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Rapid assessment of SARS-CoV-2–evolved variants using virus-like particles

2021· article· en· 354 citations· W3210408195 on OpenAlex· 10.1126/science.abl6184

Why is this work in the frame?

A frame that forgets how it found something cannot be audited. These are the routes that admitted this work.

Canadian funderA Canadian agency funded it. The work may carry no Canadian affiliation at all.

No Canadian affiliation. An affiliation-only frame — the usual design — would never have seen this work. It is one of the works that make the case for inverting the frame.

Machine scores (provisional)

Baseline scores from an immature model (maturity gate not passed, 7 training rounds). Scores rank; they never assert a category.

The two teacher heads of the student model, read on this work. A score orders the frame for review; it never asserts a category, and the validation status ships verbatim with every row.

Opus teacher head0.140
GPT teacher head0.434
Teacher spread
0.294 · how far apart the two teachers sit on this one work
Validation status
score_only:v0-immature-baseline · verbatim from the scoring run: score_only means the number may rank works, and no category label ships from it

Abstract

Efforts to determine why new severe acute respiratory syndrome coronavirus 2 (SARS-CoV-2) variants demonstrate improved fitness have been limited to analyzing mutations in the spike (S) protein with the use of S-pseudotyped particles. In this study, we show that SARS-CoV-2 virus-like particles (SC2-VLPs) can package and deliver exogenous transcripts, enabling analysis of mutations within all structural proteins and at multiple steps in the viral life cycle. In SC2-VLPs, four nucleocapsid (N) mutations found universally in more-transmissible variants independently increased messenger RNA delivery and expression ~10-fold, and in a reverse genetics model, the serine-202→arginine (S202R) and arginine-203→methionine (R203M) mutations each produced >50 times as much virus. SC2-VLPs provide a platform for rapid testing of viral variants outside of a biosafety level 3 setting and demonstrate N mutations and particle assembly to be mechanisms that could explain the increased spread of variants, including B.1.617.2 (Delta, which contains the R203M mutation).

Fetched live from OpenAlex and de-inverted. Abstracts are not stored in this database: the inverted indexes are 8.6 GB of the frame’s 9.3 GB of text, and the host has 13 GB free.

The record

Venue
Science
Topic
SARS-CoV-2 and COVID-19 Research
Field
Medicine
Canadian institutions
Funders
National Institute on AgingGladstone InstitutesBiogenNational Institutes of HealthNational Institute of Allergy and Infectious DiseasesNatural Sciences and Engineering Research Council of CanadaPfizerHoward Hughes Medical Institute
Keywords
MutationVirusVirologyBiologyVirus-like particleCoronavirusGeneticsSevere acute respiratory syndrome coronavirus 2 (SARS-CoV-2)Reverse geneticsViral life cycleArginineRNACoronavirus disease 2019 (COVID-19)GeneAmino acidViral replicationGenomeMedicineRecombinant DNA
Has abstract in OpenAlex
yes