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Record W2566120837 · doi:10.1161/hyp.64.suppl_1.058

Abstract 058: Sirt3 Impairment and SOD2 Hyperacetylation Drive Vascular Oxidative Stress and Hypertension

2014· article· en· W2566120837 on OpenAlexaff
Rafal R. Nazarewicz, Anna Dikalova, Joshua P. Fessel, Hana A. Itani, William G. McMaster, David G. Harrison, Sergey Dikalov

Bibliographic record

VenueHypertension · 2014
Typearticle
Languageen
FieldMedicine
TopicCardiovascular Health and Risk Factors
Canadian institutionsMcMaster University
Fundersnot available
KeywordsSIRT3SOD2EndocrinologyOxidative stressInternal medicineMitochondrionChemistrySuperoxide dismutaseMitochondrial ROSAngiotensin IIAcetylationMedicineBiologyPharmacologyBiochemistrySirtuinBlood pressure

Abstract

fetched live from OpenAlex

Aging is associated with increased incidence of hypertension and a decline of the mitochondrial energy regulator deacetylase Sirt3. A major mitochondrial antioxidant enzyme, SOD2, is inhibited by acetylation and its deacetylation by Sirt3 restores SOD2 activity. We hypothesized that loss of Sirt3 activity increases vascular oxidative stress due to SOD2 hyperacetylation and that this promotes hypertension. The combination of angiotensin II and TNFα, which is produced by inflammatory cells in hypertension, synergistically reduced Sirt3 expression, induced SOD2 acetylation and increased mitochondrial superoxide (O 2 • - ) in human aortic endothelial cells (HAEC). mitoEbselen, which scavenges mitochondrial H 2 O 2 , prevented SOD2 acetylation in HAEC and normalized mitochondrial O 2 • - suggesting redox-dependent modulation of Sirt3. In intact mice, chronic AngII infusion (490 ng/kg/min) reduced vascular Sirt3 expression by 20%, caused Sirt3 S-glutathionylation, SOD2 hyperacetylation and reduced SOD2 activity by 42%. Mice transgenic for mitochondria-targeted catalase exhibited reduced Sirt3 S-glutathionylation, no SOD2 hyperacetylation, maintenance of SOD2 activity and no increase in mitochondrial O 2 • - . The functional role of Sirt3 S-glutathionylation was further supported by mitoEbselen treatment of WT and Sirt3 -/- mice after the onset of AngII-induced hypertension. mitoEbselen reduced vascular oxidative stress and hypertension in WT but not in Sirt3 -/- mice. In Sirt3 -/- mice, low dose AngII (200 ng/kg/min) caused greater hypertension (150 mm Hg) than in WT (128 mm Hg, P<0.01) and AngII-infused Sirt3 -/- mice produced less nitric oxide that WT, as detected by electron spin resonance (70 pmol/aorta vs 110 WT+AngII, P< 0.01). We further showed that treatment with the mitochondria targeted SOD2 mimetic mitoTEMPO, initiated after the onset of AngII-induced hypertension lowered blood pressure and improved vasodilatation in both WT and Sirt3 -/- mice, further supporting a role of mitochondrial O 2 • - in hypertension. These data indicate that reduced Sirt3 activity occurs in hypertension due to S-glutathionylation and that this leads to SOD2 hyperacetylation and inactivation, promoting vascular oxidative stress and blood pressure elevation.

Fetched live from OpenAlex and de-inverted. Abstracts are not stored in this database: the inverted indexes are 8.6 GB of the frame’s 9.3 GB of text, and the host has 13 GB free.

How this classification was reachedexpand

Full frame machine prediction

Teacher imitation

Not calibrated prevalence, not ground truth. Human validation pending. The Gemma side is a direct model label for every work in the frame, read from the title-only record. The Codex side is a classifier learned from the 10,348 direct Codex labels and calibrated to design-weighted sample rates; fields without enough sample support carry no Codex call. Candidate is the union of the two sides; consensus is their intersection. These outputs are machine_predicted_unvalidated and are not human labels.

metaresearch head score (Codex)0.000
metaresearch head score (Gemma)0.000
Version: metacan-v3-hybrid-931329e0061cValidation status: machine_predicted_unvalidated
Candidate categoriesnone
Consensus categoriesnone
DomainCandidate signal: none · Consensus signal: none
Study designCandidate signal: Bench or experimental · Consensus signal: Bench or experimental
GenreCandidate signal: Empirical · Consensus signal: Empirical
Teacher disagreement score0.006
Threshold uncertainty score0.021

Distilled classifier scores by category (both heads)

CategoryCodexGemma
Metaresearch0.0000.000
Meta-epidemiology (narrow)0.0000.000
Meta-epidemiology (broad)0.0010.000
Bibliometrics0.0000.000
Science and technology studies0.0000.000
Scholarly communication0.0000.000
Open science0.0000.000
Research integrity0.0000.001
Insufficient payload (model declined to judge)0.0060.001

Machine scores (provisional)

The two teacher heads of the student model, read on this work. A score orders the frame for review; it never asserts a category, and the validation status ships verbatim with every row.

Baseline scores from an immature model (maturity gate not passed, 7 training rounds). Scores rank; they never assert a category.

Opus teacher head0.015
GPT teacher head0.237
Teacher spread0.222 · how far apart the two teachers sit on this one work
Validation statusscore_only:v0-immature-baseline · verbatim from the scoring run: score_only means the number may rank works, and no category label ships from it

Classification

machine, unvalidated

Machine predicted; a candidate call from one source (direct Gemma or distilled Codex), not a consensus.

The models applied no category: nothing in the taxonomy fit this work.
Study designBench or experimental
Domainnot available
GenreEmpirical

How this classification was reached, model by model and score by score, is at the end of the page under "How this classification was reached".

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Citations0
Published2014
Admission routes1
Has abstractyes

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