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Record W3161636908

The antiviral activities of a bacteriophage satellite are mechanistically tied to lateral propagation

2020· article· en· W3161636908 on OpenAlexfundno aff
Zachary K. Barth

Bibliographic record

VenueeScholarship (California Digital Library) · 2020
Typearticle
Languageen
FieldEnvironmental Science
TopicBacteriophages and microbial interactions
Canadian institutionsnot available
FundersArgonne National LaboratoryNational Institute of Allergy and Infectious DiseasesU.S. Department of Health and Human ServicesNational Institutes of HealthNorthwestern UniversityUniversity of Toronto
KeywordsBiologyBacteriophageGenomeGeneticsGeneEscherichia coli
DOInot available

Abstract

fetched live from OpenAlex

Parasitism is a common evolutionary strategy present in many branches of life, and the study of parasitism has contributed substantially to evolutionary theory. Bacteriophages, viruses that infect bacteria, are one group of parasites that have been particularly valuable models of study. Study of bacteriophages, or phages as they are also known, has vastly improved our understanding of molecular biology. The study of bacterial systems that defend against phages has provided incredibly useful tools for genetic research such as restriction enzymes and CRISPR-Cas. A particularly interesting form of anti-phage defense is hyperparasitism, parasitism of a parasite, by bacteriophage satellites that are endogenous to the bacterial genome. Bacteriophage satellites rely on bacteriophage infection for horizontal mobility while reducing or restricting production of progeny bacteriophage. This allows them to function as immune systems for the cellular populations that encode them. Within this thesis, we identify mechanisms of parasitism and bacteriophage interference by the phage inducible chromosomal island-like element or PLE, a bacteriophage satellite found within some strains of Vibrio cholerae. Using deep sequencing approaches, we have outlined DNA replication and transcriptional programs for both the PLE, and the phage it parasitizes, ICP1. Molecular and phenotypic approaches were also applied to establish how PLE parasitizes ICP1 for its own genome replication and what effect this has on the life cycle of ICP1. We have found that PLE requires ICP1 not just for induction of gene expression, but also for replication of the PLE genome. Consistent with relying on ICP1 machinery for replication and mobilization, PLE does not broadly interfere with ICP1 transcription. The sole exception to this is repression of ICP1 capsid morphogenesis genes, consistent with the PLE remodeling ICP1 virions to fit its smaller genomes. The experiments presented here, along with other recent data, suggest that PLE inhibits ICP1 through multiple mechanisms which have dual functions of also boosting PLE reproduction and mobilization. This thesis provides a foundation for understanding the parasitism strategies of ICP1 and PLE, as well as the mechanisms through which PLE restricts ICP1. Our results suggest that PLE has adapted to interfere with the replication program of ICP1 only to an extent that is beneficial to the PLE, and fit within a context pattern of pathogen and parasite evolutionary patterns.

Fetched live from OpenAlex and de-inverted. Abstracts are not stored in this database: the inverted indexes are 8.6 GB of the frame’s 9.3 GB of text, and the host has 13 GB free.

How this classification was reachedexpand

Full frame machine prediction

Teacher imitation

Not calibrated prevalence, not ground truth. Human validation pending. The Gemma side is a direct model label for every work in the frame, read from the title-only record. The Codex side is a classifier learned from the 10,348 direct Codex labels and calibrated to design-weighted sample rates; fields without enough sample support carry no Codex call. Candidate is the union of the two sides; consensus is their intersection. These outputs are machine_predicted_unvalidated and are not human labels.

metaresearch head score (Codex)0.000
metaresearch head score (Gemma)0.000
Version: metacan-v3-hybrid-931329e0061cValidation status: machine_predicted_unvalidated
Candidate categoriesnone
Consensus categoriesnone
DomainCandidate signal: none · Consensus signal: none
Study designCandidate signal: Bench or experimental · Consensus signal: Bench or experimental
GenreCandidate signal: Empirical · Consensus signal: Empirical
Teacher disagreement score0.002
Threshold uncertainty score0.005

Distilled classifier scores by category (both heads)

CategoryCodexGemma
Metaresearch0.0000.000
Meta-epidemiology (narrow)0.0000.000
Meta-epidemiology (broad)0.0000.000
Bibliometrics0.0000.000
Science and technology studies0.0000.001
Scholarly communication0.0010.001
Open science0.0000.000
Research integrity0.0000.001
Insufficient payload (model declined to judge)0.0020.000

Machine scores (provisional)

The two teacher heads of the student model, read on this work. A score orders the frame for review; it never asserts a category, and the validation status ships verbatim with every row.

Baseline scores from an immature model (maturity gate not passed, 7 training rounds). Scores rank; they never assert a category.

Opus teacher head0.010
GPT teacher head0.200
Teacher spread0.190 · how far apart the two teachers sit on this one work
Validation statusscore_only:v0-immature-baseline · verbatim from the scoring run: score_only means the number may rank works, and no category label ships from it

Classification

machine, unvalidated

Machine predicted; a candidate call from one source (direct Gemma or distilled Codex), not a consensus.

The models applied no category: nothing in the taxonomy fit this work.
Study designBench or experimental
Domainnot available
GenreEmpirical

How this classification was reached, model by model and score by score, is at the end of the page under "How this classification was reached".

Quick stats

Citations0
Published2020
Admission routes1
Has abstractyes

Explore more

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