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Record W2415694895 · doi:10.1093/neuonc/nov228.29

PTPS-29UBIQUITIN CARBOXYL TERMINAL HYDROLASE-L1 (UCH-L1) IS A TUMOR SUPPRESSOR IN ATYPICAL TERATOID RHABDOID TUMORS (ATRTs)

2015· article· en· W2415694895 on OpenAlexaff
Lin Li, James Xu, Jim Davie, Sachin Katyal, Marc Symons, Magimairajan Vanan

Bibliographic record

VenueNeuro-Oncology · 2015
Typearticle
Languageen
FieldBiochemistry, Genetics and Molecular Biology
TopicChromatin Remodeling and Cancer
Canadian institutionsChildren's Hospital of WinnipegChildren's Hospital Research Institute of ManitobaResearch Institute in Oncology and Hematology
Fundersnot available
KeywordsSuppressorTerminal (telecommunication)Cancer researchHydrolaseChemistryBiologyBiochemistryEnzymeComputer scienceGene

Abstract

fetched live from OpenAlex

BACKGROUND: Atypical teratoid rhabdoid tumor (ATRTs) constitute 20% of brain tumors in young children (<3 yrs of age) and is characterized by bi-allelic mutations of the INI-1 gene. The cell of origin in this tumor is not known and the mechanism by which INI-1 causes tumor is still not clear. Ubiquitin carboxyl-terminal hydrolase isoenzyme 1 (UCHL1), is an abundant neuronal de-ubiquitinating enzyme which acts as a tumor suppressor gene that is inactivated by promoter methylation / gene deletion in several cancers as well as an oncogene with overexpression in other cancers. The role of UCHL1 in brain tumors has not yet been studied. METHODS/RESULTS: We identified UCHL1 is silenced in ATRT cell lines (BT-12, BT-16) along with absent basal expression of p53 and NOXA by western blotting. UCHL1 silencing is mediated by promoter hyper-methylation (detected by using methylation specific PCR (MSP)); and pharmacologic de-methylation reactivated UCHL1 expression in these cell lines along with expression of p53 and NOXA. Ectopic expression of UCHL1 dramatically inhibited the growth of ATRT cell lines (using colony forming assay and MTT assay) and increased sensitivity to radiation. The growth inhibition was mediated by promoting tumor cell apoptosis resulting from activating the p53 tumor suppressor pathway. CONCLUSIONS: We have shown that UCHL1 is silenced in ATRT cell lines and acts as a tumor suppressor gene. We hypothesize that loss of p53 function due to silencing of UCHL1 drives tumor formation in the background of bi-allelic INI1 mutation in ATRT. We will further test this hypothesis using patient derived primary cell cultures and animal models (using genetically engineered mouse models INI1 +/- / UCHL1-/- mice).

Fetched live from OpenAlex and de-inverted. Abstracts are not stored in this database: the inverted indexes are 8.6 GB of the frame’s 9.3 GB of text, and the host has 13 GB free.

How this classification was reachedexpand

Full frame distilled prediction

Teacher imitation

Not calibrated prevalence, not ground truth. Human validation pending. Learned from the 10,348 direct Codex labels and 10,348 direct Gemma labels. Candidate is the union of thresholded teacher heads; consensus is their intersection. These outputs are machine_predicted_unvalidated and are not human labels or direct frontier model labels.

metaresearch head score (Codex)0.000
metaresearch head score (Gemma)0.000
Version: codex-gemma-dda1882f352aValidation status: machine_predicted_unvalidated
Candidate categoriesMeta-epidemiology (narrow)
Consensus categoriesnone
DomainCandidate signal: none · Consensus signal: none
Study designCandidate signal: Bench or experimental · Consensus signal: Bench or experimental
GenreCandidate signal: Empirical · Consensus signal: Empirical
Teacher disagreement score0.179
Threshold uncertainty score1.000

Codex and Gemma teacher scores by category

CategoryCodexGemma
Metaresearch0.0000.000
Meta-epidemiology (narrow)0.0000.000
Meta-epidemiology (broad)0.0010.000
Bibliometrics0.0000.000
Science and technology studies0.0000.000
Scholarly communication0.0000.000
Open science0.0000.000
Research integrity0.0000.000
Insufficient payload (model declined to judge)0.0000.000

Machine scores (provisional)

The two teacher heads of the student model, read on this work. A score orders the frame for review; it never asserts a category, and the validation status ships verbatim with every row.

Baseline scores from an immature model (maturity gate not passed, 7 training rounds). Scores rank; they never assert a category.

Opus teacher head0.020
GPT teacher head0.290
Teacher spread0.271 · how far apart the two teachers sit on this one work
Validation statusscore_only:v0-immature-baseline · verbatim from the scoring run: score_only means the number may rank works, and no category label ships from it

Classification

machine, unvalidated

Machine predicted; a candidate call from one teacher head, not a consensus.

Study designBench or experimental
Domainnot available
GenreEmpirical

How this classification was reached, model by model and score by score, is at the end of the page under "How this classification was reached".

Quick stats

Citations1
Published2015
Admission routes1
Has abstractyes

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