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Record W2981595522 · doi:10.1016/j.ebiom.2019.09.056

Neuropathy due to impaired axonal transport of non-fragmented mitochondria in MYH14 mutation carriers

2019· letter· en· W2981595522 on OpenAlexaboutno aff
Josef Finsterer

Bibliographic record

VenueEBioMedicine · 2019
Typeletter
Languageen
FieldBiochemistry, Genetics and Molecular Biology
TopicMitochondrial Function and Pathology
Canadian institutionsnot available
Fundersnot available
KeywordsMutationPeripheral neuropathyMitochondrionMedicineMolecular biologyBiologyGeneticsEndocrinologyGene

Abstract

fetched live from OpenAlex

With interest we read the article by Almutawa et al. about a family with neuropathy, hypoacusis, and foot deformity, being attributed to the heterozygous variant R941L in MYH14 [[1]Almutawa W. Smith C. Sabouny R. Smit R.B. Zhao T. Wong R. Lee-Glover L. Desrochers-Goyette J. Ilamathi H.S. Suchowersky O. Germain M. Mains P.E. Parboosingh J.S. Pfeffer G. Innes A.M. Shutt T.E. Care4Rare Canada ConsortiumThe R941L mutation in MYH14 disrupts mitochondrial fission and associates with peripheral neuropathy.EBioMedicine. 2019; 45: 379-392https://doi.org/10.1016/j.ebiom.2019.06.018Summary Full Text Full Text PDF Scopus (16) Google Scholar]. Neuropathy was attributed to impaired fission of mitochondria resulting in oversized organelles inappropriate for retrograde axonal transportation [1Almutawa W. Smith C. Sabouny R. Smit R.B. Zhao T. Wong R. Lee-Glover L. Desrochers-Goyette J. Ilamathi H.S. Suchowersky O. Germain M. Mains P.E. Parboosingh J.S. Pfeffer G. Innes A.M. Shutt T.E. Care4Rare Canada ConsortiumThe R941L mutation in MYH14 disrupts mitochondrial fission and associates with peripheral neuropathy.EBioMedicine. 2019; 45: 379-392https://doi.org/10.1016/j.ebiom.2019.06.018Summary Full Text Full Text PDF Scopus (16) Google Scholar, 2Ueda E. Ishihara N. Mitochondrial hyperfusion causes neuropathy in a fly model of cmt2a.EMBO Rep. 2018; 19 (pii: e46502)https://doi.org/10.15252/embr.201846502Crossref Scopus (4) Google Scholar]. We have the following concerns. A main shortcoming of the study is that no nerve biopsies were carried out. Assuming that neuropathy was due to impaired fission and thus reduced axonal transport of mitochondria [[1]Almutawa W. Smith C. Sabouny R. Smit R.B. Zhao T. Wong R. Lee-Glover L. Desrochers-Goyette J. Ilamathi H.S. Suchowersky O. Germain M. Mains P.E. Parboosingh J.S. Pfeffer G. Innes A.M. Shutt T.E. Care4Rare Canada ConsortiumThe R941L mutation in MYH14 disrupts mitochondrial fission and associates with peripheral neuropathy.EBioMedicine. 2019; 45: 379-392https://doi.org/10.1016/j.ebiom.2019.06.018Summary Full Text Full Text PDF Scopus (16) Google Scholar], it is conceivable that nerve biopsy may show paucity of mitochondria within motor and sensory axons and in nerve terminals. Concerning mitochondrial functions, it is desirable to confirm normal function of the respiratory chain by appropriate biochemical investigations [[3]Amiott E.A. Lott P. Soto J. Kang P.B. McCaffery J.M. DiMauro S. Abel E.D. Flanigan K.M. Lawson V.H. Shaw J.M. Mitochondrial fusion and function in charcot-marie-tooth type 2A patient fibroblasts with mitofusin 2 mutations.Exp Neurol. 2008; 211: 115-127Crossref PubMed Scopus (72) Google Scholar]. A further shortcoming is that neuropathy was classified as axonal [[1]Almutawa W. Smith C. Sabouny R. Smit R.B. Zhao T. Wong R. Lee-Glover L. Desrochers-Goyette J. Ilamathi H.S. Suchowersky O. Germain M. Mains P.E. Parboosingh J.S. Pfeffer G. Innes A.M. Shutt T.E. Care4Rare Canada ConsortiumThe R941L mutation in MYH14 disrupts mitochondrial fission and associates with peripheral neuropathy.EBioMedicine. 2019; 45: 379-392https://doi.org/10.1016/j.ebiom.2019.06.018Summary Full Text Full Text PDF Scopus (16) Google Scholar] although there was only borderline CMAP reduction, thus not fulfilling the criteria for axonal degeneration [[4]Yadegari S. Nafissi S. Kazemi N. Comparison of electrophysiological findings in axonal and demyelinating guillain-barre syndrome.Iran J Neurol. 2014; 13: 138-143Google Scholar]. Additionally, we should know which nerves were involved, if involvement was symmetric/asymmetric, if there was upper/lower limb predominance, and if there was distal, proximal, or diffuse distribution of the lesions. We also should know if motor and sensory nerves were equally affected and if there was involvement of the autonomic fibres. Missing is an explanation of hypoacusis. We should know if it was due to sensory or neuronal involvement, which could be best achieved by application of acoustically-evoked potentials [[5]Abou-Elew M.H. Hosni N.A. Obaid E.A. Ewida A.H. The N3 potential and the efferent cochlear pathway in profound sensorineural hearing loss.J Laryngol Otol. 2017; 131: 334-340Google Scholar]. We should know why among the cranial nerves only the acoustic nerve was affected and why this cranial nerve was affected long before the onset of peripheral neuropathy. No funding was received Author contribution: JF: design, literature search, discussion, first draft, critical comments Informed consent: was obtained The study was approved by the institutional review board None. The R941L mutation in MYH14 disrupts mitochondrial fission and associates with peripheral neuropathyAs impairments in mitochondrial fission cause peripheral neuropathy, this insight into the function of NMIIC likely explains the peripheral neuropathy phenotype associated with the R941L mutation. Full-Text PDF Open Access

