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Record W2791537540 · doi:10.1093/jcag/gwy008.117

A116 ABERRANT IMMUNE RESPONSE AGAINST INVADING BACTERIA INCREASES MORTALITY IN MUC2 MUCIN DEFICIENT MICE IN LPS INDUCED SEPSIS

2018· article· en· W2791537540 on OpenAlex

Why this work is in the frame

A frame that forgets how it found something cannot be audited. These are the routes that admitted this work.

affAt least one author lists a Canadian institution in the pinned OpenAlex snapshot.

Bibliographic record

VenueJournal of the Canadian Association of Gastroenterology · 2018
Typearticle
Languageen
FieldMedicine
TopicMicroscopic Colitis
Canadian institutionsUniversity of Calgary
Fundersnot available
KeywordsBiologyMucusImmunologyColitisInflammationImmune systemLipopolysaccharideCecumSepsisMucin

Abstract

fetched live from OpenAlex

A physical barrier in the gut, formed mainly by the mucus bilayer, keeps gut microbiota and/or components away from translocating into host tissues. Perturbation of the mucus layer, as in inflammatory bowel diseases (IBD), leads microbial penetrants to initiate local and systemic inflammation. Unregulated inflammation can aggravate into sepsis, causing mortality due to multiple organ failure. IBD patients are at high risk for developing sepsis due to their defective gut barrier function, yet is poorly understood. Muc2-/- mice without a mucus barrier have low-grade inflammation with bacterial penetrants and is an excellent model to study chronic colitis and ideal for IBD related studies. To assess immune response in Muc2-/- mice in lipopolysaccharide (LPS) induced sepsis. LPS was administered in Muc2-/- and WT littermates intra-peritoneally (i.p.) as 5mg/kg body weight (BW) and monitored for BW loss and mortality. Mice were euthanized at 24 or 48 h. Spleen and intestinal tissue were harvested for immunophenotyping and histology. Single cell suspension of splenocytes were prepared and stained with anti CD3, CD4, CD8, CD19 and CD49b antibodies for T-cells, B-cells and NK cells. Apoptotic and dead cells were determined by Annexin V and 7-AAD staining. Immunophenotyping was done using FACS Canto and data was analyzed by FlowJo software. Bacterial penetrance into distal ileum, cecum and colonic tissue was determined using fluorescence in situ hybridization (FISH) and real time RT-PCR. Muc2-/- mice basally exhibited significantly larger numbers of splenic B-cells, CD8+ cells and NK cells than WT littermates suggesting ongoing elevated systemic inflammation. CD4+ cells were unchanged. Notably, Muc2-/- mice exhibited increased apoptosis of splenic B-cells at baseline indicating systemic inflammation and immunosuppression. Following LPS induced sepsis, we observed increased bacterial penetrance through the distal ileum, cecum and colonic walls of Muc2-/- mice at 24 and 48 h as compared to WT littermates. In WT littermates, LPS treatment led to massive accumulation and secretion of mucus (mucus plug) in the lumen that impaired bacterial translocation. Moreover, splenocytes in LPS induced sepsis showed significantly increased apoptosis of B-cells, decreased apoptosis of CD4+ and CD8+ cells in Muc2-/- as compared to WT littermates. The aberrant immune response in Muc2-/- mice was correlated with significantly increased bacterial burden in the liver and spleen resulting in rapid BW loss and mortality. Our research suggests that during chronic IBD, host immune cells may become exhausted with continual exposure to microbial penetrants leaking through the altered and/or depleted intestinal mucus barrier. This can result in aberrant immune response against invading pathogens and increased susceptibility to sepsis. CCC, CIHR

Fetched live from OpenAlex and de-inverted. Abstracts are not stored in this database: the inverted indexes are 8.6 GB of the frame’s 9.3 GB of text, and the host has 13 GB free.

Full frame distilled prediction

Teacher imitation

Not calibrated prevalence, not ground truth. Human validation pending. Learned from the 10,348 direct Codex labels and 10,348 direct Gemma labels. Candidate is the union of thresholded teacher heads; consensus is their intersection. These outputs are machine_predicted_unvalidated and are not human labels or direct frontier model labels.

metaresearch head score (Codex)0.003
metaresearch head score (Gemma)0.002
Version: codex-gemma-dda1882f352aValidation status: machine_predicted_unvalidated
Candidate categoriesnone
Consensus categoriesnone
DomainCandidate signal: none · Consensus signal: none
Study designCandidate signal: Observational · Consensus signal: Observational
GenreCandidate signal: Empirical · Consensus signal: Empirical
Teacher disagreement score0.759
Threshold uncertainty score1.000

Codex and Gemma teacher scores by category

CategoryCodexGemma
Metaresearch0.0030.002
Meta-epidemiology (narrow)0.0000.000
Meta-epidemiology (broad)0.0010.000
Bibliometrics0.0010.000
Science and technology studies0.0000.000
Scholarly communication0.0000.000
Open science0.0000.000
Research integrity0.0000.000
Insufficient payload (model declined to judge)0.0000.000

Machine scores (provisional)

The two teacher heads of the student model, read on this work. A score orders the frame for review; it never asserts a category, and the validation status ships verbatim with every row.

Baseline scores from an immature model (maturity gate not passed, 7 training rounds). Scores rank; they never assert a category.

Opus teacher head0.018
GPT teacher head0.270
Teacher spread0.252 · how far apart the two teachers sit on this one work
Validation statusscore_only:v0-immature-baseline · verbatim from the scoring run: score_only means the number may rank works, and no category label ships from it