MétaCan
Menu
Back to cohort
Record W2789334038 · doi:10.1093/jcag/gwy009.085

A85 SEMAPHORIN-3E AMELIORATES INTESTINAL INFLAMMATION BY REGULATING EXPRESSION OF TIGHT JUNCTION PROTEIN AND PROINFLAMMATORY MEDIATORS

2018· article· en· W2789334038 on OpenAlex

Why this work is in the frame

A frame that forgets how it found something cannot be audited. These are the routes that admitted this work.

affAt least one author lists a Canadian institution in the pinned OpenAlex snapshot.

Bibliographic record

VenueJournal of the Canadian Association of Gastroenterology · 2018
Typearticle
Languageen
FieldBiochemistry, Genetics and Molecular Biology
TopicMedicinal Plants and Bioactive Compounds
Canadian institutionsUniversity of Manitoba
Fundersnot available
KeywordsSemaphorinPlexinProinflammatory cytokineWound healingImmune systemUlcerative colitisColitisInflammationTumor necrosis factor alphaImmunofluorescenceImmunologyChemistryReceptorAndrologyCancer researchBiologyPathologyMedicineInternal medicineAntibody

Abstract

fetched live from OpenAlex

Ulcerative colitis (UC) is characterized by an alterations of tight junction (TJ) barrier and immune responses that result in abnormal passages of exogenous antigens such as gut-derived bacterial lipopolysaccharides (LPS). Semaphorin-3E (Sema3E) and its receptor plexin D1 (PLXD1R) are key regulators of epithelia and immune systems To investigate Sema3E activity in human and animal model of colitis and its effects on the intestinal epithelial cell functions Sema3E mRNA level and its correlation with TJ proteins and mucosal healing mediator (Krupple-Like Factor [KLF]-5) were quantified in human rectal biopsies. PLXD1R expression was quantified by immunofluorescence/RT-qPCR in Caco2 epithelial cell line and mice colonic tissues. Proliferation, metabolism, viability and wound healing properties were evaluated after 24 hours of treatment with Sema3E recombinant protein (100ng/ml). TJ proteins and KLF-5 were quantified by RT-qPCR in Caco-2 cells pretreated with Sema3E for 24 h and challenged with LPS (1μg/ml) for additional 24 h. Dextran sulfate sodium (DSS)-colitis was induced in BL6/57 mice for 5 days. Sema3E treatment (10µg/kg/day/intra-peritoneal) started at day 5 after induction of colitis and lasted for 4 days. Disease activity index (DAI), macroscopic and histological scores were assessed. mRNA and protein levels of pro-inflammatory cytokines and KLF5 were quantified. Sema3E is reduced in active and mild UC patients and has strong positive correlation with CLDN1 (r = 0.54, P =0.03), ZO-1 (r = 0.53, P = 0.03), CADH1 (r = 0.60, P =0.01), OCLDN (r = 0.64, P =0.008), KLF-5 (r = 0.52, P =0.03), TLR2 (r = - 0.6147, P =0.0103) and TLR4 (r = - 0.5794, P =0.0207). PLXD1R is expressed by Caco2 cells and mice colonic tissue. Sema3E accelerated significantly the epithelial cells migration when compared to control group. No significant changes on epithelial cells proliferation, metabolism and viability were detected. Compared to LPS-induced inflammation, Sema3E treatment increased the expression levels of TJ proteins (Cldn1, Zo-1, ECadh1, Ocldn) and Klf5, and decreased Tlr2 without significant changes in Tlr4. Sema3E treatment decreases DAI, macroscopic and histological scores in DSS-induced colitis compared with control. Il6 and Mcp1 were significantly decrease in colitic mice treated with Sema3E when compared to untreated mice. Il1bTnfa tend to decrease whereas Klf5 tends to increase. No changes in colonic levels of Tlr2, Tlr4, Cldn1, Zo-1 and Ocldn were detected. Sema3E regulates intestinal inflammation through epithelial cells barrier markers in human and cell line and reduces experimental inflammation and proinflammatory mediators. These findings may lead to improved therapeutic strategies in the treatment of UC by restoring Sema3E level in UC patients. CCC, CIHR

Fetched live from OpenAlex and de-inverted. Abstracts are not stored in this database: the inverted indexes are 8.6 GB of the frame’s 9.3 GB of text, and the host has 13 GB free.

Full frame distilled prediction

Teacher imitation

Not calibrated prevalence, not ground truth. Human validation pending. Learned from the 10,348 direct Codex labels and 10,348 direct Gemma labels. Candidate is the union of thresholded teacher heads; consensus is their intersection. These outputs are machine_predicted_unvalidated and are not human labels or direct frontier model labels.

metaresearch head score (Codex)0.001
metaresearch head score (Gemma)0.000
Version: codex-gemma-dda1882f352aValidation status: machine_predicted_unvalidated
Candidate categoriesnone
Consensus categoriesnone
DomainCandidate signal: none · Consensus signal: none
Study designCandidate signal: Bench or experimental · Consensus signal: Bench or experimental
GenreCandidate signal: Empirical · Consensus signal: Empirical
Teacher disagreement score0.086
Threshold uncertainty score0.716

Codex and Gemma teacher scores by category

CategoryCodexGemma
Metaresearch0.0010.000
Meta-epidemiology (narrow)0.0000.000
Meta-epidemiology (broad)0.0000.000
Bibliometrics0.0000.000
Science and technology studies0.0000.000
Scholarly communication0.0000.000
Open science0.0000.000
Research integrity0.0000.000
Insufficient payload (model declined to judge)0.0000.000

Machine scores (provisional)

The two teacher heads of the student model, read on this work. A score orders the frame for review; it never asserts a category, and the validation status ships verbatim with every row.

Baseline scores from an immature model (maturity gate not passed, 7 training rounds). Scores rank; they never assert a category.

Opus teacher head0.004
GPT teacher head0.201
Teacher spread0.197 · how far apart the two teachers sit on this one work
Validation statusscore_only:v0-immature-baseline · verbatim from the scoring run: score_only means the number may rank works, and no category label ships from it