Fetched live from OpenAlex and de-inverted. Abstracts are not stored in this database: the inverted indexes are 8.6 GB of the frame’s 9.3 GB of text, and the host has 13 GB free.

How this classification was reachedexpand

Full frame distilled prediction

Teacher imitation

Not calibrated prevalence, not ground truth. Human validation pending. Learned from the 10,348 direct Codex labels and 10,348 direct Gemma labels. Candidate is the union of thresholded teacher heads; consensus is their intersection. These outputs are machine_predicted_unvalidated and are not human labels or direct frontier model labels.

metaresearch head score (Codex)0.000
metaresearch head score (Gemma)0.000
Version: codex-gemma-dda1882f352aValidation status: machine_predicted_unvalidated
Candidate categoriesMeta-epidemiology (narrow)
Consensus categoriesnone
DomainCandidate signal: none · Consensus signal: none
Study designCandidate signal: Not applicable · Consensus signal: Not applicable
GenreCandidate signal: Commentary · Consensus signal: none
Teacher disagreement score0.539
Threshold uncertainty score1.000

Codex and Gemma teacher scores by category

CategoryCodexGemma
Metaresearch0.0000.000
Meta-epidemiology (narrow)0.0000.000
Meta-epidemiology (broad)0.0010.000
Bibliometrics0.0000.000
Science and technology studies0.0000.000
Scholarly communication0.0000.000
Open science0.0000.000
Research integrity0.0010.001
Insufficient payload (model declined to judge)0.0000.000

Machine scores (provisional)

The two teacher heads of the student model, read on this work. A score orders the frame for review; it never asserts a category, and the validation status ships verbatim with every row.

Baseline scores from an immature model (maturity gate not passed, 7 training rounds). Scores rank; they never assert a category.

Opus teacher head0.007
GPT teacher head0.236
Teacher spread0.229 · how far apart the two teachers sit on this one work
Validation statusscore_only:v0-immature-baseline · verbatim from the scoring run: score_only means the number may rank works, and no category label ships from it

Classification

machine, unvalidated

Machine predicted; a candidate call from one teacher head, not a consensus.

Study designNot applicable
Domainnot available
GenreCommentary

How this classification was reached, model by model and score by score, is at the end of the page under "How this classification was reached".

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Citations1
Published2019
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