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Record W2411424922 · doi:10.1016/s0895-7061(00)01196-1

Vascular injury, vascular healing, hypertension and the renin system Wednesday, may 17, broadway ballroom north, 12:30 pm to 2:30 pm. targeting blood pressure goals and end-organ protection: advances in ras blockade

2000· article· en· W2411424922 on OpenAlexaff
Ernesto L. Schiffrin

Bibliographic record

VenueAmerican Journal of Hypertension · 2000
Typearticle
Languageen
FieldMedicine
TopicHormonal Regulation and Hypertension
Canadian institutionsMontreal Clinical Research Institute
Fundersnot available
KeywordsMedicineAngiotensin II receptor type 1Vascular smooth muscleBlood pressureAngiotensin IIEnd organ damageInternal medicinePathophysiology of hypertensionRenin–angiotensin systemEndotheliumAtenololEndocrinologyAngiotensin receptorAngiotensin-converting enzymeCardiology

Abstract

fetched live from OpenAlex

Alterations in structure and function of blood vessels that occur in hypertensive patients and in experimental hypertensive models may contribute to blood pressure elevation and to the complications of hypertension. Angiotensin II may influence the vasculature in hypertension through its effects on the atherogenic process and on hypertensive vascular disease per se. The actions of angiotensin II on inflammation, smooth muscle cell growth (hypertrophy or hyperplasia) and collagen deposition in blood vessels participate in mechanisms of atherogenesis and hypertensive vascular damage, and are mediated via AT1 receptors, whereas AT2 receptors appear to inhibit growth and stimulate programmed cell death or apoptosis. There is increasing evidence that angiotensin II plays a pivotal role in remodeling of both large and small arteries in experimental and human hypertension. Interruption of the renin-angiotensin system thus appears an interesting approach for the treatment of hypertension in order to correct vascular abnormalities that may contribute to cardiovascular events in hypertension. Treatment of spontaneously hypertensive rats (SHR) with angiotensin converting enzyme (ACE) inhibitors and AT1 angiotensin receptor antagonists results in regression of the altered structure of blood vessels in different vascular beds. Endothelium-dependent relaxation is also improved. Studies in hypertensive patients have now shown that treatment with some ACE inhibitors and more recently AT1 antagonists may induce similar potentially beneficial effects, particularly at the level of small arteries: both structure and endothelium-dependent relaxation are significantly improved. In contrast, treatment with the beta blocker atenolol does not result in any improvement in vascular structure or endothelial function in hypertensive patients with equally well-controlled blood pressure in different studies. It still remains to be determined whether the apparently beneficial vascular effects of blockade of the renin-angiotensin system with ACEI or with AT1 receptor antagonists and their vascular protective properties, beyond blood pressure lowering itself, will result in reduced morbidity and mortality and improved outcomes in hypertensive patients.

Fetched live from OpenAlex and de-inverted. Abstracts are not stored in this database: the inverted indexes are 8.6 GB of the frame’s 9.3 GB of text, and the host has 13 GB free.

How this classification was reachedexpand

Full frame machine prediction

Teacher imitation

Not calibrated prevalence, not ground truth. Human validation pending. The Gemma side is a direct model label for every work in the frame, read from the title-only record. The Codex side is a classifier learned from the 10,348 direct Codex labels and calibrated to design-weighted sample rates; fields without enough sample support carry no Codex call. Candidate is the union of the two sides; consensus is their intersection. These outputs are machine_predicted_unvalidated and are not human labels.

metaresearch head score (Codex)0.000
metaresearch head score (Gemma)0.000
Version: metacan-v3-hybrid-931329e0061cValidation status: machine_predicted_unvalidated
Candidate categoriesInsufficient payload (model declined to judge)
Consensus categoriesnone
DomainCandidate signal: none · Consensus signal: none
Study designCandidate signal: Not applicable · Consensus signal: Not applicable
GenreCandidate signal: Other · Consensus signal: Other
Teacher disagreement score0.359
Threshold uncertainty score0.914

Distilled classifier scores by category (both heads)

CategoryCodexGemma
Metaresearch0.0000.000
Meta-epidemiology (narrow)0.0010.000
Meta-epidemiology (broad)0.0010.001
Bibliometrics0.0010.000
Science and technology studies0.0020.001
Scholarly communication0.0030.001
Open science0.0010.001
Research integrity0.0020.002
Insufficient payload (model declined to judge)0.3590.089

Machine scores (provisional)

The two teacher heads of the student model, read on this work. A score orders the frame for review; it never asserts a category, and the validation status ships verbatim with every row.

Baseline scores from an immature model (maturity gate not passed, 7 training rounds). Scores rank; they never assert a category.

Opus teacher head0.010
GPT teacher head0.225
Teacher spread0.215 · how far apart the two teachers sit on this one work
Validation statusscore_only:v0-immature-baseline · verbatim from the scoring run: score_only means the number may rank works, and no category label ships from it

Classification

machine, unvalidated

Machine predicted; a candidate call from one source (direct Gemma or distilled Codex), not a consensus.

Study designNot applicable
Domainnot available
GenreOther

How this classification was reached, model by model and score by score, is at the end of the page under "How this classification was reached".

Quick stats

Citations0
Published2000
Admission routes1
Has abstractyes

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