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Bioinformatic Prediction of Neuropathic Pain Signaling Pathways in Rheumatoid Arthritis after high throughput miRNA analysis

2020· article· en· W3016839207 on OpenAlexaboutno aff
John Anthony Francois, Lissette Anthony Delgado-Cruzata, Milena Rodríguez Álvarez, Nickolas Almodovar

Bibliographic record

VenueThe FASEB Journal · 2020
Typearticle
Languageen
FieldMedicine
TopicRheumatoid Arthritis Research and Therapies
Canadian institutionsnot available
Fundersnot available
KeywordsKEGGmicroRNAGene silencingNeuropathic painComputational biologyGeneBioinformaticsBiological pathwayGene expressionMedicineBiologyGene ontologyGeneticsNeuroscience

Abstract

fetched live from OpenAlex

microRNAs (miRNAs) are a special subgroup of RNAs which work to prevent the expression of genes at the post‐transcriptional level by “silencing” them. That is, gene expression is prevented. This ultimately affects the pathways that they are a part of and the products produced by these pathways. Due to the ability of miRNA to bind to any base pair complementary to its own thus inhibiting expression of the sequence it is bound to, they are often involved in multiple cellular pathways. The relation between a specific clinical outcome and an exact pathway that a miRNA is involved is hard to establish because of the sheer number of potential pathways one miRNA could be involved in. Bioinformatic predictions are useful because they allow us to identify the most probable pathway. That is, through the careful analysis of miRNA’s targets and the prediction of the targets’ interactions in biological mechanisms. In this project, we predicted the pathways related to deregulation of miR‐223‐3p and miR‐16‐5p. Both miRNAs were correlated with neuropathy‐related clinical measures (neuropathic pain (ID Pain) and the Toronto Clinical Neuropathy Score (TCNS) respectively (Pearson Coeff.ID Pain=0.556, p=0.014) and (Pearson Coeff.TCNS= −0.5, p=0.02)). To predict possible neuropathic pain signalling pathways in people afflicted with rheumatoid arthritis, we used the miRDB database. We identified 1,485 targets and selected targets with scores of 60+ to carry out a gene ontology analysis using the Kyoto Encyclopedia of Genes and Genomes (KEGG). Conducted through DAVID Software from the Laboratory of Human Retrovirology and Immunoinformatics, KEGG identified 72 pathways as the most likely affected by deregulation of miR‐223‐3p and miR‐16‐5p. Several pathways on this list including the FoxO, neurotrophin and sphingolipid signalling pathways are related to pain and neuropathic outcomes. Future studies will have to determine whether both miRNAs affect these pathways directly and in relationship to RA. Support or Funding Information Support for student stipends, supplies, and/or equipment used in this research was supplied by the Program for Research Initiatives in Science and Math (PRISM) at John Jay College. PRISM is funded by the Title V program within the U.S. Department of Education; the PAESMEM program through the National Science Foundation; and New York State’s Graduate Research and Technology Initiative and NYS Education Department CSTEP program.

Fetched live from OpenAlex and de-inverted. Abstracts are not stored in this database: the inverted indexes are 8.6 GB of the frame’s 9.3 GB of text, and the host has 13 GB free.

How this classification was reachedexpand

Full frame machine prediction

Teacher imitation

Not calibrated prevalence, not ground truth. Human validation pending. The Gemma side is a direct model label for every work in the frame, read from the title-only record. The Codex side is a classifier learned from the 10,348 direct Codex labels and calibrated to design-weighted sample rates; fields without enough sample support carry no Codex call. Candidate is the union of the two sides; consensus is their intersection. These outputs are machine_predicted_unvalidated and are not human labels.

metaresearch head score (Codex)0.000
metaresearch head score (Gemma)0.001
Version: metacan-v3-hybrid-931329e0061cValidation status: machine_predicted_unvalidated
Candidate categoriesnone
Consensus categoriesnone
DomainCandidate signal: none · Consensus signal: none
Study designCandidate signal: Simulation or modeling · Consensus signal: none
GenreCandidate signal: Empirical · Consensus signal: Empirical
Teacher disagreement score0.003
Threshold uncertainty score0.008

Distilled classifier scores by category (both heads)

CategoryCodexGemma
Metaresearch0.0000.001
Meta-epidemiology (narrow)0.0010.000
Meta-epidemiology (broad)0.0010.001
Bibliometrics0.0020.001
Science and technology studies0.0000.000
Scholarly communication0.0010.000
Open science0.0000.000
Research integrity0.0000.000
Insufficient payload (model declined to judge)0.0030.001

Machine scores (provisional)

The two teacher heads of the student model, read on this work. A score orders the frame for review; it never asserts a category, and the validation status ships verbatim with every row.

Baseline scores from an immature model (maturity gate not passed, 7 training rounds). Scores rank; they never assert a category.

Opus teacher head0.019
GPT teacher head0.225
Teacher spread0.206 · how far apart the two teachers sit on this one work
Validation statusscore_only:v0-immature-baseline · verbatim from the scoring run: score_only means the number may rank works, and no category label ships from it

Classification

machine, unvalidated

Machine predicted; a candidate call from one source (direct Gemma or distilled Codex), not a consensus.

The models applied no category: nothing in the taxonomy fit this work.
Study designSimulation or modeling
Domainnot available
GenreEmpirical

How this classification was reached, model by model and score by score, is at the end of the page under "How this classification was reached".

Quick stats

Citations0
Published2020
Admission routes1
Has abstractyes

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