MétaCan
← all works

Cannabinoid receptor 2 (CB2R) mediates cannabinol (CBN) induced developmental defects in zebrafish

2022· preprint· en· 0 citations· W4225440245 on OpenAlex· 10.22541/au.164864237.79266323/v1

Why is this work in the frame?

A frame that forgets how it found something cannot be audited. These are the routes that admitted this work.

Canadian affiliationAn author listed a Canadian institution. This is the only route the usual frame has.
Canadian funderA Canadian agency funded it. The work may carry no Canadian affiliation at all.

The three-model screen

all 1,000 screened works →

All three models called this out of scope.

stratum: aff_core · design weight: 5595.24 (the sample is stratified; any rate computed without the weight is wrong)
Claude Opus 4.8OUT
genre: empirical
about Canada: no
confidence: high

Zebrafish study of cannabinol-induced developmental defects via CB2 receptors; the object is developmental toxicology.

GPT-5.6 (high)OUT
genre: empirical
about Canada: no
confidence: high

This biomedical experiment studies cannabinoid effects on zebrafish development, not research practice.

Grok 4.5OUT
genre: empirical
about Canada: no
confidence: high

Developmental toxicology of cannabinol in zebrafish embryos; biomedical domain.

Abstract

Background and Purpose: Of the three primary cannabinoids in cannabis: 9-Tetrahydrocannabinol (9-THC), Cannabidiol (CBD) and Cannabinol (CBN), very little is known about the actions of CBN, the primary oxidative metabolite of THC. Our goal was to determine if CBN exposure during gastrulation alters embryonic development, and if so, via which cannabinoid receptors. Experimental Approach: Zebrafish embryos during the gastrulation stage (5-10.75 hpf) were exposed to CBN in the presence or absence of cannabinoid receptor blockers. We examined neuronal morphology, hair cell development and locomotion. Key Results: Embryos exhibited dose-dependent malformations in morphology, increased mortality, decreased locomotion and a reduction in motor neuron branching. Larva exhibited a significant reduction in the response to sound stimuli. CBN exposure altered the development of otic vesicles and hair cells associated with the lateral line. Pharmacological block of CB2Rs with AM 630 or JTE 907 prevented many of the CBN-induced developmental defects, while block of CB1Rs with AM 251 or CP 945598 had little or no effect. Conclusion and Implications: Altogether we show that embryonic exposure to CBN results in alterations in embryonic growth, neuronal and hair cell development, and physiology and behavior via CB2R-mediated mechanisms. Our results suggest that embryos exposed to CBN may be at an increased risk of abnormal development.

Stored with the screening record, where it is evidence for the labels above.

The record

Venue
Topic
Zebrafish Biomedical Research Applications
Field
Biochemistry, Genetics and Molecular Biology
Canadian institutions
University of Alberta
Funders
Natural Sciences and Engineering Research Council of Canada
Keywords
Cannabinoid receptorCannabinoidCannabinolZebrafishBiologyEndocannabinoid systemEmbryonic stem cellGastrulationCell biologyEmbryoEmbryogenesisEndocrinologyInternal medicineReceptorNeuroscienceChemistryMedicineAgonistBiochemistry
Has abstract in OpenAlex
yes