Mutations of NOS1 and MLN regulatory sequences are a potential cause of infantile hypertrophic pyloric stenosis
Bibliographic record
Abstract
ENWEndNote BIBJabRef, Mendeley RISPapers, Reference Manager, RefWorks, Zotero AMA Jabłoński J, Drozdz I, Borowiec M, et al. Mutations of NOS1 and MLN regulatory sequences are a potential cause of infantile hypertrophic pyloric stenosis. Archives of Medical Science - Civilization Diseases. 2016;1(1):79-80. doi:10.5114/amscd.2016.62218. APA Jabłoński, J., Drozdz, I., Borowiec, M., Fendler, W., Młynarski, W., & Lewandowska, M. et al. (2016). Mutations of NOS1 and MLN regulatory sequences are a potential cause of infantile hypertrophic pyloric stenosis. Archives of Medical Science - Civilization Diseases, 1(1), 79-80. https://doi.org/10.5114/amscd.2016.62218 Chicago Jabłoński, Janusz, Izabela Drozdz, Maciej Borowiec, Wojciech Fendler, Wojciech Młynarski, Małgorzata Lewandowska, and Ewa Andrzejewska. 2016. "Mutations of NOS1 and MLN regulatory sequences are a potential cause of infantile hypertrophic pyloric stenosis". Archives of Medical Science - Civilization Diseases 1 (1): 79-80. doi:10.5114/amscd.2016.62218. Harvard Jabłoński, J., Drozdz, I., Borowiec, M., Fendler, W., Młynarski, W., Lewandowska, M., and Andrzejewska, E. (2016). Mutations of NOS1 and MLN regulatory sequences are a potential cause of infantile hypertrophic pyloric stenosis. Archives of Medical Science - Civilization Diseases, 1(1), pp.79-80. https://doi.org/10.5114/amscd.2016.62218 MLA Jabłoński, Janusz et al. "Mutations of NOS1 and MLN regulatory sequences are a potential cause of infantile hypertrophic pyloric stenosis." Archives of Medical Science - Civilization Diseases, vol. 1, no. 1, 2016, pp. 79-80. doi:10.5114/amscd.2016.62218. Vancouver Jabłoński J, Drozdz I, Borowiec M, Fendler W, Młynarski W, Lewandowska M et al. Mutations of NOS1 and MLN regulatory sequences are a potential cause of infantile hypertrophic pyloric stenosis. Archives of Medical Science - Civilization Diseases. 2016;1(1):79-80. doi:10.5114/amscd.2016.62218.
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How this classification was reachedexpand
Full frame machine prediction
Teacher imitationNot calibrated prevalence, not ground truth. Human validation pending. The Gemma side is a direct model label for every work in the frame, read from the title-only record. The Codex side is a classifier learned from the 10,348 direct Codex labels and calibrated to design-weighted sample rates; fields without enough sample support carry no Codex call. Candidate is the union of the two sides; consensus is their intersection. These outputs are machine_predicted_unvalidated and are not human labels.
Distilled classifier scores by category (both heads)
| Category | Codex | Gemma |
|---|---|---|
| Metaresearch | 0.002 | 0.006 |
| Meta-epidemiology (narrow) | 0.000 | 0.000 |
| Meta-epidemiology (broad) | 0.001 | 0.000 |
| Bibliometrics | 0.004 | 0.002 |
| Science and technology studies | 0.000 | 0.000 |
| Scholarly communication | 0.001 | 0.001 |
| Open science | 0.001 | 0.001 |
| Research integrity | 0.001 | 0.001 |
| Insufficient payload (model declined to judge) | 0.035 | 0.014 |
Machine scores (provisional)
The two teacher heads of the student model, read on this work. A score orders the frame for review; it never asserts a category, and the validation status ships verbatim with every row.
Baseline scores from an immature model (maturity gate not passed, 7 training rounds). Scores rank; they never assert a category.
score_only:v0-immature-baseline · verbatim from the scoring run: score_only means the number may rank works, and no category label ships from itClassification
machine, unvalidatedMachine predicted; a candidate call from one source (direct Gemma or distilled Codex), not a consensus.
How this classification was reached, model by model and score by score, is at the end of the page under "How this classification was reached".