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Record W3129791746 · doi:10.1681/asn.2020040490

Mutations in PRDM15 Are a Novel Cause of Galloway-Mowat Syndrome

2021· article· en· W3129791746 on OpenAlexfundno aff
Nina Mann, Slim Mzoughi, Ronen Schneider, Susanne J. Kühl, Denny Schanze, Verena Klämbt, Svjetlana Lovric, Youying Mao, Shasha Shi, Weizhen Tan, Michael Kühl, Ana C. Onuchic-Whitford, Ernestine Treimer, Thomas M. Kitzler, Franziska Kause, Sven Schumann, Makiko Nakayama, Florian Buerger, Shirlee Shril, Amelie T. van der Ven, Amar J. Majmundar, Kristina M. Holton, Amy Kolb, Daniela A. Braun, Jia Rao, Tilman Jobst‐Schwan, Eva Mildenberger, Thomas Lennert, Alma Kuechler, Dagmar Wieczorek, Oliver Groß, Beate Ermisch-Omran, Anja Werberger, Martin Skalej, Andreas Janecke, Neveen A. Soliman, Shrikant Mane, Richard P. Lifton, Jan Kadlec, Ernesto Guccione, Michael J. Schmeißer, Martin Zenker, Friedhelm Hildebrandt

Bibliographic record

VenueJournal of the American Society of Nephrology · 2021
Typearticle
Languageen
FieldMedicine
TopicRenal Diseases and Glomerulopathies
Canadian institutionsnot available
FundersNational Eye InstituteNational Center for Advancing Translational SciencesNational Human Genome Research InstituteNational Heart, Lung, and Blood InstituteAmerican Society of NephrologyBaden-Württemberg StiftungCanadian Institutes of Health ResearchBundesministerium für Bildung und ForschungHarvard Stem Cell InstituteNational Institute of Diabetes and Digestive and Kidney DiseasesVolkswagen FoundationDeutsche ForschungsgemeinschaftNational Medical Research CouncilNational Research FoundationNational Institutes of HealthCanadian Society of NephrologyEunice Kennedy Shriver National Institute of Child Health and Human DevelopmentKidney Foundation of Canada
KeywordsNephrotic syndromeKidney diseaseMembranous nephropathyPathogenesisExome sequencingBiologyGeneticsInternal medicineMedicineGlomerulonephritisKidneyMutationEndocrinologyBioinformaticsGene

Abstract

fetched live from OpenAlex

Significance Statement Proteinuric kidney disease is a leading cause of ESKD in children. It is part of Galloway-Mowat syndrome, a rare condition that includes a severe form of progressive nephropathy and prominent central nervous system features. The most common renal manifestation is steroid-resistant nephrotic syndrome. Mutations in the transcriptional regulator PRDM15 are a novel monogenic cause of both isolated early-onset nephrotic syndrome and Galloway-Mowat syndrome. Identifying variants in PRDM15 in these disorders provides insight into the molecular pathogenesis of nephrotic syndrome and implicates the gene as an important regulator of renal development. Background Galloway-Mowat syndrome (GAMOS) is characterized by neurodevelopmental defects and a progressive nephropathy, which typically manifests as steroid-resistant nephrotic syndrome. The prognosis of GAMOS is poor, and the majority of children progress to renal failure. The discovery of monogenic causes of GAMOS has uncovered molecular pathways involved in the pathogenesis of disease. Methods Homozygosity mapping, whole-exome sequencing, and linkage analysis were used to identify mutations in four families with a GAMOS-like phenotype, and high-throughput PCR technology was applied to 91 individuals with GAMOS and 816 individuals with isolated nephrotic syndrome. In vitro and in vivo studies determined the functional significance of the mutations identified. Results Three biallelic variants of the transcriptional regulator PRDM15 were detected in six families with proteinuric kidney disease. Four families with a variant in the protein’s zinc-finger (ZNF) domain have additional GAMOS-like features, including brain anomalies, cardiac defects, and skeletal defects. All variants destabilize the PRDM15 protein, and the ZNF variant additionally interferes with transcriptional activation. Morpholino oligonucleotide-mediated knockdown of Prdm15 in Xenopus embryos disrupted pronephric development. Human wild-type PRDM15 RNA rescued the disruption, but the three PRDM15 variants did not. Finally, CRISPR-mediated knockout of PRDM15 in human podocytes led to dysregulation of several renal developmental genes. Conclusions Variants in PRDM15 can cause either isolated nephrotic syndrome or a GAMOS-type syndrome on an allelic basis. PRDM15 regulates multiple developmental kidney genes, and is likely to play an essential role in renal development in humans.

Fetched live from OpenAlex and de-inverted. Abstracts are not stored in this database: the inverted indexes are 8.6 GB of the frame’s 9.3 GB of text, and the host has 13 GB free.

How this classification was reachedexpand

Full frame machine prediction

Teacher imitation

Not calibrated prevalence, not ground truth. Human validation pending. The Gemma side is a direct model label for every work in the frame, read from the title-only record. The Codex side is a classifier learned from the 10,348 direct Codex labels and calibrated to design-weighted sample rates; fields without enough sample support carry no Codex call. Candidate is the union of the two sides; consensus is their intersection. These outputs are machine_predicted_unvalidated and are not human labels.

metaresearch head score (Codex)0.000
metaresearch head score (Gemma)0.001
Version: metacan-v3-hybrid-931329e0061cValidation status: machine_predicted_unvalidated
Candidate categoriesnone
Consensus categoriesnone
DomainCandidate signal: none · Consensus signal: none
Study designCandidate signal: Observational · Consensus signal: none
GenreCandidate signal: Empirical · Consensus signal: Empirical
Teacher disagreement score0.002
Threshold uncertainty score0.005

Distilled classifier scores by category (both heads)

CategoryCodexGemma
Metaresearch0.0000.001
Meta-epidemiology (narrow)0.0010.000
Meta-epidemiology (broad)0.0000.000
Bibliometrics0.0010.001
Science and technology studies0.0010.001
Scholarly communication0.0000.000
Open science0.0000.000
Research integrity0.0010.000
Insufficient payload (model declined to judge)0.0020.000

Machine scores (provisional)

The two teacher heads of the student model, read on this work. A score orders the frame for review; it never asserts a category, and the validation status ships verbatim with every row.

Baseline scores from an immature model (maturity gate not passed, 7 training rounds). Scores rank; they never assert a category.

Opus teacher head0.021
GPT teacher head0.291
Teacher spread0.270 · how far apart the two teachers sit on this one work
Validation statusscore_only:v0-immature-baseline · verbatim from the scoring run: score_only means the number may rank works, and no category label ships from it

Classification

machine, unvalidated

Machine predicted; a candidate call from one source (direct Gemma or distilled Codex), not a consensus.

The models applied no category: nothing in the taxonomy fit this work.
Study designObservational
Domainnot available
GenreEmpirical

How this classification was reached, model by model and score by score, is at the end of the page under "How this classification was reached".

Quick stats

Citations27
Published2021
Admission routes1
Has abstractyes

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Same venueJournal of the American Society of NephrologySame topicRenal Diseases and GlomerulopathiesFrench-language works237,207