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Record W2589576518 · doi:10.1038/ncomms14294

An integrative approach unveils FOSL1 as an oncogene vulnerability in KRAS-driven lung and pancreatic cancer

2017· article· en· W2589576518 on OpenAlexaff
Adrián Vallejo, Naiara Perurena, Elizabeth Guruceaga, Paweł K. Mazur, Susana Martínez-Canarias, Carolina Zandueta, Karmele Valencia, Andrea Arricibita, Dana M. Gwinn, Leanne C. Sayles, Chen-Hua Chuang, Laura Guembe, Peter J. Bailey, David K. Chang, Andrew V. Biankin, Mariano Ponz‐Sarvisé, Jesper B. Andersen, Purvesh Khatri, Aline Bözec, E. Alejandro Sweet‐Cordero, Julien Sage, Fernando Lecanda, Silvestre Vicent

Bibliographic record

VenueNature Communications · 2017
Typearticle
Languageen
FieldMedicine
TopicPancreatic and Hepatic Oncology Research
Canadian institutionsInstitute of Infection and Immunity
FundersNational Cancer InstituteNational Institute of Allergy and Infectious DiseasesMedical Research CouncilNational Institutes of HealthPancreatic Cancer UKEuropean CommissionFP7 People: Marie-Curie ActionsMinisterio de Economía y CompetitividadNovo Nordisk FondenCancer Research UKLustgarten FoundationMinisterio de Ciencia e InnovaciónMinisterio de Educación, Cultura y DeporteWellcome TrustTobacco-Related Disease Research ProgramInternational Association for the Study of Lung CancerKræftens Bekæmpelse
KeywordsKRASVulnerability (computing)Pancreatic cancerOncogeneLung cancerMedicineCancer researchBiologyCancerComputer scienceOncologyInternal medicineColorectal cancerComputer securityCell cycle

Abstract

fetched live from OpenAlex

KRAS mutated tumours represent a large fraction of human cancers, but the vast majority remains refractory to current clinical therapies. Thus, a deeper understanding of the molecular mechanisms triggered by KRAS oncogene may yield alternative therapeutic strategies. Here we report the identification of a common transcriptional signature across mutant KRAS cancers of distinct tissue origin that includes the transcription factor FOSL1. High FOSL1 expression identifies mutant KRAS lung and pancreatic cancer patients with the worst survival outcome. Furthermore, FOSL1 genetic inhibition is detrimental to both KRAS-driven tumour types. Mechanistically, FOSL1 links the KRAS oncogene to components of the mitotic machinery, a pathway previously postulated to function orthogonally to oncogenic KRAS. FOSL1 targets include AURKA, whose inhibition impairs viability of mutant KRAS cells. Lastly, combination of AURKA and MEK inhibitors induces a deleterious effect on mutant KRAS cells. Our findings unveil KRAS downstream effectors that provide opportunities to treat KRAS-driven cancers.

Fetched live from OpenAlex and de-inverted. Abstracts are not stored in this database: the inverted indexes are 8.6 GB of the frame’s 9.3 GB of text, and the host has 13 GB free.

How this classification was reachedexpand

Full frame machine prediction

Teacher imitation

Not calibrated prevalence, not ground truth. Human validation pending. The Gemma side is a direct model label for every work in the frame, read from the title-only record. The Codex side is a classifier learned from the 10,348 direct Codex labels and calibrated to design-weighted sample rates; fields without enough sample support carry no Codex call. Candidate is the union of the two sides; consensus is their intersection. These outputs are machine_predicted_unvalidated and are not human labels.

metaresearch head score (Codex)0.000
metaresearch head score (Gemma)0.000
Version: metacan-v3-hybrid-931329e0061cValidation status: machine_predicted_unvalidated
Candidate categoriesnone
Consensus categoriesnone
DomainCandidate signal: none · Consensus signal: none
Study designCandidate signal: Bench or experimental · Consensus signal: Bench or experimental
GenreCandidate signal: Empirical · Consensus signal: Empirical
Teacher disagreement score0.002
Threshold uncertainty score0.008

Distilled classifier scores by category (both heads)

CategoryCodexGemma
Metaresearch0.0000.000
Meta-epidemiology (narrow)0.0000.000
Meta-epidemiology (broad)0.0000.000
Bibliometrics0.0000.000
Science and technology studies0.0000.000
Scholarly communication0.0010.000
Open science0.0000.001
Research integrity0.0000.001
Insufficient payload (model declined to judge)0.0020.001

Machine scores (provisional)

The two teacher heads of the student model, read on this work. A score orders the frame for review; it never asserts a category, and the validation status ships verbatim with every row.

Baseline scores from an immature model (maturity gate not passed, 7 training rounds). Scores rank; they never assert a category.

Opus teacher head0.058
GPT teacher head0.462
Teacher spread0.404 · how far apart the two teachers sit on this one work
Validation statusscore_only:v0-immature-baseline · verbatim from the scoring run: score_only means the number may rank works, and no category label ships from it

Classification

machine, unvalidated

Machine predicted; a candidate call from one source (direct Gemma or distilled Codex), not a consensus.

The models applied no category: nothing in the taxonomy fit this work.
Study designBench or experimental
Domainnot available
GenreEmpirical

How this classification was reached, model by model and score by score, is at the end of the page under "How this classification was reached".

Quick stats

Citations196
Published2017
Admission routes1
Has abstractyes

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