MétaCan
Menu
← Back to cohort
Record W2766067036 · doi:10.1016/j.jalz.2017.06.468

[P1–452]: COMBINED GLOBAL AND REGIONAL AMYLOID EFFECT ON THE DEFAULT MODE NETWORK LEADS TO COGNITIVE DECLINE

2017· article· en· W2766067036 on OpenAlexaffabout
Tharick A. Pascoal, Sulantha Mathotaarachchi, Min Su Kang, Monica Shin, Andréa Lessa Benedet, Jean‐Paul Soucy, A. Claudio Cuello, Serge Gauthier, Pedro Rosa‐Neto, Hanne Struyfs, Kok Pin Ng, Joseph Therriault

Bibliographic record

VenueAlzheimer s & Dementia · 2017
Typearticle
Languageen
FieldMedicine
TopicAlzheimer's disease research and treatments
Canadian institutionsUniversité de MontréalConcordia UniversityDouglas CollegeDouglas Mental Health University InstituteMcGill University
Fundersnot available
KeywordsDefault mode networkDementiaAmyloid (mycology)Positron emission tomographyCognitionCognitive declineVoxelNeuroimagingInternal medicinePittsburgh compound BStandardized uptake valuePsychologyNeuroscienceMedicineOncologyDiseasePathologyRadiology

Abstract

fetched live from OpenAlex

The association between amyloid-β aggregates, default mode network (DMN) dysfunction, and dementia symptoms remains unclear in Alzheimer's disease (AD). Although individuals presenting both abnormal amyloid-β and hypometabolism in posterior DMN are especially vulnerable to disease progression, the lack of a strong association between regional amyloid-β burden and metabolic or cognitive dysfunction has puzzled researchers. The present study was designed to test the hypothesis that the widespread global amyloid-β aggregation determines the metabolic dysfunction of the brain's posterior DMN, whereas a synergistic interaction between the regional toxic effects of amyloid-β aggregates and the levels of local network dysfunction determines the subsequent clinical progression to AD dementia. We performed a longitudinal study using a computational framework developed to perform voxel-wise multimodal statistics in human or animal brain image (Fig.1). We studied 347 mild cognitive impairment (MCI) ADNI participants and 20 transgenic McGill-R-Thy1-APP rats overexpressing amyloid-β precursor protein with mild cognitive symptoms with T1-weighted magnetic resonance imaging, [F]fluorodeoxyglucose and amyloid-β positron emission tomography (PET) at baseline, as well as cognition at baseline and follow-up. Voxel-wise regression analyses taking into consideration global and voxel standardized uptake values tested the associations between Aβ deposition, glucose metabolism, and cognition (MCIs, all available follow-ups up to 5.6 years; rats, 8-month follow-up). Analysis of covariance was used to further compare the models. We found that global brain amyloid-β burden determined regional metabolic hypometabolism in the functional hubs of the brain's posterior DMN at baseline (P < 0.001). Furthermore, we found that the regional, rather than the global, levels of amyloid-β aggregates in posterior DMN synergistically interact with the regional levels of network dysfunction to determine subsequent clinical progression to dementia. Notably, the same results in the posterior DMN of the transgenic amyloid-β rats, which do not form neurofibrillary tangles, supported this model as an independent mechanism of cognitive deterioration (Fig.2). These findings highlight a model where a widespread amyloid-β aggregation determines the vulnerability of the well conserved – in mammals – DMN, whereas the synergism between this vulnerability and the regional concentrations of amyloid-β aggregates determines dementia symptoms.

Fetched live from OpenAlex and de-inverted. Abstracts are not stored in this database: the inverted indexes are 8.6 GB of the frame’s 9.3 GB of text, and the host has 13 GB free.

How this classification was reachedexpand

Full frame machine prediction

Teacher imitation

Not calibrated prevalence, not ground truth. Human validation pending. The Gemma side is a direct model label for every work in the frame, read from the title-only record. The Codex side is a classifier learned from the 10,348 direct Codex labels and calibrated to design-weighted sample rates; fields without enough sample support carry no Codex call. Candidate is the union of the two sides; consensus is their intersection. These outputs are machine_predicted_unvalidated and are not human labels.

metaresearch head score (Codex)0.000
metaresearch head score (Gemma)0.001
Version: metacan-v3-hybrid-931329e0061cValidation status: machine_predicted_unvalidated
Candidate categoriesnone
Consensus categoriesnone
DomainCandidate signal: none · Consensus signal: none
Study designCandidate signal: Observational · Consensus signal: Observational
GenreCandidate signal: Empirical · Consensus signal: Empirical
Teacher disagreement score0.004
Threshold uncertainty score0.014

Distilled classifier scores by category (both heads)

CategoryCodexGemma
Metaresearch0.0000.001
Meta-epidemiology (narrow)0.0000.000
Meta-epidemiology (broad)0.0000.000
Bibliometrics0.0000.000
Science and technology studies0.0000.000
Scholarly communication0.0000.000
Open science0.0000.000
Research integrity0.0000.000
Insufficient payload (model declined to judge)0.0040.000

Machine scores (provisional)

The two teacher heads of the student model, read on this work. A score orders the frame for review; it never asserts a category, and the validation status ships verbatim with every row.

Baseline scores from an immature model (maturity gate not passed, 7 training rounds). Scores rank; they never assert a category.

Opus teacher head0.038
GPT teacher head0.350
Teacher spread0.312 · how far apart the two teachers sit on this one work
Validation statusscore_only:v0-immature-baseline · verbatim from the scoring run: score_only means the number may rank works, and no category label ships from it

Classification

machine, unvalidated

Machine predicted; a candidate call from one source (direct Gemma or distilled Codex), not a consensus.

The models applied no category: nothing in the taxonomy fit this work.
Study designObservational
Domainnot available
GenreEmpirical

How this classification was reached, model by model and score by score, is at the end of the page under "How this classification was reached".

Quick stats

Citations0
Published2017
Admission routes2
Has abstractyes

Explore more

Same venueAlzheimer s & Dementia→Same topicAlzheimer's disease research and treatments→French-language works237,207→