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Endothelium‐dependent vasorelaxation and blood pressure are preserved in mice with chronic hyperendothelinemia

2018· article· en· W3176775834 on OpenAlexaff
Zachary I. Grunewald, Thomas J. Jurrissen, James R. Ball, Makenzie L. Woodford, Lauren K. Walsh, F. Daniel Ramirez, Scott M. Brown, Annayya R. Aroor, Pierre Paradis, Ernesto L. Schiffrin, Shawn B. Bender, Luis A. Martinez‐Lemus, Jaume Padilla

Bibliographic record

VenueThe FASEB Journal · 2018
Typearticle
Languageen
FieldMedicine
TopicNitric Oxide and Endothelin Effects
Canadian institutionsMcGill UniversityJewish General Hospital
FundersNational Institutes of HealthSCAN Foundation
KeywordsInternal medicineEndocrinologyPhenylephrineVasoconstrictionEndotheliumWild typeVasodilationBlood pressureEndothelial dysfunctionEndothelin 1ChemistryBiologyMedicineReceptor

Abstract

fetched live from OpenAlex

Endothelin‐1 (ET‐1) is a potent vasoactive peptide implicated in the development of glucose intolerance, endothelial dysfunction, and hypertension. However, whether the detrimental metabolic and vascular effects caused by chronically elevated circulating ET‐1 (i.e., hyperendothelinemia) are exacerbated by obesity remains unknown. Accordingly, six‐week‐old female transgenic mice overexpressing human preproET‐1 selectively in the endothelium (eET‐ 1) and non‐transgenic (i.e., wild‐type) littermates were fed either chow or a Western diet (46% Kcal fat, 36% Kcal carbohydrate) to induce obesity for 21 weeks, creating four distinct groups (n=9–11/group): wild‐type chow‐fed, eET‐1 chow‐fed, wild‐type Western diet‐fed, eET‐1 Western diet‐fed. eET‐1 mice exhibited a 27‐fold (chow) and 18‐fold (Western diet) elevation in plasma ET‐1 concentration relative to wild‐type mice. Notably, despite such marked elevations in plasma ET‐1, blood pressure (systolic and diastolic) and aortic stiffness, assessed by pulse wave velocity, were unaltered in eET‐1 mice relative to wild‐type counterparts, regardless of diet. Similarly, independent of diet, ex vivo endothelium‐dependent relaxation (i.e., vasomotor responses to acetylcholine and insulin) in femoral and coronary arteries were not different between eET‐1 and wild‐type mice, and modestly enhanced in the aorta of eET‐1 mice. eET‐1 mice also exhibited suppressed phenylephrine‐induced aortic vasoconstriction relative to wild‐type mice, an effect that was amplified by Western diet‐feeding. Chow‐fed eET‐1 mice exhibited mild glucose intolerance and this genotype effect was not aggravated by diet‐induced obesity. Collectively, contrary to our hypothesis, we find that endothelial overexpression of ET‐1 does not lead to impaired endothelial function in lean or obese mice. The retention of a functional endothelial phenotype and suppression of alpha‐adrenergic vasoconstriction in this model of chronic hyperendothelinemia may contribute to the preservation of normal blood pressure. Follow‐up experiments are ongoing to delineate potential homeostatic compensatory adaptations that underlie this largely conserved cardiometabolic phenotype in the presence of chronic hyperendothelinemia. Support or Funding Information NIH, SCAN This abstract is from the Experimental Biology 2018 Meeting. There is no full text article associated with this abstract published in The FASEB Journal .

Fetched live from OpenAlex and de-inverted. Abstracts are not stored in this database: the inverted indexes are 8.6 GB of the frame’s 9.3 GB of text, and the host has 13 GB free.

How this classification was reachedexpand

Full frame machine prediction

Teacher imitation

Not calibrated prevalence, not ground truth. Human validation pending. The Gemma side is a direct model label for every work in the frame, read from the title-only record. The Codex side is a classifier learned from the 10,348 direct Codex labels and calibrated to design-weighted sample rates; fields without enough sample support carry no Codex call. Candidate is the union of the two sides; consensus is their intersection. These outputs are machine_predicted_unvalidated and are not human labels.

metaresearch head score (Codex)0.000
metaresearch head score (Gemma)0.000
Version: metacan-v3-hybrid-931329e0061cValidation status: machine_predicted_unvalidated
Candidate categoriesnone
Consensus categoriesnone
DomainCandidate signal: none · Consensus signal: none
Study designCandidate signal: Bench or experimental · Consensus signal: Bench or experimental
GenreCandidate signal: Empirical · Consensus signal: Empirical
Teacher disagreement score0.002
Threshold uncertainty score0.007

Distilled classifier scores by category (both heads)

CategoryCodexGemma
Metaresearch0.0000.000
Meta-epidemiology (narrow)0.0010.001
Meta-epidemiology (broad)0.0010.001
Bibliometrics0.0010.001
Science and technology studies0.0000.001
Scholarly communication0.0010.001
Open science0.0000.001
Research integrity0.0010.002
Insufficient payload (model declined to judge)0.0020.001

Machine scores (provisional)

The two teacher heads of the student model, read on this work. A score orders the frame for review; it never asserts a category, and the validation status ships verbatim with every row.

Baseline scores from an immature model (maturity gate not passed, 7 training rounds). Scores rank; they never assert a category.

Opus teacher head0.012
GPT teacher head0.238
Teacher spread0.226 · how far apart the two teachers sit on this one work
Validation statusscore_only:v0-immature-baseline · verbatim from the scoring run: score_only means the number may rank works, and no category label ships from it

Classification

machine, unvalidated

Machine predicted; a candidate call from one source (direct Gemma or distilled Codex), not a consensus.

The models applied no category: nothing in the taxonomy fit this work.
Study designBench or experimental
Domainnot available
GenreEmpirical

How this classification was reached, model by model and score by score, is at the end of the page under "How this classification was reached".

Quick stats

Citations0
Published2018
Admission routes1
Has abstractyes

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