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Overexpression of SIRT3 <sub>FL</sub> Improves Cardiac SERCA2a Function Following High Fat‐High Sucrose Feeding in Mice

2020· article· en· W3017281963 on OpenAlexaffabout
Christopher Oldfield, Teri L. Moffatt, Kimberley A. O’Hara, Bo Xiang, Vernon W. Dolinsky, Todd A. Duhamel

Bibliographic record

VenueThe FASEB Journal · 2020
Typearticle
Languageen
FieldMedicine
TopicSirtuins and Resveratrol in Medicine
Canadian institutionsUniversity of ManitobaSt. Boniface Hospital
Fundersnot available
KeywordsInternal medicineSIRT3EndocrinologySirtuinVentricleGene isoformCardiomyopathyDiabetic cardiomyopathyCardiac function curveTransgeneChemistrySkeletal muscleDiabetes mellitusMedicineBiologyAcetylationHeart failureBiochemistry

Abstract

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Background Diabetes induces a distinct form of cardiomyopathy, termed diabetic cardiomyopathy (DCM). A key pathophysiological mechanism of DCM is reduced level and activity of sarco(endo)plasmic reticulum Ca 2+ ATPase 2a (SERCA2a). Recently, it has been discovered that the function of SERCA2a is negatively regulated by the post‐translational modification, acetylation. The sirtuin (SIRT) isoform, SIRT3 full‐length (SIRT3 FL ), deacetylates protein substrates, though it is unknown if SIRT3 FL deacetylates SERCA2a. Therefore, the objective of this study was to determine if the overexpression of SIRT3 FL alters cardiac SERCA2a acetylation and function in a mouse model of diabetes, induced by high fat‐high sucrose (HFHS) feeding. Methods and Results Adult (12 weeks of age) male C57BL/6J Wild Type (WT) mice and adult male MCK‐mSIRT3‐M1‐Flag transgenic (SIRT3 FL TG) mice on a C57BL/6J background, who overexpress the SIRT3 FL isoform in skeletal and cardiac muscle, were randomized into groups fed either a standard lab diet (Control) or a HFHS diet for 4‐months. After 4‐months, HFHS‐fed mice exhibited increased body weight, left ventricle (LV) weight, LV weight relative to body weight, and liver weight, compared to control fed‐mice. LV systolic parameters were unchanged by HFHS feeding, while the A and E wave forms of HFHS‐fed mice were slowed, compared to control‐fed mice. HFHS‐fed mice exhibited LV thickening, compared to control‐fed mice. Glucose tolerance was impaired in HFHS‐fed mice, compared to control‐fed mice. Diet did not alter SIRT3 protein content. Likewise, HFHS feeding did not alter the level or function of cardiac SERCA2a. LV SERCA2a protein content was not altered by genotype. However, SIRT3 FL TG mice, regardless of diet, had 21% greater maximal SERCA activity (V max ) and an 11% greater Hill coefficient, compared to WT mice. The Ca50, which is the Ca 2+ concentration that elicits 50% of V max , was 24% lower in SIRT3 FL TG‐HFHS‐fed mice, compared to WT‐HFHS‐fed mice, indicating an increase in Ca 2+ sensitivity. Conclusion HFHS feeding induced a diabetic phenotype in WT and SIRT3 FL TG mice but level and function of cardiac SERCA2a was unchanged. However, overexpression of the SIRT3 FL isoform improved cardiac SERCA2a function, regardless of HFHS feeding. Support or Funding Information This work was supported by a grant‐in‐aid from the Heart and Stroke Foundation of Canada

Fetched live from OpenAlex and de-inverted. Abstracts are not stored in this database: the inverted indexes are 8.6 GB of the frame’s 9.3 GB of text, and the host has 13 GB free.

How this classification was reachedexpand

Full frame machine prediction

Teacher imitation

Not calibrated prevalence, not ground truth. Human validation pending. The Gemma side is a direct model label for every work in the frame, read from the title-only record. The Codex side is a classifier learned from the 10,348 direct Codex labels and calibrated to design-weighted sample rates; fields without enough sample support carry no Codex call. Candidate is the union of the two sides; consensus is their intersection. These outputs are machine_predicted_unvalidated and are not human labels.

metaresearch head score (Codex)0.000
metaresearch head score (Gemma)0.000
Version: metacan-v3-hybrid-931329e0061cValidation status: machine_predicted_unvalidated
Candidate categoriesnone
Consensus categoriesnone
DomainCandidate signal: none · Consensus signal: none
Study designCandidate signal: Bench or experimental · Consensus signal: Bench or experimental
GenreCandidate signal: Empirical · Consensus signal: Empirical
Teacher disagreement score0.003
Threshold uncertainty score0.009

Distilled classifier scores by category (both heads)

CategoryCodexGemma
Metaresearch0.0000.000
Meta-epidemiology (narrow)0.0010.000
Meta-epidemiology (broad)0.0010.001
Bibliometrics0.0010.000
Science and technology studies0.0000.001
Scholarly communication0.0000.000
Open science0.0000.000
Research integrity0.0010.002
Insufficient payload (model declined to judge)0.0030.001

Machine scores (provisional)

The two teacher heads of the student model, read on this work. A score orders the frame for review; it never asserts a category, and the validation status ships verbatim with every row.

Baseline scores from an immature model (maturity gate not passed, 7 training rounds). Scores rank; they never assert a category.

Opus teacher head0.013
GPT teacher head0.238
Teacher spread0.225 · how far apart the two teachers sit on this one work
Validation statusscore_only:v0-immature-baseline · verbatim from the scoring run: score_only means the number may rank works, and no category label ships from it

Classification

machine, unvalidated

Machine predicted; a candidate call from one source (direct Gemma or distilled Codex), not a consensus.

The models applied no category: nothing in the taxonomy fit this work.
Study designBench or experimental
Domainnot available
GenreEmpirical

How this classification was reached, model by model and score by score, is at the end of the page under "How this classification was reached".

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Citations0
Published2020
Admission routes2
Has abstractyes

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