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Record W2773425422 · doi:10.1096/fj.201700700r

Correction of GSK3ß at young age prevents muscle pathology in mice with myotonic dystrophy type 1

2017· article· en· W2773425422 on OpenAlexaff
Christina Wei, Lauren Stock, Leila Valanejad, Zachary A. Zalewski, Rebekah Karns, Jack Puymirat, David L. Nelson, David P. Witte, Nikolai A. Timchenko, Lubov Imchenko

Bibliographic record

VenueThe FASEB Journal · 2017
Typearticle
Languageen
FieldNeuroscience
TopicGenetic Neurodegenerative Diseases
Canadian institutionsLunenfeld-Tanenbaum Research InstituteMount Sinai HospitalCentre hospitalier de l'Université Laval
FundersNational Institute of Arthritis and Musculoskeletal and Skin DiseasesNational Institutes of HealthNational Cancer InstituteNational Institute of Diabetes and Digestive and Kidney DiseasesUniversity of California, DavisNational Institute of General Medical SciencesUniversity of Rochester
KeywordsMyotonic dystrophyBiologyMyogenesisEndocrinologyInternal medicineCell biologySkeletal muscleGeneticsMedicine

Abstract

fetched live from OpenAlex

Myotonic dystrophy type 1 (DM1) is a progressive neuromuscular disease caused by expanded CUG repeats, which misregulate RNA metabolism through several RNA‐binding proteins, including CUG‐binding protein/CUGBP1 elav‐like factor 1 (CUGBP1/CELF1) and muscleblind 1 protein. Mutant CUG repeats elevate CUGBP1 and alter CUGBP1 activity via a glycogen synthase kinase 3β (GSK3β)‐cyclin D3‐cyclin D‐dependent kinase 4 (CDK4) signaling pathway. Inhibition of GSK3β corrects abnormal activity of CUGBP1 in DM1 mice [human skeletal actin mRNA, containing long repeats ( HSA LR ) model]. Here, we show that the inhibition of GSK3β in young HSA LR mice prevents development of DM1 muscle pathology. Skeletal muscle in 1‐yr‐old hsa lr mice, treated at 1.5 mo for 6 wk with the inhibitors of GSK3, exhibits high fiber density, corrected atrophy, normal fiber size, with reduced central nuclei and normalized grip strength. Because CUG‐GSK3β‐cyclin D3‐CDK4 converts the active form of CUGBP1 into a form of translational repressor, we examined the contribution of CUGBP1 in myogenesis using Celf1 knockout mice. We found that a loss of CUGBP1 disrupts myogenesis, affecting genes that regulate differentiation and the extracellular matrix. Proteins of those pathways are also misregulated in young hsa lr mice and in muscle biopsies of patients with congenital DM1. These findings suggest that the correction of GSK3β‐CUGBP1 pathway in young hsa lr mice might have a positive effect on the myogenesis over time.— Wei, C., Stock, L., Valanejad, L., Zalewski, Z. A., Karns, R., Puymirat, J., Nelson, D., Witte, D., Woodgett, J., Timchenko, N. A., Timchenko, L. Correction of GSK3ß at young age prevents muscle pathology in mice with myotonic dystrophy type 1. FASEB J. 32, 2073–2085 (2018). www.fasebj.org

Fetched live from OpenAlex and de-inverted. Abstracts are not stored in this database: the inverted indexes are 8.6 GB of the frame’s 9.3 GB of text, and the host has 13 GB free.

How this classification was reachedexpand

Full frame distilled prediction

Teacher imitation

Not calibrated prevalence, not ground truth. Human validation pending. Learned from the 10,348 direct Codex labels and 10,348 direct Gemma labels. Candidate is the union of thresholded teacher heads; consensus is their intersection. These outputs are machine_predicted_unvalidated and are not human labels or direct frontier model labels.

metaresearch head score (Codex)0.000
metaresearch head score (Gemma)0.000
Version: codex-gemma-dda1882f352aValidation status: machine_predicted_unvalidated
Candidate categoriesnone
Consensus categoriesnone
DomainCandidate signal: none · Consensus signal: none
Study designCandidate signal: Bench or experimental · Consensus signal: Bench or experimental
GenreCandidate signal: Empirical · Consensus signal: Empirical
Teacher disagreement score0.235
Threshold uncertainty score0.321

Codex and Gemma teacher scores by category

CategoryCodexGemma
Metaresearch0.0000.000
Meta-epidemiology (narrow)0.0000.000
Meta-epidemiology (broad)0.0000.000
Bibliometrics0.0000.000
Science and technology studies0.0000.000
Scholarly communication0.0000.000
Open science0.0000.000
Research integrity0.0000.000
Insufficient payload (model declined to judge)0.0000.000

Machine scores (provisional)

The two teacher heads of the student model, read on this work. A score orders the frame for review; it never asserts a category, and the validation status ships verbatim with every row.

Baseline scores from an immature model (maturity gate not passed, 7 training rounds). Scores rank; they never assert a category.

Opus teacher head0.028
GPT teacher head0.268
Teacher spread0.241 · how far apart the two teachers sit on this one work
Validation statusscore_only:v0-immature-baseline · verbatim from the scoring run: score_only means the number may rank works, and no category label ships from it

Classification

machine, unvalidated

Machine predicted; a candidate call from one teacher head, not a consensus.

The models applied no category: nothing in the taxonomy fit this work.
Study designBench or experimental
Domainnot available
GenreEmpirical

How this classification was reached, model by model and score by score, is at the end of the page under "How this classification was reached".

Quick stats

Citations31
Published2017
Admission routes1
Has abstractyes

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