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Record W2772722798

Resveratrol mediates cell fate in human uterine cancer cells through prostaglandin inhibition

2005· article· en· W2772722798 on OpenAlexaff
Émilie Sexton, Kim LeBlanc, Pascal Lemoine, Éric Asselin

Bibliographic record

VenueCancer Research · 2005
Typearticle
Languageen
FieldMedicine
TopicInflammatory mediators and NSAID effects
Canadian institutionsUniversité du Québec à Trois-Rivières
Fundersnot available
KeywordsResveratrolCell growthProstaglandin E2 receptorProstaglandinCancer cellApoptosisCell cultureArachidonic acidReceptorProstaglandin E2CellCell cycleHeLaProstaglandin EChemistryMTT assayCancer researchBiologyCancerPharmacologyEndocrinologyInternal medicineBiochemistryMedicineEnzyme
DOInot available

Abstract

fetched live from OpenAlex

751 In the Western world, endometrial cancer is the most common gynecologic cancer and is the fourth importance among all feminine cancers. Cyclooxygenase-1 and -2 (COX-1 and -2) are the limiting enzyme that catalyzes the synthesis of prostaglandins (PGs) from arachidonic acid. The PGs have been shown to be implicated in evolution and tumor transformation, particularly, PGE2. Four prostaglandin E2 receptors have been found: EP1, EP2, EP3 and EP4. The aim of this study was to determine if resveratrol, a natural anti-oxidant found in red wine, could influence the production of PGs and apoptotic cell death or cellular proliferation. Six different human uterine cancer cell lines were used for this study. Real-time RT-PCR and Western analyses were carried out to measure COX-1, COX-2, EP1, EP2, EP3 and EP4 expression. The results indicated that all cell lines expressed both COX isoforms and the four EP receptors at different levels. In experiment two, cells were treated with various concentrations of resveratrol for different length of time. MTT proliferation assay revealed that resveratrol induced maximal cell proliferation of RL-95-2 cells in a dose dependent manner after 24 hours, which was reduced after 48 and 72 hours. Resveratrol had no effect on KLE cells in term of proliferation but inhibited cell proliferation in HeLa and HEC-1-A in a dose and time dependant manner. In Ishikawa and TOV-1078D cells, a novel human endometrial cancer cell line, the effect of resveratrol was biphasic: cell proliferation was increased at low concentration whereas higher concentrations resulted in a decreased of cell proliferation in a time dependent manner. Hoechst nuclear staining indicated that resveratrol induced apoptosis in HeLa, HEC-1-A, Ishikawa and TOV-1078D but not in KLE and RL-95-2 cells. Since we have showed that Akt activity can directly up-regulate COX-2 gene expression and PGE2 secretion, in experiment two Western blot analyses were carried out to determine the possible involvement of the PI3-K/PTEN/Akt pathway in this process. Results indicate that, depending on the cell line, resveratrol can act positively and negatively on Akt phosphorylation in those wild types (HeLa, HEC-1-A and KLE) and mutated PTEN (RL-95-2, Ishikawa and TOV-1078D) human uterine cancer cells and that COX-1 and COX-2 expressions are influenced by resveratrol. In response to resveratrol PGE2 secretion was decreased in HeLa, HEC-1-A and Ishikawa cells. The results suggest that resveratrol, through its action on prostaglandin biosynthesis, could be an important regulator of cell survival in uterine cancer. Supported by CIHR (MOP-66987).

Fetched live from OpenAlex and de-inverted. Abstracts are not stored in this database: the inverted indexes are 8.6 GB of the frame’s 9.3 GB of text, and the host has 13 GB free.

How this classification was reachedexpand

Full frame machine prediction

Teacher imitation

Not calibrated prevalence, not ground truth. Human validation pending. The Gemma side is a direct model label for every work in the frame, read from the title-only record. The Codex side is a classifier learned from the 10,348 direct Codex labels and calibrated to design-weighted sample rates; fields without enough sample support carry no Codex call. Candidate is the union of the two sides; consensus is their intersection. These outputs are machine_predicted_unvalidated and are not human labels.

metaresearch head score (Codex)0.000
metaresearch head score (Gemma)0.000
Version: metacan-v3-hybrid-931329e0061cValidation status: machine_predicted_unvalidated
Candidate categoriesnone
Consensus categoriesnone
DomainCandidate signal: none · Consensus signal: none
Study designCandidate signal: Bench or experimental · Consensus signal: Bench or experimental
GenreCandidate signal: Empirical · Consensus signal: Empirical
Teacher disagreement score0.002
Threshold uncertainty score0.007

Distilled classifier scores by category (both heads)

CategoryCodexGemma
Metaresearch0.0000.000
Meta-epidemiology (narrow)0.0000.000
Meta-epidemiology (broad)0.0000.000
Bibliometrics0.0000.000
Science and technology studies0.0000.000
Scholarly communication0.0000.000
Open science0.0000.000
Research integrity0.0000.001
Insufficient payload (model declined to judge)0.0020.001

Machine scores (provisional)

The two teacher heads of the student model, read on this work. A score orders the frame for review; it never asserts a category, and the validation status ships verbatim with every row.

Baseline scores from an immature model (maturity gate not passed, 7 training rounds). Scores rank; they never assert a category.

Opus teacher head0.051
GPT teacher head0.411
Teacher spread0.360 · how far apart the two teachers sit on this one work
Validation statusscore_only:v0-immature-baseline · verbatim from the scoring run: score_only means the number may rank works, and no category label ships from it

Classification

machine, unvalidated

Machine predicted; a candidate call from one source (direct Gemma or distilled Codex), not a consensus.

The models applied no category: nothing in the taxonomy fit this work.
Study designBench or experimental
Domainnot available
GenreEmpirical

How this classification was reached, model by model and score by score, is at the end of the page under "How this classification was reached".

Quick stats

Citations2
Published2005
Admission routes1
Has abstractyes

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