MétaCan
Menu
← Back to cohort
Record W2340302582

Molecular Pathogenesis of T Lymphoblastic Lymphomas in an Atm-deficient Mouse Model

2013· dissertation· en· W2340302582 on OpenAlexfundno aff
Peggy Wong

Bibliographic record

VenueTSpace (University of Toronto) · 2013
Typedissertation
Languageen
FieldBiochemistry, Genetics and Molecular Biology
TopicUbiquitin and proteasome pathways
Canadian institutionsnot available
FundersHospital for Sick ChildrenTerry Fox FoundationGenome Canada
KeywordsPathogenesisLymphoblastic LeukemiaCancer researchBiologyMedicineComputational biologyGeneticsImmunologyLeukemia
DOInot available

Abstract

fetched live from OpenAlex

T lymphoblastic lymphoma/leukemia (T-LL) represent malignant disease of T-cell progenitors. Dysregulation of Notch1 and IL-7R pathways promotes T-LL, however the timing of these events remains unclear. Furthermore, the spectrum of Notch1 mutations has not been fully characterized in T-LL models. Chapter 2 demonstrated 3 T-LLs classified based on activated Notch1 (ICN1) expression that arose spontaneously in mice lacking ataxia telangiectasia mutated (ATM) protein. One group expressed truncated forms of ICN1 (T-ICN1) resulting from frame-shifting PEST mutations. T-LL cells but not normal thymocytes expressed a proteoglycan, syndecan 1 (SDC1). SDC1+ blasts were detected in thymi with normal cellularity from clinically healthy mice, thus representing the earliest discernible stage of T-LL progression. T-ICN1 and IL-7Rα were both aberrantly expressed at this early T-LL stage. Therefore, these data suggest that Notch1 mutations, aberrant expression of IL-7Rα and SDC1 arise early during T-LL progression. Chapter 3 demonstrated a paucity of HD mutations, yet T-LL cells proliferated in a Notch ligand-independent fashion. Analysis of ICN1+ T-LLs revealed truncated Notch1 mRNAs lacking ligand-binding domain and part of negative regulatory region. All truncated transcripts contained the intramembranous methionine that has previously been shown to serve as the translational start site. Collectively, our data identify truncated Notch1 mRNAs in T-LLs that likely encode for polypeptides undergoing ligand-independent activation. The third group lacked ICN1 expression (UD-ICN1). Chapter 4 revealed that UD-ICN1 T-LLs expressed low levels of Notch targets and proliferated independently of Notch signal. Gene set enrichment analyses of genes differentially expressed between UD-ICN1 and T-ICN1 T-LLs revealed higher levels of transcription factor krüppel-like factor 9 (Klf9) and other transcriptional regulators associated with the highly proliferative pre-DP stage of normal T-cell development. siRNA knockdown experiments demonstrated that Klf9 promoted proliferation of UD-ICN1 T-LL cells. Although further studies are warranted, the findings in this thesis reveal oncogenic activation of Notch1 as an early event in Atm-/- T-LL progression, and truncated Notch1 transcripts likely represent a novel mode of ligand-independent Notch1 activation. On the other hand, Klf9 promotes proliferation of Notch-independent T-LLs and may represent a potential therapeutic target for this T-LL group.

Fetched live from OpenAlex and de-inverted. Abstracts are not stored in this database: the inverted indexes are 8.6 GB of the frame’s 9.3 GB of text, and the host has 13 GB free.

How this classification was reachedexpand

Full frame machine prediction

Teacher imitation

Not calibrated prevalence, not ground truth. Human validation pending. The Gemma side is a direct model label for every work in the frame, read from the title-only record. The Codex side is a classifier learned from the 10,348 direct Codex labels and calibrated to design-weighted sample rates; fields without enough sample support carry no Codex call. Candidate is the union of the two sides; consensus is their intersection. These outputs are machine_predicted_unvalidated and are not human labels.

metaresearch head score (Codex)0.000
metaresearch head score (Gemma)0.000
Version: metacan-v3-hybrid-931329e0061cValidation status: machine_predicted_unvalidated
Candidate categoriesnone
Consensus categoriesnone
DomainCandidate signal: none · Consensus signal: none
Study designCandidate signal: Bench or experimental · Consensus signal: Bench or experimental
GenreCandidate signal: Empirical · Consensus signal: Empirical
Teacher disagreement score0.003
Threshold uncertainty score0.008

Distilled classifier scores by category (both heads)

CategoryCodexGemma
Metaresearch0.0000.000
Meta-epidemiology (narrow)0.0010.000
Meta-epidemiology (broad)0.0000.000
Bibliometrics0.0010.000
Science and technology studies0.0000.000
Scholarly communication0.0000.000
Open science0.0000.000
Research integrity0.0010.001
Insufficient payload (model declined to judge)0.0030.001

Machine scores (provisional)

The two teacher heads of the student model, read on this work. A score orders the frame for review; it never asserts a category, and the validation status ships verbatim with every row.

Baseline scores from an immature model (maturity gate not passed, 7 training rounds). Scores rank; they never assert a category.

Opus teacher head0.010
GPT teacher head0.228
Teacher spread0.218 · how far apart the two teachers sit on this one work
Validation statusscore_only:v0-immature-baseline · verbatim from the scoring run: score_only means the number may rank works, and no category label ships from it

Classification

machine, unvalidated

Machine predicted; a candidate call from one source (direct Gemma or distilled Codex), not a consensus.

The models applied no category: nothing in the taxonomy fit this work.
Study designBench or experimental
Domainnot available
GenreEmpirical

How this classification was reached, model by model and score by score, is at the end of the page under "How this classification was reached".

Quick stats

Citations0
Published2013
Admission routes1
Has abstractyes

Explore more

Same venueTSpace (University of Toronto)→Same topicUbiquitin and proteasome pathways→French-language works237,207→