordsMedicineCardiologyInternal medicineTelomereHeart failure with preserved ejection fractionCohortEjection fractionHeart failure

Abstract

fetched live from OpenAlex

Introduction Cross-sectional adult studies report associations between short leukocyte telomere length (LTL) and measures of vascular and cardiac damage. However, due to high inter-individual differences in LTL at birth and thereafter, it is unclear whether this relationship is due to inherited short telomere length or a higher rate of LTL shortening through life. In this study we explored whether the baseline LTL or the rate of LTL shortening could predict cardiovascular (CV) phenotypes during 10 year follow up in the MRC National Survey of Health and Development (NSHD, also known as 1946 birth cohort) study. Methods 689 participants with measures of LTL and CV risk factors at 52 and 62 years underwent measurements of right common carotid intima-media thickness (cIMT), cardiac mass and left ventricular ejection fraction at 62 years. LTL was measured by real time PCR. Results At both time points a negative association was found between LTL and age (p<0.01), with longer LTL detected in females than males (p<0.01). The rate of LTL shortening was faster in subjects with longer LTL at baseline (p<0.001), in males (p<0.01 vs females) and in those with carotid plaques (p<0.05). The measure of LTL at 52 years did not predict CV phenotypes at 62 years. However, a faster rate of LTL shortening between 52 to 62 years was associated with increased values of cIMT at 62 years (r=0.01; p<0.01) leading to a cross-sectional negative association between LTL and cIMT observed at 62 years (r=−0.075; p<0.05). At both time points, no associations were detected between levels of CV risk factors and cross-sectional and longitudinal measures of LTL. Cardiac measurements were not associated with 52 and 62 year LTL measures, nor with the rate of LTL shortening. Similar trends were observed in male and females and adjustments for CV risk factors, treatments and history of CV disease did not materially affect the results (table 1). Conclusions/implications This is the first study showing an association between rate of LTL attrition and final vascular phenotype. These findings suggest that the rate of progression of cellular aging in late midlife (reflected by the rate of LTL attrition) influences predisposition to vascular damage, over and above the contribution of CV risk factors. Therefore, current results identified a possible novel pathway associated to an increased risk of CV disease. This can possibly improve the CV risk stratification of the patients. Furthermore, a better understanding of the factors influencing the rate of LTL has the potential to identify novel terapeutic targets and lead to a more effective CV disease prevention.

Fetched live from OpenAlex and de-inverted. Abstracts are not stored in this database: the inverted indexes are 8.6 GB of the frame’s 9.3 GB of text, and the host has 13 GB free.

How this classification was reachedexpand

Full frame distilled prediction

Teacher imitation

Not calibrated prevalence, not ground truth. Human validation pending. Learned from the 10,348 direct Codex labels and 10,348 direct Gemma labels. Candidate is the union of thresholded teacher heads; consensus is their intersection. These outputs are machine_predicted_unvalidated and are not human labels or direct frontier model labels.

metaresearch head score (Codex)0.000
metaresearch head score (Gemma)0.000
Version: codex-gemma-dda1882f352aValidation status: machine_predicted_unvalidated
Candidate categoriesInsufficient payload (model declined to judge)
Consensus categoriesInsufficient payload (model declined to judge)
DomainCandidate signal: none · Consensus signal: none
Study designCandidate signal: Observational · Consensus signal: Observational
GenreCandidate signal: Empirical · Consensus signal: Empirical
Teacher disagreement score0.223
Threshold uncertainty score1.000

Codex and Gemma teacher scores by category

CategoryCodexGemma
Metaresearch0.0000.000
Meta-epidemiology (narrow)0.0000.000
Meta-epidemiology (broad)0.0000.000
Bibliometrics0.0000.000
Science and technology studies0.0000.000
Scholarly communication0.0000.000
Open science0.0000.000
Research integrity0.0000.000
Insufficient payload (model declined to judge)0.0020.001

Machine scores (provisional)

The two teacher heads of the student model, read on this work. A score orders the frame for review; it never asserts a category, and the validation status ships verbatim with every row.

Baseline scores from an immature model (maturity gate not passed, 7 training rounds). Scores rank; they never assert a category.

Opus teacher head0.010
GPT teacher head0.210
Teacher spread0.200 · how far apart the two teachers sit on this one work
Validation statusscore_only:v0-immature-baseline · verbatim from the scoring run: score_only means the number may rank works, and no category label ships from it

Classification

machine, unvalidated

Machine predicted; both teacher heads agree on what is shown here.

Study designObservational
Domainnot available
GenreEmpirical

How this classification was reached, model by model and score by score, is at the end of the page under "How this classification was reached".

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Citations0
Published2013
Admission routes1
Has abstractyes

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