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Record W2595238557 · doi:10.1093/biolreprod/83.s1.601

Role of Prohibitin in Regulating FSH-Induced Steroidogenesis During Follicular Development.

2010· article· en· W2595238557 on OpenAlexaffabout
Qi Wang, Benjamin K. Tsang

Bibliographic record

VenueBiology of Reproduction · 2010
Typearticle
Languageen
FieldMedicine
TopicReproductive Biology and Fertility
Canadian institutionsUniversity of OttawaOttawa Hospital
Fundersnot available
KeywordsBiologyGonadotropinFollicular phaseGranulosa cellAntral follicleInternal medicineOvarian follicleEndocrinologyHuman chorionic gonadotropinCell biologyHormoneMedicine

Abstract

fetched live from OpenAlex

With the advances in gonadotropin research, treatment of human infertility with gonadotropin preparations has become a common practice. It plays critical roles in stimulating ovarian cell proliferation, differentiation and suppressing apoptosis, which ultimately induces ovarian follicle growth and ovulation. The action of gonadotropin during follicular development is however complex and is stage-dependent, thus making optimization of gonadotropin treatment for various ovarian disorders often difficult. Prohibitin (PHB) is an intracellular protein important for cell cycle regulation, apoptosis and cellular differentiation. Ovarian PHB is up-regulated by equine chorionic gonadotropin (eCG) in vivo. Although PHB prevents cell apoptosis induced by the alkaloid staurosporine in undifferentiated granulosa cells (from primarily preantral/early antral follicles), whether the expression and action of PHB are similarly regulated in differentiated granulosa cells (large antral and preovulatory follicles) are unknown. We hypothesize that PHB is an important regulator of granulosa cell fate and plays a role in regulating steroidogenesis during follicular development. Our objective is to better understand the function and regulation of PHB during follicular development, especially during FSH-induced antral follicle growth. Using granulosa cells from eCG-primed immature rats, we observed that (1) FSH up-regulates granulosa cell PHB expression in a follicular stage-dependent manner in vitro. (2) Akt content and its phosphorylation are required for the regulation of PHB by FSH. (3) Exogenous PHB significantly reduces FSH-stimulated estradiol and progesterone production in granulosa cells. (4) cyp11a1 (p450 side-chain cleavage enzyme) mRNA and protein expression in granulosa cells are down-regulated by exogenous PHB. (5) The mRNA levels of other steroidogenic enzymes, such as cyp19 (aromatase) and Steroidogenic acute regulatory protein (StAR), are also down-regulated by exogenous PHB. Conclusion: This study demonstrates that granulosa cell PHB is regulated by FSH in a follicular stage-dependent manner and that PHB regulates FSH-induced steroidogenesis in granulosa cells. Our findings raise the possibility that the dysregulation of PHB expression and function may be linked to infertility associated with compromised follicular growth and differentiation (e.g. Polycystic ovarian syndrome) and that PHB may be a therapeutic target for the treatment of infertility. Further experiments are required to test these possibilities.Supported by Canadian Institutes of Health Research (CIHR). (poster)

Fetched live from OpenAlex and de-inverted. Abstracts are not stored in this database: the inverted indexes are 8.6 GB of the frame’s 9.3 GB of text, and the host has 13 GB free.

How this classification was reachedexpand

Full frame machine prediction

Teacher imitation

Not calibrated prevalence, not ground truth. Human validation pending. The Gemma side is a direct model label for every work in the frame, read from the title-only record. The Codex side is a classifier learned from the 10,348 direct Codex labels and calibrated to design-weighted sample rates; fields without enough sample support carry no Codex call. Candidate is the union of the two sides; consensus is their intersection. These outputs are machine_predicted_unvalidated and are not human labels.

metaresearch head score (Codex)0.000
metaresearch head score (Gemma)0.000
Version: metacan-v3-hybrid-931329e0061cValidation status: machine_predicted_unvalidated
Candidate categoriesnone
Consensus categoriesnone
DomainCandidate signal: none · Consensus signal: none
Study designCandidate signal: Bench or experimental · Consensus signal: Bench or experimental
GenreCandidate signal: Empirical · Consensus signal: Empirical
Teacher disagreement score0.002
Threshold uncertainty score0.004

Distilled classifier scores by category (both heads)

CategoryCodexGemma
Metaresearch0.0000.000
Meta-epidemiology (narrow)0.0000.000
Meta-epidemiology (broad)0.0000.000
Bibliometrics0.0000.000
Science and technology studies0.0000.000
Scholarly communication0.0000.000
Open science0.0000.000
Research integrity0.0000.000
Insufficient payload (model declined to judge)0.0010.000

Machine scores (provisional)

The two teacher heads of the student model, read on this work. A score orders the frame for review; it never asserts a category, and the validation status ships verbatim with every row.

Baseline scores from an immature model (maturity gate not passed, 7 training rounds). Scores rank; they never assert a category.

Opus teacher head0.018
GPT teacher head0.267
Teacher spread0.248 · how far apart the two teachers sit on this one work
Validation statusscore_only:v0-immature-baseline · verbatim from the scoring run: score_only means the number may rank works, and no category label ships from it

Classification

machine, unvalidated

Machine predicted; a candidate call from one source (direct Gemma or distilled Codex), not a consensus.

The models applied no category: nothing in the taxonomy fit this work.
Study designBench or experimental
Domainnot available
GenreEmpirical

How this classification was reached, model by model and score by score, is at the end of the page under "How this classification was reached".

Quick stats

Citations0
Published2010
Admission routes2
Has abstractyes

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