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MG-136 Functional analysis of the autism and intellectual disability gene PTCHD1 reveals hedgehog receptor-like functions and pdz-binding domain-specific regulation of CNTNAP1 and NLGN1

2015· article· en· W2411643890 on OpenAlexaff
Kirti Mittal, Bryan Degagne, Taimoor I. Sheikh, John B. Vincent

Bibliographic record

Venuenot available
Typearticle
Languageen
FieldBiochemistry, Genetics and Molecular Biology
TopicHedgehog Signaling Pathway Studies
Canadian institutionsCentre for Addiction and Mental Health
Fundersnot available
KeywordsSmoothenedBiologyGeneticsSonic hedgehogHedgehogGeneHedgehog signaling pathwayPTCH1

Abstract

fetched live from OpenAlex

Background Genomic copy number variants (CNVs) have identified genes associated with autism spectrum disorder (ASD) and intellectual disability (ID). Recently, we reported on 23 individuals in 17 families with loss CNVs or frame-shifting indels disrupting PTCHD1. However, little is known for the function of PTCHD1. Objectives PTCHD1, an X-linked gene for ASD and/or (ID), encodes a protein with homology to the sonic hedgehog (Shh) receptors PTCH1 and 2, and has been reported to show similar Gli repression activity. However, little else is known about the function of PTCHD1 or the pathway (s) that are perturbed by PTCHD1 mutations leading to ASD/ID. Design/method We looked at the effect of over-expression of PTCHD1 on transcription levels of neuronal and synapse-related genes. We also included a truncated construct lacking the C-terminal four amino acids, Ile-Thr-Thr-Val (ITTV) of PTCHD1, which is predicted to interact with the PDZ domains of proteins. We tested the transcription of Shh and its putative receptor, Ptchd1, and Smoothened (Smo), from mouse embryonic and postnatal brains. We also analysed the expression of PTCHD1 and Smo in primary cilia. Results Transcriptional analysis showed Shh, Ptch1, and Smo expressed in developing mouse embryonic and postnatal hippocampus, but with gradually decreasing levels, whereas Ptchd1 levels steadily increase, suggesting that Ptchd1 may be more relevant in later embryonic and postnatal neurodevelopment. Preliminary results indicate that PTCHD1 is able to localise to the cilia. A significant increase in level of transcription of NLGN1 and CNTNAP1 that is dependent on the C-terminal four amino acids of PTCHD1 suggests either a regulatory or a downstream effect of PTCHD1 on these genes via a PDZ-domain containing protein. Positive PTCHD1 immunolabeling was visible in the hippocampal neurons. Preliminary results also suggest localization of PTCHD1 in cilia. Conclusions We report here a potential role for the PDZ binding domain in PTCHD1 in regulating expression of Nlgn1 and Cntnap1. PDZ domains are found in many eukaryotic and bacterial proteins, and in particular many proteins that are important for synaptic functioning. We speculate that PTCHD1 localization to primary cilia in hippocampal neurons could inhibit the Hh pathway by excluding SMO, allowing cilia to function as Shh sensors, in a manner similar to that of PTCH1, during neurodevelopment. The PTCHD1 gene has a role in a neurobiological pathway that delivers information to cells during brain development and CNVs and mutations may disrupt crucial developmental processes, contributing to the onset of autism.

Fetched live from OpenAlex and de-inverted. Abstracts are not stored in this database: the inverted indexes are 8.6 GB of the frame’s 9.3 GB of text, and the host has 13 GB free.

How this classification was reachedexpand

Full frame machine prediction

Teacher imitation

Not calibrated prevalence, not ground truth. Human validation pending. The Gemma side is a direct model label for every work in the frame, read from the title-only record. The Codex side is a classifier learned from the 10,348 direct Codex labels and calibrated to design-weighted sample rates; fields without enough sample support carry no Codex call. Candidate is the union of the two sides; consensus is their intersection. These outputs are machine_predicted_unvalidated and are not human labels.

metaresearch head score (Codex)0.000
metaresearch head score (Gemma)0.000
Version: metacan-v3-hybrid-931329e0061cValidation status: machine_predicted_unvalidated
Candidate categoriesnone
Consensus categoriesnone
DomainCandidate signal: none · Consensus signal: none
Study designCandidate signal: Bench or experimental · Consensus signal: Bench or experimental
GenreCandidate signal: Empirical · Consensus signal: Empirical
Teacher disagreement score0.003
Threshold uncertainty score0.007

Distilled classifier scores by category (both heads)

CategoryCodexGemma
Metaresearch0.0000.000
Meta-epidemiology (narrow)0.0000.000
Meta-epidemiology (broad)0.0000.000
Bibliometrics0.0000.000
Science and technology studies0.0000.000
Scholarly communication0.0000.000
Open science0.0000.000
Research integrity0.0000.000
Insufficient payload (model declined to judge)0.0010.000

Machine scores (provisional)

The two teacher heads of the student model, read on this work. A score orders the frame for review; it never asserts a category, and the validation status ships verbatim with every row.

Baseline scores from an immature model (maturity gate not passed, 7 training rounds). Scores rank; they never assert a category.

Opus teacher head0.034
GPT teacher head0.240
Teacher spread0.205 · how far apart the two teachers sit on this one work
Validation statusscore_only:v0-immature-baseline · verbatim from the scoring run: score_only means the number may rank works, and no category label ships from it

Classification

machine, unvalidated

Machine predicted; a candidate call from one source (direct Gemma or distilled Codex), not a consensus.

The models applied no category: nothing in the taxonomy fit this work.
Study designBench or experimental
Domainnot available
GenreEmpirical

How this classification was reached, model by model and score by score, is at the end of the page under "How this classification was reached".

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Citations0
Published2015
Admission routes1
Has abstractyes

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