MétaCan
Menu
Back to cohort

Mitochondrial Damage-Associated Molecular Patterns (MTDs) Released from Hepatic Ischemia Reperfusion Induce Inflammatory Response

2018· article· en· W2884488306 on OpenAlexaff
Qianni Hu, Lauren P. Westhaver, Jean S. Marshall, Ian P.J. Alwayn

Bibliographic record

VenueTransplantation · 2018
Typearticle
Languageen
FieldMedicine
TopicOrgan Transplantation Techniques and Outcomes
Canadian institutionsDalhousie University
Fundersnot available
KeywordsProinflammatory cytokineIn vivoReperfusion injuryCytokineFlow cytometryEx vivoTumor necrosis factor alphaApoptosisInflammationBiologyImmune systemOxidative stressProgrammed cell deathPharmacologyImmunologyLiver injuryViability assayIschemiaMedicineBiochemistryInternal medicine

Abstract

fetched live from OpenAlex

Background During liver transplantation ischemia and reperfusion injury (IRI), an immune driven inflammatory response provoked by cellular oxygen deprivation, is unavoidable. The inflammatory responses, resulting from acute oxidative stress and consequent hepatocellular death during the early reperfusion phase, cause the release of damage-associated molecular patterns. Recent studies show that the release of MTDs is directly linked with functionally important immune consequences and injury. We investigated the roles of MTDs in liver IRI. Methods Rat and human hepatocytes were used in the in vitro models, mice were used in an in vivo model of hepatic IR. Liver perfusate samples were obtained from porcine and human normothermic ex vivo liver perfusion (NMP) transplants from our collaborators and correlated to outcomes. MTDs were extracted and quantified by qPCR and PicoGreen assay. Liver biopsies were assayed by transmission electron microscopy (TEM) to assess for damaged mitochondria. MitoTracker was used to test the mitochondrial homeostasis by flow cytometry. The hepatocytes or macrophages were co-cultured with different concentrations of MTDs, and cell viability was demonstrated by apoptosis detection kit; the inflammatory cytokines (TNF-α, IL-6, and IL-1β) were tested by ELISA. Results The MTDs levels were significantly higher in IRI in vitro, and marked mitochondria damage was detected by TEM and flow cytometry. Co-culture of MTDs with hepatocytes significantly increased the cell death in a dose-dependent manner. Co-culture MTDs with macrophages significantly increased proinflammatory cytokine release via TLRs/MyD88 NF-κB pathway. The release of MTDs detected in vivo was associated with inflammatory cytokine secretion and more severe liver injury compared to the control. MTDs acted as an alarmin after hepatic IRI, which might contribute to the initiation of systemic inflammatory response. MTDs levels from pig DCD perfusate were higher than DBD perfusate samples in the pig NMP model, and higher level of MTDs were associated with worse clinical outcome in both the pig and human liver transplants following NMP. Conclusion Our results indicate that IRI can elicit a significant increase of MTDs both in vitro and in vivo, and that MTDs in ex vivo perfusates can be used as potential biomarkers of hepatic IRI and may predict outcomes after liver transplantation.

Fetched live from OpenAlex and de-inverted. Abstracts are not stored in this database: the inverted indexes are 8.6 GB of the frame’s 9.3 GB of text, and the host has 13 GB free.

How this classification was reachedexpand

Full frame machine prediction

Teacher imitation

Not calibrated prevalence, not ground truth. Human validation pending. The Gemma side is a direct model label for every work in the frame, read from the title-only record. The Codex side is a classifier learned from the 10,348 direct Codex labels and calibrated to design-weighted sample rates; fields without enough sample support carry no Codex call. Candidate is the union of the two sides; consensus is their intersection. These outputs are machine_predicted_unvalidated and are not human labels.

metaresearch head score (Codex)0.000
metaresearch head score (Gemma)0.000
Version: metacan-v3-hybrid-931329e0061cValidation status: machine_predicted_unvalidated
Candidate categoriesnone
Consensus categoriesnone
DomainCandidate signal: none · Consensus signal: none
Study designCandidate signal: Bench or experimental · Consensus signal: Bench or experimental
GenreCandidate signal: Empirical · Consensus signal: Empirical
Teacher disagreement score0.002
Threshold uncertainty score0.006

Distilled classifier scores by category (both heads)

CategoryCodexGemma
Metaresearch0.0000.000
Meta-epidemiology (narrow)0.0000.000
Meta-epidemiology (broad)0.0000.000
Bibliometrics0.0000.000
Science and technology studies0.0000.000
Scholarly communication0.0000.000
Open science0.0000.000
Research integrity0.0000.001
Insufficient payload (model declined to judge)0.0020.000

Machine scores (provisional)

The two teacher heads of the student model, read on this work. A score orders the frame for review; it never asserts a category, and the validation status ships verbatim with every row.

Baseline scores from an immature model (maturity gate not passed, 7 training rounds). Scores rank; they never assert a category.

Opus teacher head0.008
GPT teacher head0.259
Teacher spread0.251 · how far apart the two teachers sit on this one work
Validation statusscore_only:v0-immature-baseline · verbatim from the scoring run: score_only means the number may rank works, and no category label ships from it

Classification

machine, unvalidated

Machine predicted; a candidate call from one source (direct Gemma or distilled Codex), not a consensus.

The models applied no category: nothing in the taxonomy fit this work.
Study designBench or experimental
Domainnot available
GenreEmpirical

How this classification was reached, model by model and score by score, is at the end of the page under "How this classification was reached".

Quick stats

Citations2
Published2018
Admission routes1
Has abstractyes

Explore more

Same venueTransplantationSame topicOrgan Transplantation Techniques and OutcomesFrench-language works237,207